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Hyperhomocysteinemia:     more detail

Hyperhomocysteinemia: Webster's Timeline History, 1992 - 2007 by Icon Group International, 2009-02-20 Hyperhomocysteinemia as a result of the methylenetetrahydrofolate reductase (MTHFR) C677T polymorphism causes an increased risk of cerebrovascular disease: ... An article from: Original Internist by Robert A., Jr. Duca, 2010-09-01 Hyperhomocysteinemia in end stage renal disease: is treatment necessary? (Continuing Education).: An article from: Nephrology Nursing Journal by Jennifer Snavely, 2002-04-01

lists with details

41. Entrez PubMed
    hyperhomocysteinemia, atherosclerosis and thrombosis. hyperhomocysteinemia/epidemiology; hyperhomocysteinemia/genetics; Male; Methionine/metabolism;  
    http://genomebiology.com/pubmed/10063987
    
    Extractions: Summary Brief Abstract Citation ASN.1 MEDLINE XML UI List LinkOut Related Articles Cited in Books CancerChrom Links Domain Links 3D Domain Links GEO DataSet Links Gene Links Genome Links GEO Links HomoloGene Links Nucleotide Links OMIM Links PMC Links Cited in PMC PopSet Links Protein Links SNP Links Structure Links UniSTS Links Show: Sort Author Journal Pub Date Text File Clipboard E-mail Order

42. ACSH > Search > Page Not Found
    hyperhomocysteinemia HeartDisease Risk Factor-or New Red Herring? Therefore, some researchers deduced that hyperhomocysteinemia is causally related to CVD.  
    http://www.acsh.org/publications/priorities/1102/hyper.html

43. Hyperhomocysteinemia And Thrombosis
    top. 1.4.2 Abstract Abundant experimental investigation, clinical observation, and epidemiological evidence have confirmed hyperhomocysteinemia (HC) as a risk  
    http://www.intervencionismosidi.com/revistas/may03/may03_1.4.html

44. Elixirhealth.co.uk - Detox, Slimming, Herbal Supplements, Juicers, Vitamins & Mi
    hyperhomocysteinemia Breaking the term hyperhomocysteinemia into its roots does much to clarify this disease hyper (too much) homocysteine.  
    http://www.elixirhealth.co.uk/asps/ailment-encyclopaedia-details.asp?id=496

45. Quest Health Library .com - Health Conditions, Vitamins, Minerals, Supplements,
    hyperhomocysteinemia. DESCRIPTION. Breaking the term hyperhomocysteinemia into its roots does much to clarify this disease hyper (too much) homocysteine.  
    http://www.questhealthlibrary.com/health_conditions/hyperhomocysteinemia
    
    Extractions: Breaking the term "hyperhomocysteinemia" into its roots does much to clarify this disease - hyper (too much) homocysteine. Normally, homocysteine, a modified amino acid, is formed as a by-product of a natural chemical conversion within the body. Because homocysteine is actually a toxin, the body has specific mechanisms to keep homocysteine levels controlled. When these mechanisms fail to work, homocysteine increases in the blood, thereby scarring artery walls and encouraging plaque formation in the arteries. An elevated homocysteine levels is becoming recognised as a risk factor for heart disease. Forty percent of all Coronary Heart Disease patients show evidence of hyperhomocysteinemia. Recently, elevated homocysteine levels have been linked to cancer and systemic Lupus erythematosus Many hyperhomocysteinemia patients exhibit low levels of Folic Acid , vitamin and/or vitamin . However, the most severe form becomes evident in infancy and could lead to premature Aging and death. This form is typically genetically acquired, although researchers have said that

46. Entrez PubMed
    Click here to read hyperhomocysteinemia an independent risk factor or a simple marker of vascular disease?. 1. Basic data Article  
    http://www.biomedcentral.com/pubmed/12801808
    
    Extractions: Summary Brief Abstract Citation ASN.1 MEDLINE XML UI List LinkOut Related Articles Cited in Books CancerChrom Links Domain Links 3D Domain Links GEO DataSet Links Gene Links Genome Links GEO Links HomoloGene Links Nucleotide Links OMIM Links PMC Links Cited in PMC PopSet Links Protein Links SNP Links Structure Links UniSTS Links Show: Sort Author Journal Pub Date Text File Clipboard E-mail Order

47. Entrez PubMed
    Click here to read hyperhomocysteinemia an independent risk factor or a simple marker of vascular disease? 2. Epidemiological  
    http://www.biomedcentral.com/pubmed/12801809
    
    Extractions: Summary Brief Abstract Citation ASN.1 MEDLINE XML UI List LinkOut Related Articles Cited in Books CancerChrom Links Domain Links 3D Domain Links GEO DataSet Links Gene Links Genome Links GEO Links HomoloGene Links Nucleotide Links OMIM Links PMC Links Cited in PMC PopSet Links Protein Links SNP Links Structure Links UniSTS Links Show: Sort Author Journal Pub Date Text File Clipboard E-mail Order

48. Symptoms Library :: Hyperhomocysteinemia
    hyperhomocysteinemia. DESCRIPTION Breaking the term hyperhomocysteinemia into its roots does much to clarify this disease hyper (too much) homocysteine.  
    http://www.discountvitaminsandherbs.com/library_symptoms_hyperhomocysteinemia.ph
    
    Extractions: Breaking the term "hyperhomocysteinemia" into its roots does much to clarify this disease - hyper (too much) homocysteine. Normally, homocysteine, a modified amino acid, is formed as a by-product of a natural chemical conversion within the body. Because homocysteine is actually a toxin, the body has specific mechanisms to keep homocysteine levels controlled. When these mechanisms fail to work, homocysteine increases in the blood, thereby scarring artery walls and encouraging plaque formation in the arteries. An elevated homocysteine levels is becoming recognised as a risk factor for heart disease. Forty percent of all Coronary Heart Disease patients show evidence of hyperhomocysteinemia. Recently, elevated homocysteine levels have been linked to cancer and systemic Lupus erythematosus. Many hyperhomocysteinemia patients exhibit low levels of Folic Acid Vitamin B6 and/or Vitamin B12 . However, the most severe form becomes evident in infancy and could lead to premature Aging and death. This form is typically genetically acquired, although researchers have said that

49. Hyperhomocysteinemia Jako Czynnik Ryzyka Udaru Mózgu
    hyperhomocysteinemia jako czynnik ryzyka udaru mózgu. Bozena Adamkiewicz, Andrzej Klimek. Oddzial Neurologiczny WSS im. M. Kopernika. Abstract.  
    http://www.neurologiapolska.pl/streszczenia/aaa5.htm
    
    Extractions: Hyperhomocysteinemia jako czynnik ryzyka udaru mózgu Bo¿ena Adamkiewicz, Andrzej Klimek Oddzia³ Neurologiczny WSS im. M. Kopernika Abstract Hyperhomocysteinemia as a risk factor of stroke Since 1995 hyperhomocysteinemia (hyperHcy) is consider as a new, independent risk factor of vascular diseases. The level of Hcy was performed among 100 patients with ischaemic stroke confirmed by CT scan/MRI to aprove this hypothesis.The rutine lab tests were undertaken. The control group was consist of 20 healthy patients. In the first group the level of Hcy was statistical different and higher than in control. In 17 patients Hcy was elevated, the half of them were over 65 year-old. 30 % of patients had advanced arteriosclerotic process. In the patient with hyperHcy the prevalence of DM was higher, among the elderly with hyperHcy hyperlipidemia or IHD. Authors on the base of this results note that hyperHcy should be treated as the independent risk factor of ischaemic stroke and hyperHcy corelate with intensity of arteriosclerosis. Streszczenie Oddzia³ Neurologiczny WSS im. M. Kopernika

50. Hyperhomocysteinemia And Hemorrhagic Strokes
    Ask the Experts about General Critical Care. from Medscape Critical Care. hyperhomocysteinemia and Hemorrhagic Strokes. Question Is  
    http://www.medscape.com/viewarticle/441502

51. Hypercoagulability Clinical Assessment And Treatment
    hyperhomocysteinemia. Folic acid deficiency or methylenetetrahydrofolate reductase (MTHFR) deficiency are also causes of hyperhomocysteinemia.  
    http://www.medscape.com/viewarticle/415086_7

52. Entrez PubMed
    Click here to read hyperhomocysteinemia and traditional cardiovascular disease risk factors in endstage renal disease patients on dialysis a case-control  
    http://www.lipidworld.com/pubmed/7605381
    
    Extractions: Summary Brief Abstract Citation ASN.1 MEDLINE XML UI List LinkOut Related Articles Cited in Books CancerChrom Links Domain Links 3D Domain Links GEO DataSet Links Gene Links Genome Links GEO Links HomoloGene Links Nucleotide Links OMIM Links PMC Links Cited in PMC PopSet Links Protein Links SNP Links Structure Links UniSTS Links Show: Sort Author Journal Pub Date Text File Clipboard E-mail Order

53. Entrez PubMed
    Click here to read hyperhomocysteinemia predicts cardiovascular outcomes in hemodialysis patients. Mallamaci F, Zoccali C, Tripepi  
    http://www.lipidworld.com/pubmed/11849403
    
    Extractions: Summary Brief Abstract Citation ASN.1 MEDLINE XML UI List LinkOut Related Articles Cited in Books CancerChrom Links Domain Links 3D Domain Links GEO DataSet Links Gene Links Genome Links GEO Links HomoloGene Links Nucleotide Links OMIM Links PMC Links Cited in PMC PopSet Links Protein Links SNP Links Structure Links UniSTS Links Show: Sort Author Journal Pub Date Text File Clipboard E-mail Order

54. Legumeworld
    Translate this page Folate, Embarazo y hyperhomocysteinemia. Folate y Embarazo ¿Que es el Folate? hyperhomocysteinemia Lo que NO come, puede perjudicarle.  
    http://www.legumechef.com/beneficios9_sp.htm

55. French Text Hyperhomocysteinemia
    Translate this page à partir des mots clés MeSH (vedettes-matières médicales) «homocysteine» (homocystéine), «hyperhomocysteinemia» (hyperhomocystéinémie), «methionine  
    http://www.ctfphc.org/French_Text/Hyperhomocysteinemia.htm
    
    Extractions: JAMC PDF Objectif : Options : et B et B de source alimentaire. Preuves : Valeurs : Recommandations : et B Validation : Commanditaires : Introduction La Figure 1 e ou 95 e homocysteine hyperhomocysteinemia methionine arteriosclerosis myocardial ischemia folic acid vitamin B vitamin B pyridoxine Annexe 1 Les , de vitamine B et de folates. Lien l'hypertension, ou d'angine de novo Selon l'essai clinique MRFIT, Tableau 1 Shunt Occlusion Trial Lien mais non chez les patients de race blanche. Lien ou de vitamine B et le risque d'accident coronarien n'ont aucun lien avec la maladie coronarienne; en revanche, la vitamine B Effet du traitement vitaminique , et non de vitamine B Les doses moindres, de 0,1 mg par exemple, semblent inefficaces. Si la vitamine B , de vitamine B ou d'autres micronutriments. Ainsi, la recommandation actuelle de la Food and Drug Administration n de la maladie coronarienne pourrait influer sur la survenue des accidents coronariens majeurs. Selon la Nurses' Health Study (25 mg) et de vitamine B Recommandations Tableau 2 et de vitamine B de source alimentaire ( Tableau 3 ou d'ajouter des doses plus fortes de vitamine B Recommandations d'autres groupes Les Directives de l' American Heart Association Les Tableau 4 r r D r D r Membres : D rs Personnes-ressources : Cet article a fait l'objet d'un examen par les pairs.

56. CTF Selected References: Hyperhomocysteinemia
    Selected References. Preventive Health Care, 2000 Update Screening and Management of hyperhomocysteinemia for the Prevention of Coronary Artery Disease Events.  
    http://www.ctfphc.org/References/Homocyst_bib.html
    
    Extractions: Prepared by Gillian L. Booth, MD, Departments of Medicine, Clinical Epidemiology and Health Care Research Program, University of Toronto, Elaine E. L. Wang, MD, Departments of Pediatrics, Clinical Epidemiology and Health Care Research Program, University of Toronto These recommendations were finalized by the Task Force in July 1999 Statistics Canada. Causes of death, 1995 (Catalogue 84-208-XPB). 1997. Ottawa, Minister of Industry. Brophy JM. The epidemiology of acute myocardial infarction and ischemic heart disease in Canada. data from 1976 to 1991. Can J Cardiol 1997;13:474-478. Skovby F. Inborn errors of metabolism causing homocysteinemia and related vascular involvement. Haemostasis 1989;19(suppl 1):4-9. Mudd SH, Skovby F, Levy HL, Pettigrew KD, Wilcken B, Pyeritz RE, Andria G, Boers GHJ, Bromberg IL, Cerone R, Fowler B, Gröbe H, Schmidt H, Schweitzer L. The natural history of homocystinuria due to cystathione-synthase deficiency. Am J Hum Genet 1985;37:1-31. Moghadasian MH, McManus BM, Frohlich JJ. Homocyst(e)ine and coronary artery disease. Clinical evidence and genetic and metabolic background. Arch Intern Med 1997;157:2299-2308.

57. Hyperhomocysteinemia Due To Folate Deficciency
    Danish Medical Bulletin No. 1. February 2004. Vol. 51 Page. Hyperhomocysteinaemia due to folate deficiency. Impact on haemostasis and vascular biology.  
    http://www.danmedbul.dk/DMB_2004/0104/0104-phd/DMB3561.htm
    
    Extractions: due to folate deficiency Impact on haemostasis and vascular biology Liselotte M. Sabroe Ebbesen PDF This PhD dissertation was accepted by the Faculty of Health Sciences of the University of Aarhus, and defended on September 19, 2003. Dan Med Bull 2004;51:129. Hyperhomocysteinaemia (HH) has been identified as a risk factor for arterial and venous thrombosis in epidemiologic studies, although the pathogenesis is still largely unresolved. The aim was to evaluate the influence of HH on the whole blood coagulation profile (WBCP), the genetic regulation of blood cells and on balloon-induced neointima formation. Treatment of HH rats with the folate containing diet reversed the coagulation changes, as estimated by WBCP analyses and single coagulation factor functions. Indication of increased numbers of GpIIb/IIIa receptors was found in HH animals due to saturation kinetics as measured by WBCP following blockage of the platelet GpIIb/IIIa receptor. Up-regulation of uPAR, L-, E- and P-selectins was found and may contribute to increased tethering, rolling of leukocytes and migration of these to the vessel wall. A reduced amount of neointima was found in HH animals following endothelial cell denudation. This may be due to a reduced methylation capacity related to the severeness of folate depletion in experiment 1 animals.

58. Diabetes Care: Hyperhomocysteinemia And Macroangiopathy In Type 2 Diabetes
    Print friendly Tell a friend Find subscription deals hyperhomocysteinemia and Macroangiopathy in Type 2 Diabetes Diabetes Care, June, 2001.  
    http://articles.findarticles.com/p/articles/mi_m0CUH/is_6_24/ai_76610128
    
    Extractions: Elevated homocysteine is widely regarded as an independent risk factor for macrovascular disease, particularly in patients with type 2 diabetes (1). The increased prevalence of macroangopathy in type 2 diabetes is further demonstrated by Buysschaert et al. [2]. The authors found a significant increase in the prevalence of macroangiopathy in type 2 diabetic patients with hyperhomocysteinemia. Their data, however, should not be overinterpreted because the etiological role of homocysteine in atherosclerosis in the presence of macroangiopathy is far from clear [3]. First, the metabolism of homocysteine is altered for a period of several days following acute ischemic events, such as myocardial infarction [4] and stroke [5], as a result of an increased release from damaged tissues. This is caused by methylation of DNA, RNA, and various proteins, leading to an increase in S-adenosylhomocysteine and subsequently to increased homocysteine production [3]. Furthermore, nephropathy may reduce homocysteine clearance in type 2 diabetic patients. To our knowledge, there are no published studies examining the time course of homocysteine concentrations following acute ischemia, which may be silent. Hence, assessing homocysteine levels in patients with preexistent macroangiopathy may produce misleading results. This may in part account for the conflicting results regarding the association of homocysteine levels with insulin resistance that have been reported in various studies [6].

59. Diabetes Care: Response To Chan Et Al.: Hyperhomocysteinemia And Macroangiopathy
    Print friendly Tell a friend Find subscription deals Response to Chan et al. hyperhomocysteinemia and macroangiopathy in type 2 diabetes Diabetes Care, June  
    http://articles.findarticles.com/p/articles/mi_m0CUH/is_6_24/ai_76610129
    
    Extractions: We thank Chan et al. [1] for their interest in our work [2] and their relevant comments. We agree that there is still much to be learned about the relationship between homocysteine and atherosclerosis, as was recently stressed by Christen et al. [3]. We also agree that homocysteine metabolism is altered for a few days after acute ischemic events, as reported by Egerton et al. [4] and Lindgren et al. [5]. Therefore, patients with acute cardiovascular events were excluded from our analysis and could not interfere with the interpretation of our results. Chan et al. [1] pertinently mention the role of fibrates and metformin as potential confounders of homocysteine concentrations. Our data in type 2 diabetic patients confirm that fibrates significantly increased homocysteine levels, whereas there was no influence from metformin treatment.

60. Project: Behandeling Van Patiënten Met Veneuze Trombose En Hyperhomocysteinemia
    entire NIWI site (nl), Help. Project Behandeling van patiënten met veneuze trombose en hyperhomocysteinemia om herhaling van trombose te voorkomen.  
    http://www.niwi.knaw.nl/nl/oi/nod/onderzoek/OND1263833/toon
    
    Extractions: Login NIWI (nl) Onderzoek Informatie NOD - Nederlandse Onderzoek Databank ... Powered by from "Project: Behandeling van..." entire NIWI site (nl) Titel-Eng Treatment with vitamins of patients with venous thrombosis and hyperhomocysteinemia to prevent recurrent thrombosis. Samenvatting Hyperhomocysteinemia is een risicofactor voor trombose en herhaalde trombose. Verhoogde homocysteine spiegels kunnen genormaliseerd worden met behulp van de B-vitamines foliumzuur, B6 en B12. In de huidige studie wordt het klinische effect van homocysteine verlagende therapie onderzocht, hetgeen nog nooit is bestudeerd. Hiervoor worden ongeveer 5000 patiënten met trombose op hun homocysteine gehalte in bloed gescreend en vervolgens wordt met de hyperhomocysteinemische patiënten een dubbel-blinde gerandomiseerde placebo-gecontroleerde studie uitgevoerd. Het eindpunt is herhaling van de trombose. Samenvatting-Eng Hyperhomocysteinemia is a risk factor for thrombosis and recurrent thrombosis. Elevated homocysteine levels can be normalised by the B-vitamins folate, B6 and B12. In the present study the clinical effect of homocysteine-lowering therapy is studied (has not been performed before). About 5000 patients with thrombosis will be screened for their homocysteine levels in blood. Those with elevated homocysteine will enter a double-blind randomised placebo-controlled study with end-point recurrent thrombosis. Looptijd Status afgesloten Betrokken organisaties

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