Mad Cow Disease Deer, Elk, get mad cow disease. The disease satisfaction Is it possible to contract a version of mad cow disease from eating deer or elk? Moreover http://home.pacifier.com/~real/madcow.html
Extractions: The disease, which is one of several belonging to a family of disorders called transmissible spongiform encephalopathies (TSE) that literally turns the brain into a sponge. The human form, Creutzfeldt-Jakob Disease (CJD) , occurs in about one/million people over the age of 50 and about one/billion at the age of 30. TSE is thought to be caused by a mysterious particle, a renegade protein called a prion , which, abnormally for proteins, is apparently indestructible - it cannot be destroyed by boiling or burning. When medical instruments contaminated with prions are boiled at high temperatures for 30 minutes, the prions remain infectious When infected materials are incinerated, the ash contains prions, which remain infectious. Funeral homes are refusing to embalm... if an autopsy, which would expose brain tissue, is performed. (My experience from microbiology is that protein will denature at 108 degrees, a relatively low temperature, so that cooking meat well done normally gives protection from protein based diseases. It is truly surprising to me that a protein would survive high temperatures.) People are worried about a version of mad cow disease that is endemic in deer and elk in parts of Colorado and Wyoming, raising the possibility that hunters who eat infected animals might similarly develop a human form of the disease. Health and Wildlife officials are quick to point out that there is no proof that mad deer or elk disease has "jumped" to humans. (No proof could also mean there is no research.)
Mad Cow Disease NOTE Ken Miller and Joe Levine have written two new virtual textbook pages on prions and mad cow disease. Mad Cow Disease appears in the United States. http://www.millerandlevine.com/news/bse/
Extractions: Click to Return to the Home Page of the Dragonfly Book Teacher Resources Looking for teaching ideas on CJD (mad cow)? Here are several lesson plans recommended by the American Association for the Advancement of Science (AAAS) through their Science NetLinks Program: Diseases without Borders - studying the spread of disease across geographical boundaries. ("Students will use the European Union's struggle with Mad Cow Disease as a starting point to study the spread of infectious diseases across geographical boundaries.") Invisible Invaders - Studying the effects of epidemics on different aspects of society ("In this lesson, students research various epidemics that have devastated the world population at different points in history, focusing on the historical events taking place during the times of the epidemics")
Extractions: According to the Center for Disease Control, 76 million Americans every year get food poisoning. If you do the math, that's about 1 in 4 of us every year. You know that "24-hour flu" you had this year? Well, you should know that there's no such thing as a 24-hour flu. You probably got food poisoning. From last year's Listeria and E. coli to a burgeoning antibiotic-resistant salmonella, in today's food safety lottery there's a 1 in 840 chance that we Americans will be hospitalized and a 1 in 55,000 chance that we will die from food-borne illness every year. But E. coli? Easily destroyed by proper cooking. This new super-salmonella threat? Luckily, we still have big-gun antibiotics to kill it. Even bugs like Listeria are pretty wimpy pathogens. After millennia of humans eating the bodies and bodily fluids of other animals, you'd think Mother Nature could cook up a nastier microbe. Well, let's use our imagination. What if there was something in our food supply that wasn't affected by cooking or antibiotics? Something new and undetectable, perhaps?Someultimate pathogen, practically indestructible, evading the immune system and maybe causing some, oh, invariably fatal neurodegenerative disease? Welcome to the world of bovine spongiform encephalopathy (BSE), or mad cow disease.
US Violates WHO Guidelines For Mad Cow Disease Rense.com US Violates WHO Guidelines For mad cow disease A Comparison of North American and European Safeguards By Michael Greger, MD For the Organic Consumers http://www.rense.com/general46/usfup.html
Extractions: The National Cattlemen's Beef Association describes government and industry efforts to safeguard the American public from mad cow disease as "swift," "decisive" and "aggressive." The US Secretary of Agriculture adds "diligent," "vigilant" and "strong." The world's authority on these diseases disagrees. Dr. Stanley Prusiner is the scientist who won the Nobel Prize in Medicine for his discovery of prions, the infectious agents thought to cause bovine spongiform encephalopathy (BSE), or mad cow disease. The word Dr. Prusiner uses to describe the efforts of the U.S. government and the cattle industry is "terrible." What are these "stringent protective measures" that the Cattlemen's Association is talking about, and how do they compare to global standards and internationally recognized guidelines? In 1996, in response to the revelation that young people in Britain were dying from variant Creutzfeldt Jakob Disease (vCJD), the human equivalent of mad cow disease, the World Health Organization (WHO) issued seven "Recommendations." Numbers 5-7 were observations and/or recommendations for further research. The first four recommendations, however, were concrete proscriptions to reduce the likelihood of mad cow disease spreading to human populations. To this day, the United States government continues to violate each and every one of these four guidelines.
Mad Cow Disease mad cow disease. Drawn from other sources as credited.. Click here HARD EVIDENCE LINKING mad cow disease WITH HUMAN DEATHS. Contrary to http://inquirer.gn.apc.org/madcow.html
Extractions: Mad Cow Disease Drawn from other sources as credited.- Click here to read even more about mad cow disease Click here to read about mad cow disease in the United States. Creutzfeld-Jacob Disease used to take decades to kill. Sufferers tended to be over 55. But CJD is changing. Now it kills the young. Can it be a coincidence that, in the last 10 years, we've eaten 1.5 million BSE-infected animals? On Friday morning at 11 o'clock, 20-year-old Peter Hall was buried at Chester-le-Street Parish Church cemetery, near Sunderland, Co Durham. He was killed by Creutzfeldt Jakob Disease (CJD), the human equivalent of Mad Cow Disease (BSE). First striking him at 19, just 13 months ago, it made Peter Hall the fourth British teenager to succumb to the disease to date. Two others died last year-Stephen Churchill, at 19, and an un-named 17-year-old girl. The very first teenage CJD victim, Victoria Rimmer, 16 when she was diagnosed by live biopsy late in 1994, is still in a coma. Government scientists argue that the recent increase in surveillance is the most likely reason why more cases, including teenage cases, are now being identified. But Britain's leading CJD specialist, Dr John Collinge, at St. Mary's Hospital, Paddington, disagrees. 'Given the rarity of CJD in teenagers,' he says, 'It is unlikely that such cases would have been previously missed.' In other words, we're faced with something new. The Government still insists that there is no evidence to link BSE and CJD. Most recently, Health Secretary Stephen Dorrell said that 'there was no conceivable risk' in eating beef. But the facts tell a different story.
Extractions: "The organophosphates interact with manganese by acting as an oxydising agent. They change the manganese from a safe form into a highly lethal free-radical generating form so to get spongiform disease of the new variant type you would need to be simultaneously exposed to these two environmental factors. In the traditional form of the disease one is exposed to a much weaker oxidising agent in the form of ultra violet radiation that is why traditional spongiform disease always starts in the retina in the eye and it occurs in populations that live high up in snow-covered mountains - with UV light is notoriously high. " AND "Avoid use of head lice shampoos and exposure to other forms of organophosphate insecticides. Also be a bit cautious of using mobile phones too intensively because they have an oxydising effect on the brain just like organophosphates. Also avoid living too near to industries that pollute the atmosphere with high levels of manganese such as industries that use manganese as a lead replacement in petrol etc." Mark Purdey, whose information I've posted before, from the UK was on a special report (kind of like 60 minutes) on BBC 2 Sunday night (Correspondent). They did an excellent job. It seems to be about high levels of manganese and organophosphates (or other oxydizing things)
Extractions: Bovine spongiform encephalopathy (BSE) is a progressive neurological disorder of cattle that results from infection by an unconventional transmissible agent. Through the end of November 2003, more than 183,000 cases of BSE were confirmed in the United Kingdom alone in more than 35,000 herds. Regularly updated numbers of reported BSE cases, by country, are available on the website of the Office International Des Epizooties at: http://www.oie.int/eng/info/en_esb.htm The BSE epidemic in the United Kingdom peaked in January 1993 at almost 1,000 new cases per week. The outbreak may have resulted from the feeding of scrapie-containing sheep meat-and-bone meal to cattle. There is strong evidence and general agreement that the outbreak was amplified by feeding rendered bovine meat-and-bone meal to young calves. The nature of the transmissible agent is unknown. Currently, the most accepted theory is that the agent is a modified form of a normal cell surface component known as prion protein. The pathogenic form of the protein is both less soluble and more resistant to enzyme degradation than the normal form.
Extractions: The Web CNN.com Home Page World U.S. Weather ... Special Reports SERVICES Video E-Mail Services CNNtoGO SEARCH Web CNN.com Story Tools LONDON, England (AP) Scientists have dramatically lowered their estimates of how many people are likely to die from the human form of mad cow disease, though experts say much uncertainty remains. Part of the reason the estimate has dropped is because early predictions of worse-case scenarios did not materialize, forcing a shift in calculations. Experts previously estimated that anywhere between a few hundred to 100,000 people in Britain could eventually get the fatal brain-wasting illness, variant Creutzfeldt-Jakob disease. Now, new research published by the Royal Society, Britain's academy of scientists, forecasts that as few as 10 additional people and as many as 7,000 could get the illness by 2080. Variant CJD has killed 132 people so far 122 of them in Britain. Much uncertainty still surrounds the likely course of the epidemic, said James Ironside, director of Britain's national Creutzfeldt-Jakob disease surveillance unit, who was not involved in the research. Since the disease emerged in England in 1996, the task of predicting how many people are likely to get it and when they will get sick has been a struggle because there have been fewer than 150 cases so far and the incubation period is unknown. Scientists believe it may be longer than 30 years.
Extractions: The Web CNN.com Home Page World U.S. Weather ... Special Reports SERVICES Video E-Mail Services CNNtoGO SEARCH Web CNN.com Story Tools (AP) Scientists working with mice have blocked the development of a fatal brain illness resembling mad cow disease a boost for efforts toward finding a treatment for the human version. Mice that get weekly injections of an experimental treatment have remained healthy for more than 500 days so far after getting a dose of the rogue proteins that cause the disease. Untreated mice generally died within about 200 days. The treatment sharply reduced the buildup of the dangerous proteins in the spleen, and the researchers hope that further work will show the same effect in the brain. The results are far from providing a useful treatment for the human version of mad cow disease, called Creutzfeldt-Jakob disease, or CJD. For one thing, the experimental treatment failed when begun in mice that had already developed symptoms. Still, the work indicates that the approach is worth pursuing, researchers from Imperial College London and University College London conclude in Thursday's issue of the journal Nature. "We've made a promising start," said Simon Hawke of Imperial College London. But "there is much more developmental work to be done before we can begin to think about translating this research" to treating people.
Extractions: PLAY VIDEO RELATED Mad cow disease reported in Canada Fear, mystery of mad cow disease HEALTH LIBRARY Health Library Men's Health Women's Health Care for the whole family HUMAN THREAT Mad cow disease was first reported in the United Kingdom in 1986, peaking in 1993 with almost 1,000 new cases per week.
Extractions: PLAY VIDEO RELATED Mad cow disease reported in Canada Fear, mystery of mad cow disease HEALTH LIBRARY Health Library Men's Health Women's Health Care for the whole family THE HUMAN LINK Mad cow disease was first reported in the United Kingdom in 1986, peaking in 1993 with almost 1,000 new cases per week.
The Seattle Times: Mad-cow Disease 2004 Page updated at 0212 am. The first US case of mad-cow disease was detected in a cow from a farm in Mabton, near Yakima, Washington, in December 2003. http://seattletimes.nwsource.com/html/madcowdisease/
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Bovine Spongiform Encephalopathy (eg. BSE) SEE ALSO HORIZON PROGRAMMES BELOW also, for easy reading, try cow to cannibal , which is a journalistic article. Also http://bse.airtime.co.uk/welcome.htm
Extractions: "ABSENCE OF EVIDENCE IS NOT THE SAME AS EVIDENCE OF ABSENCE" - MJ REES For latest news and updates see www.priondata.org Major increase in addresses and emails of worldwide researchers into TSEs It is important that everyone checks their address, email and direction of research. This a major list used to invite people to meetings
Extractions: Transmissible spongiform encephalopathies (TSEs) are a family of diseases of humans and animals characterized by spongy degeneration of the brain with severe and fatal neurological signs and symptoms. In animals, scrapie is a common disease in sheep and goats. Mink and North American mule deer and elk can contract TSEs. Bovine spongiform encephalopathy (BSE) is also a TSE, affecting a number of species (cattle, human, cats, some types of animals in 300 settings). BSE is a transmissible, neuro-degenerative fatal brain disease of cattle. The disease has a long incubation period of 4-5 years and it is fatal for cattle within weeks to months of its onset. The nature of the BSE agent is still being debated. Strong evidence currently available supports the theory that the agent is composed largely, if not entirely, of a self-replicating protein, referred to as a prion. It is transmitted through the consumption of BSE-contaminated meat and bone meal supplements in cattle feed. FOR MORE INFORMATION WHO fact sheet on BSE
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String Breaker Home Page The String Breaker Homepage has moved The String Breaker is a hard rock/alternative/grunge band from Pittsburgh. This page contains http://www.angelfire.com/pa/madcowdisease/