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Hyperparathyroidism: more books (71)

The 'sentinel clot' sign in spontaneous retropharyngeal hematoma secondary to parathyroid apoplexy.: An article from: Ear, Nose and Throat Journal by George Koulouris, Marcus Pianta, et all 2006-09-01
Hyperparathyroid-related cognitive dysfunction improves with surgery.(Metabolic Disorders): An article from: Family Practice News by Bruce Jancin, 2007-03-15
Gale Encyclopedia of Medicine: Parathyroid hormone test by Janis O. Flores, 2002-01-01
Postop PTH test unnecessary when calcium is normal.(Metabolic Disorders)(parathyroid hormone): An article from: Family Practice News by Jeff Evans, 2006-05-01
Cinacalcet lowers PTH and calcium in dialysis patients.(parathyroid hormone ): An article from: Internal Medicine News by Steve Perlstein, 2004-07-15
Audio-digest: General Surgery: Management of Thyroid Diseases (Vol. 49, Issue 13, July 7, 2002) by MD Jon A. Heerden, 2002
Cost-effective workup for kidney stones (Postgraduate Medicine) by MD Neil Baum, 2010-06-21
Surgery of the Thyroid and Parathyroid Glands (Clinical Surgery International) (No.6)
The logic of parathyroid surgery and the viscerovertebral angle by Louis T Tenta, 1987

lists with details

PetPlace.com - Article: Nutritional Secondary Hyperparathyroidism
Easy to scan format allows you to quickly identify topics of interest. NutritionalSecondary hyperparathyroidism, by Dr. Bari Spielman. Overview.
http://www.petplace.com/articles/artshow.asp?artID=4469
Hyperparathyroidism
hyperparathyroidism. Definition hyperparathyroidism is excessiveproduction of parathyroid hormone by the parathyroid glands.
http://www.healthscout.com/ency/article/001215.htm

Extractions: Causes, incidence, and risk factors: The parathyroid glands are located at the front and base of the neck around the thyroid gland. They produce parathyroid hormone , which regulates calcium, phosphorus, and magnesium levels within the blood and bone. When calcium levels are too low, the body responds by increasing production of parathyroid hormone. This increase in parathyroid hormone causes more calcium to be taken from the bone and more calcium to be reabsorbed by the intestines and kidney. When the cacium level returns to normal, parathyroid hormone production slows down. Excess parathyroid hormone can be produced for a variety of reasons. In some cases, parathyroid hormone is produced without regard to the calcium levels. This is called "primary hyperparathyroidism" and is caused by enlargement of one or more of the parathyroid glands. It is a common disorder affecting about 1 in 1000 people. The high levels of calcium and parathyroid hormone affect several body systems, including the skeletal, gastrointestinal
Hyperparathyroidism
hyperparathyroidism. Frederick Kaplan, MD Professor of Orthopedic MolecularMedicine University of Pennsylvania Philadelphia, Pennsylvania.
http://www.ortho.hyperguides.com/Tutorials/basic_science/hyperpara/default.asp
MayoClinic.com - Hyperparathyroidism
hyperparathyroidism By Mayo Clinic staff Overview Sometimes, however, the glandsproduce too much PTH a condition known as hyperparathyroidism.
http://www.mayoclinic.com/invoke.cfm?id=DS00396
BioSpace News: Hyperparathyroidism
Find DiseaseSpecific News BioSpace News hyperparathyroidism. Abgenix(ABGX) Begins Phase 1 Study Of ABX-PTH In Patients With Secondary
http://www.biospace.com/news_rxtarget.cfm?RxTargetID=111
Uhrad.com - Musculoskeletal Imaging Teaching Files
Case Sixty Five Secondary hyperparathyroidism Related to Renal Osteodystrophy. DiagnosisSecondary hyperparathyroidism related to renal osteodystrophy.
http://www.uhrad.com/msiarc/msi065.htm

Extractions: Click On Images for Enlarged View Clinical History: Hand and low back pain in a patient on dialysis. Findings: A radiograph of both hands and an AP radiograph of the spine were obtained. The radiograph of the hand demonstrates osteopenia. Multiple lytic and somewhat expansile lesions with well-defined borders, that are in part sclerotic, are present bilaterally. These lesions are present in the distal aspects of the metacarpals and in the proximal, mid and distal phalanges. Subperiosteal resorption is present along the radial aspect of the proximal and mid phalanges bilaterally involving the first through fifth fingers. Generalized thinning of the cortices is present of all the metacarpals suggestive of endosteal bone resorption. The radiograph of the spine demonstrates extensive erosions of both SI joints, which are widened bilaterally and symmetrically. Diagnosis: Secondary hyperparathyroidism related to renal osteodystrophy.
A To Z Encyclopedia Topic: Hyperparathyroidism
hyperparathyroidism. What is hyperparathyroidism? hyperparathyroidism is caused byoveractive parathyroid glands. hyperparathyroidism is quite rare in children.
http://web1.tch.harvard.edu/cfapps/A2ZtopicDisplay.cfm?Topic=Hyperparathyroidism
Hyperparathyroidism
hyperparathyroidism, At least 50% of patients with primary hyperparathyroidismhave no symptoms, and approximately 1% of cases go undiagnosed.
http://www.ivillagehealth.com/library/onemed/content/0,,241012_245636,00.html

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Diabetes & Other Endocrine And Metabolic Disorders - Hyperparathyroidism
hyperparathyroidism. What is hyperparathyroidism? hyperparathyroidism is causedby overactive parathyroid glands. What causes hyperparathyroidism?
http://www.musckids.com/health_library/diabetes/hpt.htm

Extractions: Print Version Hyperparathyroidism is caused by overactive parathyroid glands. Overactive parathyroid glands produce high levels of parathyroid hormones, which, in turn, results in increased levels of calcium in the blood stream. The excess calcium released by the bones leads to osteoporosis and osteomalacia (both bone-weakening diseases). Another result of hyperparathyroidism is kidney stones, because of high levels of calcium excreted into the urine by the kidneys. Hyperparathyroidism is quite rare in children. Causes of hyperparathyroidism include benign (non-cancerous) tumors on the parathyroid glands or enlargement of the parathyroid glands. According to a recent study, children with hyperparathyroidism experience more severe symptoms than adults. The following are the most common symptoms of hyperparathyroidism in children. However, each child may experience symptoms differently. Symptoms may include: The symptoms of hyperparathyroidism may resemble other conditions or medical problems. Always consult your child's physician for a diagnosis.
Diabetes & Other Endocrine And Metabolic Disorders - Hyperparathyroidism
here. hyperparathyroidism. What is hyperparathyroidism? hyperparathyroidismis caused by overactive parathyroid glands. Overactive
http://www.chkd.org/Diabetes/Hpt.asp

Extractions: Adolescent Medicine Allergy/Immunology Anesthesiology Arthritis Burns Cardiology Craniofacial Dental Medicine Dermatology Developmental Peds Diabetes Digestive Ear, Nose, Throat Genetics Gastroenterology Growth Hematology High Risk Newborn High Risk Pregnancy Infectious Disease Mental Health Neonatology Nephrology Neurology Normal Newborn Normal Pregnancy Oncology Ophthalmology Orthopedics Otolaryngology Pediatric Intensive Care Pediatric Surgery Pediatrics Physical Medicine Plastic Surgery Respiratory/Pulmonology Rheumatology Safety Surgery Terminal Transplant Urology Site Search Hyperparathyroidism is caused by overactive parathyroid glands. Overactive parathyroid glands produce high levels of parathyroid hormones, which, in turn, results in increased levels of calcium in the blood stream. The excess calcium released by the bones leads to osteoporosis and osteomalacia (both bone-weakening diseases). Another result of hyperparathyroidism is kidney stones, because of high levels of calcium excreted into the urine by the kidneys. Hyperparathyroidism is quite rare in children. What causes hyperparathyroidism?
Hyperparathyroidism - Lucile Packard Children's Hospital
hyperparathyroidism What is hyperparathyroidism? hyperparathyroidism is caused byoveractive parathyroid glands. hyperparathyroidism is quite rare in children.
http://www.lpch.org/DiseaseHealthInfo/HealthLibrary/diabetes/hpt.html

Extractions: Hyperparathyroidism is caused by overactive parathyroid glands. Overactive parathyroid glands produce high levels of parathyroid hormones, which, in turn, results in increased levels of calcium in the blood stream. The excess calcium released by the bones leads to osteoporosis and osteomalacia (both bone-weakening diseases). Another result of hyperparathyroidism is kidney stones, because of high levels of calcium excreted into the urine by the kidneys. Hyperparathyroidism is quite rare in children.
Wheeless' Textbook Of Orthopaedics
Main Menu Home Page hyperparathyroidism Primary hyperparathyroidism changingpatterns in presentation and treatment decisions in the eighties. Rao DS.
http://www.wheelessonline.com/05/304.htm

Extractions: Main Menu Home Page - See: - Renal Osteodystrophy Remodeling of Bone - Discussion: - a disorder which is caused by excessive production of parathyroid hormone which leads to hypercalcemia causes: - in most cases is due to single parathyroid adenoma (80% of patients); - malignant tumor: occurs in about 1% of patients with hyperparathyroidism; - occurs often in association w/ multiple endocrine neoplasia syndrome, and rarely to parathyroid carcinoma; - hyperparathyoidism is sometimes seen in renal cell carcinoma and squamous cell carcinoma; - diff dx : - occult tumor; - multiple myeloma (often associated w/ hypercalcemia); - classification: - primary defect of the parathyroid gland w/ hypersecretion of PTH as seen w/ adenoma's of the parathyroid gland; - secondaray causes arise from conditions that produces abnormally low ionic plasma Ca levels and thereby stimulates production of PTH (see renal disease: effects on bone ): - tertiary conditions in which PTH secretion has become autonomous after prolonged stimulation of gland owing to secondary parathyroidism; - references: - Primary hyperparathyroidism: incidence, morbidity, and potential economic impact in a community. Heath H III, Hodgson SF, Kennedy MA. N Engl J Med 1980;302:189-93. - Pathology of the parathyroids in hyperparathyroidism: discussion of recent advances in the anatomy and pathology of the parathyroid glands. Roth SI. Arch Pathol 1962;73:495-510. - Recent advances in parathyroid gland pathology. Roth SI. Am J Med 1971;50:612-22. - Primary hyperparathyroidism: changing patterns in presentation and treatment decisions in the eighties. Rao DS. Henry Ford Hosp Med J 1985;33:194-7.
Endocrinology - Hyperparathyroidism
hyperparathyroidism What is hyperparathyroidism? What are symptoms of hyperparathyroidism?The following are the most common symptoms of hyperparathyroidism.
http://www.muschealth.com/endocrin/hyperpar.htm

Extractions: Hyperparathyroidism is caused by overactive parathyroid glands. Overactive parathyroid glands produce too much parathyroid hormones, which in turn stimulate increased levels of calcium in the blood stream. The excess calcium released by the bones leads to osteoporosis and osteomalacia (both bone-weakening diseases). Other results of hyperparathyroidism are kidney stones, because of high levels of calcium excreted into the urine by the kidneys.
SMJ June 1996 - Intravenous Pamidronate For Hypercalcemia...
Intravenous Pamidronate for Hypercalcemia of Primary hyperparathyroidism. Thisis a welldescribed consequence of hyperparathyroidism.
http://www.sma.org/smj/96jun20.htm

Extractions: Intravenous Pamidronate for Hypercalcemia of Primary Hyperparathyroidism ALEXANDER TAL, MD, and LELAND GRAVES, MD, Kansas City, Mo ABSTRACT: Intravenous pamidronate disodium has been used successfully in the treatment of malignancy-associated hypercalcemia and Paget's disease of bone. Although the definitive treatment of primary hyperparathyroidism (PHPT) is surgical, intravenous pamidronate has been used to treat hypercalcemia of PHPT when surgery was contraindicated. We report two cases of PHPT in which intravenous pamidronate effectively reduced hypercalcemia and significantly improved the clinical symptoms. The results of our study and the experience in the literature suggest that pamidronate is a safe and effective method of reducing serum calcium in patients with PHPT when definitive surgery is contraindicated or must be postponed. The new generation of bisphosphonates, including pamidronate (aminohydroxypropylidene diphosphonate [APD]) and clod-ronate (dichloromethylene diphosphonate [Cl2MBP]), are potent inhibitors of osteoclastic bone resorption. In recent years, excellent results have been reported from treatment of malignancy-associated hypercalcemia with bisphosphonates. Bisphosphonates have also been used to treat hypercalcemia of PHPT, but the results reported are variable. We report on two patients with PHPT who were treated with pamidronate for severe hypercalcemia, and we review the relevant published experience with pamidronate and other bisphosphonates in PHPT.
Hyperparathyroidism, Secondary
hyperparathyroidism, Secondary. Image URL Nutritional secondary hyperparathyroidism,Image URL Nutritional secondary hyperparathyroidism,
http://www.brisbio.ac.uk/ROADS/subject-listing/hyperparathyroidismsecondary.html
InteliHealth Forums Disease And Conditions Hyperparathyroidism
Home Disease And Conditions hyperparathyroidism, Not logged in. Login inor create an account. Has anyone had a diagnosis of hyperparathyroidism?
http://community.intelihealth.com:8081/jive/InteliHealth/viewThread.jsp?forum=3&
»»Hyperparathyroidism Reviews««
hyperparathyroidism Reviews. Related Subjects HormoneReplacement-TherapyMore Pages hyperparathyroidism Page 1 2. Search for a Health Issue Book.
http://www.health-issue-books.com/Hormone-Replacement-Therapy/Hyperparathyroidis

Extractions: More Pages: Hyperparathyroidism Page 1 Book reviews for "Hyperparathyroidism" sorted by average review score: Current Controversy in Parathyroid Operation and Reoperation (Medical Intelligence Unit) Published in Hardcover by R G Landes Co (November, 1994) Authors: Goran Akerstrom and Claes Juhlin Amazon base price: Average review score: A good review of a difficult subject There are not many suitable books on the market that deal with the issues involved in parathyroid disease and surgery. If you are looking for a text book telling you how to deal with parathyroid disease surgically this book is not the one (although no book should be). However, for an overview of a complex subject covering a wide range of clinical situations, this book is excellent. I strongly recommend it to any parathyroid surgeon. Bone densitometry patients with asymptomatic primary hyperparathyroidism (SuDoc HE 20.6512/7:995/6) Published in Unknown Binding by U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research May be purchased from the National Technical Information Service (1995) Author: U.S. Dept of Health and Human Services
Cinacalcet Helpful In Uncontrolled Secondary Hyperparathyroidism
Cinacalcet Helpful in Uncontrolled Secondary hyperparathyroidism CME.News Author Laurie Barclay, MD CME Author Désirée Lie, MD
http://www.medscape.com/viewarticle/473016
MJA: Edis, Hyperparathyroidism: What Does Surgery Have To Offer?
hyperparathyroidism what does surgery have to offer? Surgery for primaryhyperparathyroidism 19621996 indications and outcomes.
http://www.mja.com.au/public/issues/feb16/edis/edis.html

Extractions: Home Issues Classifieds Contact ... Search Even asymptomatic patients should be considered for surgery MJA The report by Delbridge et al in this issue of the Journal describes the changing clinical picture of primary hyperparathyroidism (PHPT) in patients referred for surgical treatment at Royal North Shore Hospital, Sydney, over the past 30 years. The most common presentation in the 1990s is in women over the age of 50 years with bone loss indistinguishable from that of postmenopausal or senile osteoporosis (31%). Patients with kidney stones still constitute 15%, and those with subtle but distressing symptoms of fatigue, depression or altered cognitive function constitute 20%. Delbridge et al have also observed an "exponential" increase in the number of cases referred for surgery over the past decade, reflecting contemporary epidemiological studies which have unearthed a virtual epidemic of PHPT among postmenopausal women it affects 2% to 3% of women in this age group. Even so, the absolute number of patients undergoing operation at Royal North Shore Hospital each year remains small when compared with the number expected by extrapolation from this prevalence. Presumably, this is due in part to underdiagnosis of PHPT, as well as continuing reluctance among physicians to refer patients for surgery unless they have marked hypercalcaemia or overt "bone or stone" disease.
Hyperparathyroidism
hyperparathyroidism. Definition. Primary hyperparathyroidism Specialistsin Primary hyperparathyroidism. Endocrinologists, nephrologists
http://www.osteoporosis.org.za/excerpt5.htm

Extractions: Member Society HYPERPARATHYROIDISM Primary hyperparathyroidism is a metabolic disorder that can result in the loss of bone. It occurs when one or more parathyroid glands become enlarged and overactive, producing too much parathyroid hormone. Hyperparathyroidism increases bone remodeling and therefore magnifies the imbalance between bone formation and resorption seen after the menopause, leading to a greater rate of bone loss. Women outnumber men by two to one and the frequency of the condition increases with age. Parathyroid hormone (PTH) regulates calcium metabolism and when too much PTH is released it causes a rise in blood levels of calcium. The most common symptoms of hypercalcaemia are loss of appetite, thirst, urinary frequency, lethargy, fatigue, muscle weakness, joint pains and constipation. More severe symptoms of higher levels may include nausea and vomiting , abdominal pain, memory loss and depression. Primary hyperparathyroidism may be associated with an increased risk of vertebral crush fractures.

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