Guillain Barre Syndrome Foundation Discussion Forums Welcome to the Guillain Barre syndrome Foundation Discussion Forums. 0418-2004 0222 AM by Brandy2, Go to last post. miller fisher Variants, 460, 87, http://www.guillain-barre.com/forums/index.php
Abstract millerfisher syndrome is a rare variant of Guillain-Barre syndrome. miller-fisher syndrome is a rare variant of Guillain-Barre syndrome. http://www.ia.pw.edu.pl/~wujek/neurol/str/s2-02-e321.html
Extractions: Miller-Fisher syndrome is a rare variant of Guillain-Barre syndrome. The triad of ophtalmoplegia, ataxia and areflexia characterizes it. We report a case of 46-year old woman with Miller-Fisher syndrome and hepatitis C. First symptom occurred after week from upper respiratory track infection of viral type. First patient noted diplopia, weakness and vertigo, then appeared ophtalmoplegia, ataxia. We didn't noted prolonged latencies and slowing of conduction velocity and there were no abnormalities in CSF examination. During first days of hospitalisation symptoms progressed. In blood examination Enterovirus IgG antibody was found. There was possitive anti-HCV blood test and with coexisting increase of hepatic enzymes we diagnosed hepatitis type C. We included the treatment with gammaglobulin 21g/day during 5 days. We noted slow recovery with ataxia and areflexia firstly and then ophtalmoplegia. Miller-Fisher syndrome is a rare variant of Guillain-Barre syndrome. The triad of ophtalmoplegia, ataxia and areflexia characterizes it. We report a case of 46-year old woman with Miller-Fisher syndrome and hepatitis C. First symptom occurred after week from upper respiratory track infection of viral type. First patient noted diplopia, weakness and vertigo, then appeared ophtalmoplegia, ataxia. We didn't noted prolonged latencies and slowing of conduction velocity and there were no abnormalities in CSF examination. During first days of hospitalisation symptoms progressed. In blood examination Enterovirus IgG antibody was found. There was possitive anti-HCV blood test and with coexisting increase of hepatic enzymes we diagnosed hepatitis type C. We included the treatment with gammaglobulin 21g/day during 5 days. We noted slow recovery with ataxia and areflexia firstly and then ophtalmoplegia.
Guillain-Barre Syndrome millerfisher syndrome The most common subtype is miller-fisher syndrome (MFS), or acute disseminated encephalomyeloradiculopathy, which is characterized by http://www.davidson.edu/academic/psychology/ramirezsite/neuroscience/psy324/jeca
Extractions: In 1859, Jean B.O. Landry, a French physician, described a disorder in which the nerves of the legs, arms, neck, and breathing muscles were paralyzed. Georges Guillain, Jean Alexander Barre, and Andre Strohl discovered the characteristic abnormality of increased protein but normal cell count in the cerebrospinal fluid in 1916. Several names have been given for the syndrome including acute idiopathic polyneuritis, acute idiopathic polyradiculoneuritis, Landrys ascending paralysis, and Guillain Barre syndrome. The causes of Guillain Barre syndrome are unknown. Many researchers theorize that the autoimmune reaction in which macrophages and T-cells attack myelin in the peripheral and cranial nerves is associated with a bacteria or virus, as many cases occur a few days to a few weeks after an infection including the common cold, sore throat, and stomach and intestinal viruses with vomiting and diarrhea. The virus might induce the demyelination via a possible mimicry between the effector virus and a human ganglioside. Salloway and colleagues (1996) discovered that the lipopolysaccharide structure of some strains of Campylobacter jejuni , specifically, the terminal structures of the core oligosaccharide, resemble the human gangliosides GM1 and GD1a. A possible mimicry also exists between the influenza A NS2 protein and a sequence region of the human P2 (myelin) protein thought to be neuritogenic in animals and mitogenic for lymphocytes from patients with GBS. This finding could provide a possible link between the large number of cases of GBS associated with the 1976 USA swine flu vaccination program (Weise and Carnegie, 1988).
GQ1b-Ak The determination of antiGQ1b antibodies is used for determination of the miller-fisher syndrome. Method ELISA. Material needed 5 ml serum. http://www.akh-wien.ac.at/kin/Englische Version/diagnostik/Neurochemie/eGQ1b.htm
Index gbs.org. A collection of resources and information about GuillainBarré, CIDP, miller fisher and other related syndromes. Welcome to gbs.org! http://www.gbs.org/
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