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Spinocerebellar Degenerations:     more detail

Spinocerebellar degenerations: Proceedings of the International Symposium on Spinocerebellar Degenerations held October 12-14, 1978, Tokyo Trh and Spinocerebellar Degeneration Spinocerebellar Degenerations: The Ataxias and Spastic Paraplegias: Blue Books of Neurology Series, Volume 31 by Alexis Brice MD, Stefan-M. Pulst MD, 2007-05-23 Hereditary Neuropathies and Spinocerebellar Atrophies The Spino-Cerebellar Degenerations by J G Greenfield, 1954-01-01 The Spino-Cerebellar Degenerations by J G Greenfield, 1954-01-01 Advances in Research on Neurodegeneration.: Volume 7 (Journal of Neural Transmission Supplementum) Handbook of Ataxia Disorders (Neurological Disease and Therapy)

lists with details

41. Sp : On Medical Dictionary Online
    Familial Spinocerebellar Degeneration, Hereditary Spinocerebellar Degeneration, Inheritedspinocerebellar degenerations spinocerebellar degenerations, Familial  
    http://www.online-medical-dictionary.org/?q=~Sp

42. SPINOCEREBELLAR DEGENERATIONS
    ataxia A serious medical condition. Resources to obtain and spinocerebellar degenerations Tokyo, 1978. International Symposium onspinocerebellar degenerations. Ten Years to Live Henry J. Schut.  
    http://www.websters-online-dictionary.org/definition/english/Sp/Spinocerebellar
    
    Extractions: Philip M. Parker, INSEAD. SPINOCEREBELLAR DEGENERATIONS Specialty Definition: SPINOCEREBELLAR DEGENERATIONS Domain Definition A heterogenous group of degenerative syndromes marked by progressive cerebellar dysfunction either in isolation or combined with other neurologic manifestations. Sporadic and inherited subtypes occur. Inheritance patterns include autosomal dominant, autosomal recessive, and X-linked. ( references Source: compiled by the editor from various references ; see credits. Top Commercial Usage: SPINOCEREBELLAR DEGENERATIONS Domain Title Source: compiled by the editor from various references ; see credits. Top Alternative Orthography: SPINOCEREBELLAR DEGENERATIONS Hexadecimal (or equivalents, 770AD-1900s) references Leonardo da Vinci (1452-1519; backwards) references Binary Code (1918-1938, probably earlier) references HTML Code references ISO 10646 references Encryption (beginner's substitution cypher): references Top google_alternate_ad_url="http://www.websters-online-dictionary.org/js/googlead.asp?kw=SPINOCEREBELLAR DEGENERATIONS";google_ad_client="pub-7500086874932040"; google_ad_width=728; google_ad_height=90; google_ad_format="728x90_as"; google_color_border="336699"; google_color_bg="FFFFFF";google_color_link="0000FF";google_color_url="008000"; google_color_text="000000"; var rotate_delay = 3000;

43. Cerebellar Ataxia : A Serious Medical Condition. Resources To Obtain And Further
    spinocerebellar degenerations Tokyo, 1978. International Symposium onspinocerebellar degenerations. brought to you by Creative Power!  
    http://databank.oxydex.com/compendium_bibliographium/advanced_medical_specialtie

44. Neurology -- Abstracts: Rosenberg Et Al. 31 (8): 1003
    The dominantly inherited spinocerebellar degenerations may result from a primarydeficit of glialneuronal interaction, resulting in neuronal loss but with a  
    http://www.neurology.org/cgi/content/abstract/31/8/1003
    
    Extractions: Proteins were separated on two-dimensional acrylamide gels obtained from brain samples of patients with Joseph disease, Huntington disease (HD) and multiple sclerosis. Similar protein separations were made from cultured skin fibroblasts of Joseph disease patients. Two major classes of proteins, one with a MW of 50,000 probably representing the glial filamentous acidic protein, or another class with a MW of 40,000 (proteins Jc, Jd, L1 and L2) were increased in the cerebellum of six Joseph disease patients. The same protein species were abnormally increased in HD brains, mainly in the basal

45. Neurology -- Abstracts: Kark And Rodriguez-Budelli 29 (1): 126
    ARTICLES. Pyruvate dehydrogenase deficiency in spinocerebellar degenerations.RA Kark and M RodriguezBudelli. To study the incidence  
    http://www.neurology.org/cgi/content/abstract/29/1/126
    
    Extractions: To study the incidence of abnormalities of the pyruvate (PDH) or ketoglutarate (KGDH) dehydrogenase complexes in patients with spinocerebellar degenerations, we measured the activities of PDH and KGDH in platelet-enriched preparations from the blood of 14 patients. Low PDH was found in 6 of the 14 patients; low KGDH was found in 2 of the 6. PDH-normal and PDH-abnormal patients could not be distinguished by clinical criteria. These results extend previous studies, which suggested abnormalities of pyruvate oxidation in patients with hereditary ataxias. The data imply that deficient activity of the PDH complex may be associated with spinocerebellar degenerations, and that the clinical phenotypes of the

46. Parkinsonism - The Doctors Lounge(TM)
    Wilson’s disease. HallervordenSpatz disease. Olivopontocerebellar andspinocerebellar degenerations. Familial basal ganglia calcification.  
    http://www.thedoctorslounge.net/clinlounge/diseases/neurology/parkinsonism.htm

47. Spinocerebellar Ataxia
    Of particular interest are these quotes under the spinocerebellar degenerations section Many of these syndromes are hereditary; others are sporadic.  
    http://stonecoldsober.deep-ice.com/research/spinocerr-ataxia.html
    
    Extractions: Web Site Hosting fre e web hosting ... Reseller Hosting This is the results of my digging up information on Spinocerebellar Ataxia. This Cerebellar and Spinocerebellar Disorders page has some basic information on movement disorders, of which this is one. Of particular interest are these quotes under the "Spinocerebellar Degenerations" section: "Many of these syndromes are hereditary; others are sporadic. Spinocerebellar degenerations can be broadly categorized as predominantly spinal ataxias, cerebellar ataxias, or multiple systems atrophy (see Table 179-4 ). There is no treatment." "Cerebellar ataxias generally begin between ages 30 and 50 and may be sporadic or dominantly inherited. Pathologic changes are restricted to the cerebellum and, occasionally, the inferior olives. Clinically, only signs of cerebellar dysfunction can be detected." Under the "Structural Lesions" section I find this: "Alcoholism with nutritional deprivation can cause degeneration of the vermis and anterior cerebellum with profound gait ataxia. Acquired cerebellar syndromes may also be caused by hypothyroidism, various toxins (carbon monoxide, heavy metals, phenytoin), hyperpyrexia, and repeated head trauma. Rarely, reversible pancerebellar dysfunction may follow viral infections in children. A rare, profound cerebellar degeneration may accompany certain malignancies in adults."

48. M2cnsdeg
    spinocerebellar degenerations include Pontocerebellar Atrophy, Friedreich sAtaxia. Motor Neuron diseases include Amyotrophic Lateral Sclerosis.  
    http://www.neuro.nwu.edu/meded/m2/m2cnsdeg.html
    
    Extractions: CNS Degenerative Disease Numa Marquez-Sterling, MD, Northwestern University Medical School Return to Education Index This material is the lecture handout for the lecture of the same title for the Scientific Basis of Medicine Course, offered to 2nd Year Medical Students at Northwestern University Medical School. Background CNS degenerative diseases are characterized by progressive, selective neuronal loss in defined regions or functional systems (e.g., cerebellum, basal ganglia, upper or lower motor neurons, associative neocortex) with varying degrees of replacement gliosis . Associated histopathologic changes include neuronal inclusions (e.g., Lewy bodies , neurofibrillary tangles) and extraneuronal accumulations (e.g., senile plaques). Etiology or primary inciting pathophysiologic event is generally unknown (i.e., these diseases are generally regarded as idiopathic ). Degenerative diseases may be sporadic or inherited . For some diseases both sporadic (predominate) and inherited (lesser component; generally autosomal dominant) forms exist (e.g., sporadic and familial Alzheimer's Disease). Degenerative diseases range from very common (e.g., Alzheimer's Disease) to very rare (e.g., Hallervorden-Spatz Disease). Prevalence generally increases with age . Treatment options are generally quite limited and consist largely of palliative/supportive care. Goals / Basic Concepts 1. Understand the basic paradigm for neurodegenerative disease as summarized above (SEE also summary figures below).

49. CCHS Clinical Digital Library
    spinocerebellar degenerations Access document. Family Practice Handbook4th Ed.2001 Table of contents Chapter 9 Neurology Table  
    http://cchs-dl.slis.ua.edu/clinical/neurology/cns/ataxia.htm
    
    Extractions: Clinical Resources by Topic: Neurology Ataxia Clinical Resources Pediatrics Geriatrics Pathology Genetics ... Miscellaneous Resources See also: Chapter 364: Ataxic Disorders Table of contents The Inherited Ataxias: Access document Machado-Joseph Disease/SCA3: Access document Access document Autosomal Recessive Ataxias: Access document Ataxia Telangiectasia: Access document Mitochondrial Ataxias: Access document Chapter 363: Parkinson's Disease and Other Extrapyramidal Disorders Table of contents Machado-Joseph Disease (Spinocerebellar Ataxia Type 3): Access document Drug-Induced Movement Disorders: Access document Treatment: Access document Neuroleptic Malignant Syndrome:

50. DeCS - Changed Terms
    SINUS THROMBOSIS, SINUS THROMBOSIS, INTRACRANIAL. SPINOCEREBELLAR DEGENERATION,spinocerebellar degenerations. STIFFMAN SYNDROME, STIFF-PERSON SYNDROME.  
    http://decs.bvs.br/I/rep_i2000.htm
    
    Extractions: DeCS 2000 - Changed terms Replaced Replaced-by ACOUSTIC NERVE VESTIBULOCOCHLEAR NERVE ACOUSTIC NERVE DISEASES VESTIBULOCOCHLEAR NERVE DISEASES ADIE'S SYNDROME ADIE SYNDROME AMYGDALOID BODY AMYGDALA ANDROGEN-BINDING PROTEINS ANDROGEN-BINDING PROTEIN ANOSMIA OLFACTION DISORDERS APRAXIA APRAXIAS ARNOLD-CHIARI DEFORMITY ARNOLD-CHIARI MALFORMATION ASPARTIC PROTEINASES ASPARTIC ENDOPEPTIDASES CALLUS BONY CALLUS CEREBELLAR DYSSYNERGIA MYOCLONIC CEREBELLAR DYSSYNERGIA CEREBRAL ANEURYSM INTRACRANIAL ANEURYSM CEREBRAL ANOXIA HYPOXIA, BRAIN CEREBRAL ARTERIOSCLEROSIS INTRACRANIAL ARTERIOSCLEROSIS CEREBRAL ARTERIOVENOUS MALFORMATIONS INTRACRANIAL ARTERIOVENOUS MALFORMATIONS CEREBRAL ARTERY DISEASES CEREBRAL ARTERIAL DISEASES CEREBRAL EMBOLISM AND THROMBOSIS INTRACRANIAL EMBOLISM AND THROMBOSIS CEREBRAL ISCHEMIA BRAIN ISCHEMIA CEREBRAL ISCHEMIA, TRANSIENT ISCHEMIC ATTACK, TRANSIENT CEREBRAL SCLEROSIS, DIFFUSE DIFFUSE CEREBRAL SCLEROSIS OF SCHILDER CEREBROSPINAL OTORRHEA CEREBROSPINAL FLUID OTORRHEA CEREBROSPINAL RHINORRHEA CEREBROSPINAL FLUID RHINORRHEA CHARCOT-MARIE DISEASE CHARCOT-MARIE-TOOTH DISEASE CHORDATA CHORDATA, NONVERTEBRATE

51. NodeWorks - Spinocerebellar Degenerations: Friedreich's Ataxia
    Directory.  
    http://dir.nodeworks.com/Health/Conditions_and_Diseases/Neurological_Disorders/S
    
    Extractions: in entire NodeWorks Directory in Health in Neurological Disorders in Spinal Cord in Spinocerebellar Degenerations in ++ Friedreich's Ataxia Top Health Neurological Disorders Spinal Cord ... Spinocerebellar Degenerations Friedreich's Ataxia An autosomal recessive disease, usually of childhood onset, characterized pathologically by degeneration of the spinocerebellar tracts, posterior columns, and to a lesser extent the corticospinal tracts. GeneClinics: Friedreich Ataxia An in depth look at this disorder. Includes a summary, diagnosing, molecular genetic testing, clinical description and management. Living Life with Ataxia An informative site about living a positive life in spite of having a progressive hereditary neurological disease called Friedreich's Ataxia. NINDS Friedreich's Ataxia An information sheet the National Institute of Neurological Disorders and Stroke . NORD: Friedreich's Ataxia Offers the synonyms, a general discussion and further resources.

52. Myelitis
    spinocerebellar degenerations Friedreich s ataxia and other hereditaryforms of systemic spinal degeneration. Spinal muscular atrophies.  
    http://www.amershamhealth.com/medcyclopaedia/medical/Volume VI 1/MYELITIS.ASP
    
    Extractions: Amershamhealth.com Search for: Type a word or a phrase. All forms of the word are searchable. Browse entry words starting with: A B C D ... Other characters Myelitis, a term that is sometimes improperly used as a synonym for myelopathy; the two terms, however, have quite different meanings. Both indicate spinal cord involvement by some pathological event: myelopathy is a more inclusive term that does not imply an aetiological factor while myelitis should be restricted to inflammatory diseases of the spinal cord. Myelopathy includes myelitis; myelitis produces a myelopathy. Any process that impairs the function of the spinal cord produces a myelopathy; in the common use, however, the term is usually employed with an adjective such as "compressive", "degenerative" or "congenital". Despite the fact that conditions such as spinal cord tumours or hydrosyringomyelia produce symptoms of myelopathy they are usually considered as specific, separate entities. On the other hand, "myelitis" is frequently a sort of wastebasket in which all myelopathies of unknown or undetectable aetiology are included. With these caveats in mind an attempt at classification can be made. MYELITIS Viral infections Usually of acute onset due to viruses that affect the grey matter (poliomyelitis). Herpes, coxackie and polio viruses are the most common agents.

53. Olivopontocerebellar Atrophy
    Dear Dave Cerebellar degenerations (also called spinocerebellar degenerations)can either be sporadic or heredofamilial. Heredofamilial  
    http://www.medhelp.org/perl6/neuro/archive/15346.html
    
    Extractions: : : Hello: : : Can you tell me what diagnostic test currently are used to diagnose and confirm OPCA? : : Thank you very much!! : Dear Dave: : Cerebellar degenerations (also called spinocerebellar degenerations) can either be sporadic or heredofamilial. Heredofamilial cerebellar degenerations can be either autosomal dominant (various types termed spinocerebellar ataxia = SCA 1 - 7, DRPLA, Machado-Joseph disease, etc) or autosomal recessive (Friedreich's ataxia, and some other rare types). : The syndrome of progressive ataxia (imbalance of gait, incoordination of hands, slurred speech), in association with some other characteristic abnormalities (peripheral neuropathy, optic atrophy, retinitis pigmentosa, dementia, corticospinal dysfunction, oculomotor dysfunction, etc) is seen with heredofamilial forms. Ataxia is associated with autonomic dysfunction and/or parkinsonian features in sporadic cases. Either sporadic or autosomal dominant forms of cerebellar degeneration can be associated with atrophy of the cerebellum, pons and inferior olives, either on MRI, or at autopsy. Hence, OPCA (olivopontocerebellar atrophy) is used as a descriptive term for these disorders.

54. Re: Olivopontocerebellar Atrophy
    Dear Dave Cerebellar degenerations (also called spinocerebellar degenerations)can either be sporadic or heredofamilial. Heredofamilial  
    http://www.medhelp.org/forums/neuro/archive/15044.html
    
    Extractions: : Hello: : Can you tell me what diagnostic test currently are used to diagnose and confirm OPCA? : Thank you very much!! Dear Dave: Cerebellar degenerations (also called spinocerebellar degenerations) can either be sporadic or heredofamilial. Heredofamilial cerebellar degenerations can be either autosomal dominant (various types termed spinocerebellar ataxia = SCA 1 - 7, DRPLA, Machado-Joseph disease, etc) or autosomal recessive (Friedreich's ataxia, and some other rare types). The syndrome of progressive ataxia (imbalance of gait, incoordination of hands, slurred speech), in association with some other characteristic abnormalities (peripheral neuropathy, optic atrophy, retinitis pigmentosa, dementia, corticospinal dysfunction, oculomotor dysfunction, etc) is seen with heredofamilial forms. Ataxia is associated with autonomic dysfunction and/or parkinsonian features in sporadic cases. Either sporadic or autosomal dominant forms of cerebellar degeneration can be associated with atrophy of the cerebellum, pons and inferior olives, either on MRI, or at autopsy. Hence, OPCA (olivopontocerebellar atrophy) is used as a descriptive term for these disorders.

55. Korsakov's Journal Of Neurology And Psychiatry - Abstracts ¹2 2004
    MA Kulikov, SN Illarioshkin, ED Markova Voluntary postural control learning witha use of visual biofeedback in patients with spinocerebellar degenerations.  
    http://www.mediasphera.aha.ru/neurol/2004/2/e2-04ref.htm
    
    Extractions: The peculiarities of personality and mental state in children and adolescents with chronic tension-type headache Author's address: Neurological and psychiatric examination of 334 children, aged 7–15 years, 87 (23%) of them suffering from chronic tension-type headache (CTTH) is presented. All the patients had mental (mostly depressive and anxiety) disorders and 95.3% of them – psychosomatic ones. Clinical heterogeneity of CTTH was revealed: in the cases of daily continuous headache there were depressive disorders, in daily intermittent headache – depressive and anxiety-phobic disorders and in frequent headache pattern neurasthenia was predominantly detected. Personality features of CTTH patients were as follows: high anxiety, aggressiveness, asthenia, low “Ego” power, high frustration, sensitivity, hypochondriac, depressive and hysterical tendencies, affective rigidity. Patients realized mostly non-adaptive coping styles both for high and low pain intensiveness. Combination of psychopharmacology, psychotherapy and biofeedback for the CTTH treatment is recommended.

56. Neurogenetics
    Friedreichs Ataxia * Cerebellar degeneration (OPCA) * Spastic Ataxia * Inheriteddiseases other than spinocerebellar degenerations * Huntington s Chorea  
    http://neurology.medsch.ucla.edu/neurogen.htm

57. Classification Of Ataxias
    Classification of Ataxias. (Cerebellar and spinocerebellar degenerations). 12 March2003. Primary. Inherited. Autosomal recessive. Autosomal dominant. Sex linked.  
    http://www.angelfire.com/retro/michaelpoon168/classification_of_ataxias.htm
    
    Extractions: Michael Poon's Shrine of Neurology HOME CONTENTS CONTACT US HOME ... CONTACT US Classification of Ataxias (Cerebellar and Spinocerebellar Degenerations) 12 March 2003 Primary Inherited Autosomal recessive Autosomal dominant Sex linked Sporadic Multisystem atrophy Idiopathic cerebellar degeneration Secondary Metabolic disorders Mitochondrial disease Toxic disorders Vascular disease Infective disorders Prion disease Paraneoplastic cerebellar degeneration Classification of inherited ataxias Autosomal recessive Friedreich’s ataxia Spinocerebellar ataxia with retained tendon reflexes Spinocerebellar ataxia with isolated Vitamin E deficiency Ataxia telengiectasia Other rare recessive ataxias Hypogonadotrophic hypogonadism (Matthews – Rundle syn) Learning disability, optic atrophy, spasticity (Behr’s syn)

58. Neuro Seminars
    Rigshospitalet, Clinical and genetic aspects of spinocerebellar degenerations.Steven Haugbøl Neurobiology Research Unit, Rigshospitalet,  
    http://www.neuroseminars.ku.dk/
    
    Extractions: Seminars are held the last Friday of every month at 3 pm in room 22.1.29 (The library at The Department of Medical Anatomy, MAI) unless announced otherwise. As a general rule we have two talks each lasting 20 minutes and ten minutes question time. After the talks there will be some sort of refreshment. Organisers Nanna MacAuley , Department of Medical Physiology Milena Penkova , Department of Medical Anatomy Ole Kjaerulff , Department of Medical Physiology Jens B. Nielsen , Institute of Exercise and Sport Sciences Nicolas T. Petersen , Department of Medical Physiology Frederik Vilhardt , Department of Medical Anatomy 2003 Seminars Changes are unlikely to occur Date Speaker Title 30 january 2004 Institute of Molecular Pathology, University of Copenhagen Programmed cell death in cerebral ischemia: a morphological perspective Susana Aznar Neurobiology Research Unit, Rigshospitalet Non-serotonergic Dorsal and Median Raphe projection onto parvalbumin- and calbindin-containing neurons in hippocampus and septum 27 february 2004 Jonathan Cole University of Bournemouth and University of Southhampton The physiology and phenomenology of loss of touch and proprioception below the neck 26 march 2004 Hanne Roed IVIG treatment for patients with acute optic neuritis 30 April 2004 Department of Medical Biochemistry and Genetics, Rigshospitalet

59. Sites Médicaux Francophones : Classement Par Mots Clés MeSH, Qualificatifs Et
    spinocérébelleuses spinocerebellar degenerations;  
    http://www.chu-rouen.fr/ssf/santpathd.html
    
    Extractions: dacryocystite [dacryocystitis] dacryocysto-rhinostomie [dacryocystorhinostomy] dactylologie [manual communication] danazol [danazol] Danemark [Denmark] danse [dancing] dapsone [dapsone] datura [datura] datura stramonium [datura stramonium] daunorubicine [daunorubicin] De Lange, syndrome [De Lange syndrome] débit cardiaque [Cardiac Output] débit cardiaque élevé [cardiac output, high] débit expiratoire forcé [forced expiratory flow rates] débit expiratoire pointe [peak expiratory flow rate] débit plasmatique rénal [renal plasma flow] débit sanguin [blood flow velocity] début accouchement [labor onset] décalage horaire, syndrome [jet lag syndrome] décalage ribosomique [frameshifting, ribosomal] decapoda (crustacea) [decapoda (crustacea)] déchets [waste products] déchets dangereux [hazardous waste] déchets dentaires [dental waste] déchets médicaux [medical waste] déchets radioactifs [radioactive waste] décision médicale non réanimation [resuscitation orders] déclaration Helsinki [Helsinki declaration] décollement rétine [retinal detachment] décompression [decompression] décompression, maladie

60. E-b-5
    research is focused on diseases caused by expansions of CAG repeats encoding polyglutaminestretches, which include hereditary spinocerebellar degenerations.  
    http://www.nig.ac.jp/labs/AR98e/E/E-b-5.html

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