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Hemochromatosis:     more books (55)

Iron Overload in the Erythropoietin Era.: An article from: Nephrology Nursing Journal by Karen C. Robbins, 2000-04-01 Journal of Genetic Counseling February 2008 (An Interdisciplinary Journal Focused on the Advancement of Knowledge about Genetic Counseling, Volume 17, Number 1) by Inc. National Society of Genetic Counselors, 2008 Iron overload and cutaneous disease: an emphasis on clinicopathological correlations.(NEWS, VIEWS & REVIEWS)(Report): An article from: Journal of Drugs in Dermatology by Laura Englander, Adam Friedman, 2010-06-01 Primary Iron Overload Also a Problem in African Americans.(Brief Article): An article from: Family Practice News by Mitchel L. Zoler, 2001-10-01 Audio-digest: Gastroenterology: Toxic Livers (Vol. 16, Issue 1, January 2002) by MD William Carey, MD Willis C. Maddrey, 2002 Phlebotomy: An entry from Gale's <i>Gale Encyclopedia of Surgery</i> by Paula Anne Ford-Martin, Richard Robinson, 2004 Cooking With Less Iron: Easy-To-Prepare, Reasonably Priced Meals That Reduce the Amount of Iron in Your Diet by Richard A. Passwater, 2001-11-01 Progress in Iron Research (Advances in Experimental Medicine and Biology) Clinical disorders of iron metabolism by Ernest Beutler, 1963 Iron Elephant: What You Should Know about the Dangers of Excess Body Iron by Roberta Crawford, 1993-01-01 Haemochromatosis (Oxford medical publications) by J. H Sheldon, 1935 Blood supply availability of blood : statement of Janet Heinrich, Associate Director, Health Financing and Public Health Issues, Health, Education, and ... (SuDoc GA 1.5/2:T-HEHS-99-195) by Janet Heinrich, 1999 Changing faces: 50 years of caring by Andrew Michael Wyner, 2003

lists with details

61. Porphyrias: Clinical Manifestations, Diagnosis And Treatment MEDSTUDENTS-METABOL
    A short review of hemochromatosis, giving atention mainly to its etiology, clinical manifestations, treatment and prognosis.Importance about early diagnosis is  
    http://www.medstudents.com.br/metdis/metdis4.htm
    
    Extractions: Medstudents' Homepage Hemochromatosis is a disease due to increased iron absorption that leads to accumulation of iron in diverse tissues. It can be hereditary (primary hemochromatosis) or secondary to chronic anemias as ß-thalassemias (ineffective erythropoiesis leading to increased erythrocitic destruction) or defects in hemoglobin synthesis ( as in porphyria cutanea tarda) ;multiple transfusion therapy ;or others less common conditions. Iron absorption from the gut occurs normally in a rate of 1 mg per day. In hemochromatosis this absortion reaches the rate of 4 to 5 mg/d and there is a progressive accumulation to 15 to 40 grams of body iron (normal ~ 2 to 3 g). Thus, the disease develops in men above age 20 and postmenopausal women, due to stopping in iron loss in menses. The iron accumulates in every tissue of the body, but main clinical manifestations are secondary to iron deposition in: Liver, thyroid, hypothalamus, heart, pancreas, gonads and joints. Many patients are asymptomatic. Common symptoms are fatigue, arthalgias, impotence, amenorrhea and palpitations. The most characteristical clinical manifestation occur in the liver because the portal circulation filters the iron initially absorbed fom the gut. Usually patients develop cirrhosis. Heart manifestations include arrythmias and disturbances of conduction. Skin adquires an diffuse hyperpigmentation mainly in scars, sun exposed and unexposed areas and genitalia. Arthropathy occurs in wrists, hips, knees and ankles. To distinguish this arthopathy from others, searching for initial involvement of second and third metacarpophalangeal joints is helpful. Hypogonadism hipogonadothrofic is secondary to iron deposition in hypophisary cells and leads to loss of libido, amenorrhea, impotence and body hair losing. Diabetes Mellitus results from pancreatic accomitment and the signs and symptons, as the treatment, do not differ from conventional diabetes.

62. Recognition And Management Of Hereditary Hemochromatosis - March 1, 2002 - Ameri
    American Family Physician. Recognition and Management of Hereditary hemochromatosis. Hereditary hemochromatosis is associated with mutations in the HFE gene.  
    http://www.aafp.org/afp/20020301/853.html
    
    Extractions: Mayo Medical School, Rochester, Minnesota Hereditary hemochromatosis is the most common inherited single-gene disorder in people of northern European descent. It is characterized by increased intestinal absorption of iron, with deposition of the iron in multiple organs. Previously, the classic description was combined diabetes mellitus, cutaneous hyperpigmentation and cirrhosis. Increasingly, however, hereditary hemochromatosis is being diagnosed at an earlier, less symptomatic stage. The diagnosis is based on a combination of clinical, laboratory and pathologic findings, including elevated serum transferrin saturation. Life expectancy is usually normal if phlebotomy is initiated before the development of cirrhosis or diabetes mellitus. Hereditary hemochromatosis is associated with mutations in the HFE gene. Between 60 and 93 percent of patients with the disorder are homozygous for a mutation designated C282Y. The

63. Hemochromatosis
    hemochromatosis The Liver The liver is the largest organ in the body. What is hemochromatosis? hemochromatosis is commonly known as iron overload disease.  
    http://www.gicare.com/pated/ecdlv36.htm
    
    Extractions: The liver is the largest organ in the body. It is found high in the right upper abdomen, behind the ribs. It is a very complex organ and has many functions. They include: The liver shoulders a heavy work load for the body, and almost never complains. It even has a remarkable power to regenerate itself. Still, it should not be taken for granted. The liver is subject to illnesses that can lead to permanent damage. One example is autoimmune hepatitis, a condition in which the body fights against its own liver. What is Hemochromatosis? Hemochromatosis is commonly known as iron overload disease. A malfunction in the way iron is absorbed from the intestinal tract causes the body to absorb and store too much iron. Certain vitamins and minerals are normally stored in the liver, and iron is one of them. However, too much iron stored in the liver can damage it. With hemochromatosis, excess iron is also deposited in other organs and tissues, especially the heart and pancreas, causing damage to these organs, too. This disease occurs more commonly than was originally thought. It is estimated that hemochromatosis affects about one in every 300-400 people. Occasionally, a secondary type of iron overload (called hemosiderosis) occurs in people who take large amounts of iron supplements, or in people who must have repeated blood transfusions, or who have certain other diseases. However, the body usually returns to normal processing of iron when the excess intake is eliminated.

64. Diabetes Types, Hemocromatosis
    hemochromatosis Can Cause Diabetes. hemochromatosis is an inherited disease that causes excessive amounts of iron to accumulate in the body.  
    http://www.diabetesnet.com/diabetes_types/hemocromatosis.php
    
    Extractions: about diabetes. People Insulins What Type You Have Type 1 ... Gestational Diabetes Hemocromatosis Cystic Fibrosis by John Walsh, P.A., C.D.E. Hemochromatosis is an inherited disease that causes excessive amounts of iron to accumulate in the body. Although diabetes can be one of many unwanted side effects of the iron overload, the rate of hemochromatosis is no higher in those with diabetes than those without. Diabetes caused by damage to the pancreas occurs after many years of hemochromatosis. In one French study, diabetes was found in 40% of 115 people at the time their hemochromatosis was diagnosed (Acta Clin Belg 1999 Dec; 54(6): pgs. 334-45). This type of secondary diabetes is often referred to as "bronze diabetes" because it is sometimes accompanied by a bronze color of the skin. Screening for hemochromatosis should be done in people with diabetes who also have liver disease. Hemochromatosis is the most common genetic disease in this country with 13% of the population carrying the gene, and one in every 200 people having it.

65. Kimball Genetics - Hemochromatosis DNA Test
    A laboratory specializing in genetic testing and counseling for hemochromatosis, venous thrombosis, fragile X and other common disorders.  
    http://www.kimballgenetics.com/tests-hemochrom.html
    
    Extractions: Hereditary Hemochromatosis (HH) is the most common genetic disorder known, with a prevalence of 1/200 and a carrier frequency of 1/8 in Caucasians. This disorder involves excess iron absorption and storage in organs and leads to high morbidity and mortality if untreated. Historically, most cases have remained undiagnosed until late in the course of the disease when irreversible damage has already occurred. Early diagnosis allows for prevention of symptoms through treatment by phlebotomy. Two mutations associated with hemochromatosis have been discovered in the HFE gene. The major mutation, Cys282Tyr (also known as 845A) is present in the homozygous state in 85% of Caucasians with clinically diagnosed hemochromatosis. Most homozygotes (with two copies of the mutation) and some heterozygotes (with one copy of the mutation) develop iron overload and hemochromatosis. A second mutation, His63Asp (also known as 187G), appears to somewhat increase risk of iron overload. Although only a small percent of individuals (up to 6%) with this mutation develop hemochromatosis, DNA testing for hemochromatosis provides for effective early diagnosis and prevention.

66. CLF : Hemochromatosis
    hemochromatosis What is hemochromatosis? hemochromatosis is an inherited disease in which the body absorbs too much iron from the diet.  
    http://www.liver.ca/english/liverdisease/hemochromatosis.html
    
    Extractions: Hemochromatosis is an inherited disease in which the body absorbs too much iron from the diet. Hemochromatosis is one of the most common genetic diseases in Canada affecting 1 in 327 Canadians. Normally, only enough iron to meet the body's daily requirements is absorbed; the remainder is usually excreted through the bowels. In hemochromatosis, however, iron continues to be absorbed and stored in different organs and tissues long after body needs are met. Hemochromatosis is hereditary. In order to have the disease, a patient must have inherited two defective genes, one from each parent; siblings of an affected individual have a 25 per cent chance of getting this disease. Children are less likely to be affected since the other parent must be a carrier of a hemochromatosis gene.

67. Iron_enemy S Home Page
    God Bless. There is a support group of people who have hemochromatosis, or someone in their family has it. hemochromatosis. Are You Dying From Too Much Iron?  
    http://www.geocities.com/HotSprings/Villa/6540/

68. National Human Genome Research Institute - Learning About Hereditary Hemochromat
    Answers to frequently asked questions about hereditary hemochromatosis, published by the National Human Genome Research Institute.  
    http://www.genome.gov/10001214
    
    Extractions: Hereditary hemochromatosis (HH) is a genetic disease that alters the body's ability to regulate iron absorption. If correctly diagnosed, HH is easily and effectively treated, but if untreated, it can lead to severe organ damage. Caucasians of northern European descent are at highest risk. An estimated one million people in the United States have hereditary hemochromatosis. HH causes the body to absorb too much iron. Normally humans extract needed iron from food via the intestines. When there is an adequate amount of iron, the body reduces its absorption to avoid excessive accumulations. In a person with HH, the mechanism for regulating iron absorption is faulty and the body absorbs too much iron. Over time - several years - this excess iron is deposited in the cells of the liver, heart, pancreas, joints and pituitary gland, leading to diseases such as cirrhosis of the liver, liver cancer, diabetes, heart disease and joint disease.

69. Hemochromatosis.be
    Webpage still under construction. Soon you will see information here about hemochromatosis. Please visit us again soon. For info mail info@hemochromatosis.be.  
    http://www.hemochromatosis.be/

70. Hemochromatosis: Iron Storage Disease
    hemochromatosis (Iron Storage Disease). Yet with this massive overdosing of iron, none of the birds had developed hemochromatosis.  
    http://www.starlingtalk.com/hemochromatosis.htm
    
    Extractions: Iron storage is not usually caused by dietary iron, but some years ago the theory was that perhaps the iron in the diets of some captive birds was causing the overload of iron, and some genetic factor was the reason for it. Hemochromatosis is not known to be in wild populations of birds; it is considered to be a disease of captive birds, yet in the wild, a mynah's main diet is figs, which have a very high iron content; if this were a genetic problem, then you would expect to see it in wild populations. Green Tea. Additionally, Zhi Zhong, PhD, from the University of North Carolina at Chapel Hill, and colleagues looked at whether the powerful, free-radical scavenging antioxidants in green tea could alleviate some of the problems associated with fatty livers. They found that "Green tea extract scavenges harmful free radicals in fatty livers and therefore could be an effective treatment to prevent failure of liver transplants." In a study at Brookfield Zoo, fifty eight European Starlings were studied after feeding one group a low iron diet of 148 ppm and the other group a high iron diet of 3,035 ppm of iron. All birds were euthanized after four months and tested for iron storage. Birds in both groups had similar liver iron content, but the birds on the high iron diet had significantly higher values. Yet with this massive overdosing of iron, none of the birds had developed Hemochromatosis.

71. Hemochromatosis
    A chapter from The Cleveland Clinic Disease Management Project written by Anthony S. Tavill, MD, which discusses hemochromatosis. The Cleveland Clinic.  
    http://www.clevelandclinicmeded.com/diseasemanagement/gastro/hemochromatosis/hem
    
    Extractions: The Cleveland Clinic Foundation There are many inherited metabolic diseases that may have a pathologic impact on the liver. In many cases the liver component of these diseases is only an epiphenomenon of a more generalized systemic disorder. Examples of such epiphenomena are glycogen and lipid storage diseases in which hepatomegaly is a manifestation of the underlying metabolic defect although the liver is not necessarily the major target organ. However, there are three genetically determined diseases in which the liver may be the principal target organ, with manifestations of acute, subacute, or chronic disease that may become evident in early or later life. These are hereditary hemochromatosis (HH), a major disorder of iron overload; Wilson's disease , a genetic disorder of copper overload; and

72. Hemochromatosis
    hemochromatosis information and links to national and international support groups, clinics with genetic counselors and geneticists. hemochromatosis.  
    http://www.kumc.edu/gec/support/hemochro.html
    
    Extractions: E-mail: margaret@haemochromatosis.org Web site: www.haemochromatosis.org/ Haemochromatosis Society , England Association Hemochromatose , France Hemochromatosis , Belgium Also See: National organizations , information on genetic conditions or birth defects Hemochromatosis Pamphlet , American Liver Foundation Hemochromatosis , Iron Disorders Institute National Digestive Diseases Information Clearinghouse (NDDIC) , National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health Hemochromatosis case study CDC webcast, May 2000, public health and genetics, view online

73. Hemochromatosis, Inflammation And Anemia
    hemochromatosis, inflammation and anemia. The HFE gene is mutated in patients suffering from the common iron overload disease hemochromatosis.  
    http://www.eurekalert.org/pub_releases/2004-04/embl-hia041504.php
    
    Extractions: European Molecular Biology Laboratory Patients with inflammatory diseases such as arthritis, chronic infections and some types of cancer, often become anemic – a condition called anemia of chronic disease (ACD). While ACD rarely kills patients, it can make their lives miserable. A discovery at EMBL, in collaboration with researchers at Children's Hospital Boston and Harvard Medical School, now links the gene HFE to ACD. The HFE gene is mutated in patients suffering from the common iron overload disease hemochromatosis. This finding gives hope that one day an effective and specific therapy may be developed to treat ACD (featured in Nature Genetics , April 18, 2004). When people are infected with microbes, the level of iron in their blood drops. This has an important function: iron is essential for the growth of infectious microbes, so one way for the body to fight back is to lower the amount of iron in circulation. "Unfortunately, while this decrease in iron hinders the spread of parasites and is beneficial in the short term, it can cause anemia," notes EMBL Group Leader Matthias Hentze. "During a long term inflammatory condition, low levels of iron can starve the bone marrow of this metal which is essential for blood cells, leading to ACD."

74. Hemochromatosis
    Informs patients about hemochromatosis, an inborn error of iron metabolism, which can lead to liver cirrhosis, liver cancer and heart failure.  
    http://www.ikp.unibe.ch/lab2/hemce.html
    
    Extractions: Hemochromatosis is the most prevalent genetic disease of adults. One in 10 carries the faulty gene and 1/400 will get the disease. Based on this gene frequency we have to assume that in the Swiss population there are 17'500 - 20'000 patients affected. The main mutation in the gene, now called HFE, was described in 1996 by Feder and colleagues. The mutation leads to uncontrolled absorption of iron and deposition of this potentially toxic heavy metal in different organs. 2. What are the signs and symptoms? The first symptoms can originate from the liver, the articulations, the pancreas, the heart and endocrine glands. This first signs can be very subtle and the diagnosis is missed if the treating physician does not think about the possibility of hemochromatosis. Liver Fatigue, dull aches or pains in the upper belly Articulations Inflammation, pain and swelling Pancreas Diabetes (thirst, increased urination)

75. Dr. Rose's Peripheral Brain--HEMOCHROMATOSIS
    hemochromatosis. I. Pathophysiology. Often get macrocytosis ( due to liver dysfunction ?); Genetic screens (for hereditary hemochromatosis).  
    http://faculty.washington.edu/momus/PB/hemochro.htm
    
    Extractions: HEMOCHROMATOSIS I. Pathophysiology Iron overload causing tissue damage Can be hereditary (mostly Caucasian), in which case excess intestinal absorption of iron occurs In the hereditary form, clinical manifestations occur after years of excess absorption Women homozygous for the hereditary defect express the disease 1/10 as often as men, prob. b/c of menstrual loss of iron Ethanol abuse or hepatitis accelerates liver and pancreatic damage, prob. due to high serum levels of ferritin after hepatocyte injury A candidate gene has been identified: HFEsee below Or secondary, caused by Chronic anemias, e.g. homozygous beta-thal Multiple transfusions Porphyria cutanea tarda Portocaval shunt, etc. Iron accumulation-ubiquitous, as ferritin and hemosiderin Liver All Fe absorbed from intestine passes through liver; any Fe that exceeds binding capacity of transferrin is deposited Pancreas, heart, gonads, thyroid, hypothalamus, pituitary, and joints, causing dysfunction in all these organs

76. HEMOCHROMATOSIS IN LORIES
    hemochromatosis IN LORIES. Margrethe Warden. hemochromatosis atra). In most cases, hemochromatosis is asymptomatic until shortly before death.  
    http://www.lorikeets.com/hemochromatosis.htm
    
    Extractions: HEMOCHROMATOSIS IN LORIES Margrethe Warden There have not been many studies conducted on avian hemochromatosis; therefore, much of what is commonly believed about the disorder is speculation. Most of the information relating to Iron Storage Disease and lories is anecdotal and as such is not always appropriate data for an actual scientific discussion. I began learning more about this condition in 1999 when it was discovered in sev eral of my own lories, specifically in five of my black lories ( Chalcopsitta atra). This disorder usually shows up in lories around five or six years of age; however, it has been discovered in much younger birds than that who have been fed a diet high in iron content. It is ultimately a fatal disorder that can take months, even years, to develop and become severe enough to result in death. The prevalence of hemochromatosis in lories is not well documented; therefore, it is not known if it has become a common problem in our captive population. The lack of documentation underscores the importance of performing a complete necropsy on every lory that dies. A liver stain can diagnose iron storage. Even when hemochromatosis does not appear to be the primary cause of death, it is important to conduct the histopathology to determine if the disorder was developing in the individual. Only through these tests and the compilation of information will the pervasiveness of this disease be documented and understood. Since most lory species are no longer being imported into this country, we must be diligent in maintaining the good health of the existing population.

77. Hereditary Hemochromatosis
    Your 68year-old Irish uncle was just diagnosed with hereditary hemochromatosis, a genetic disorder common among people of Northern European descent.  
    http://kidshealth.org/parent/general/aches/hh.html
    
    Extractions: Your 68-year-old Irish uncle has diabetes and has also been bothered lately by chronic fatigue and a swollen stomach. After seeing his doctor, he still isn't feeling well. Finally, he visits a new doctor who diagnoses him with hereditary hemochromatosis, a genetic disorder common among people of Northern European descent. You've never heard of this condition before, and now your head is spinning with questions. Could other family members have it? What are the symptoms? Are you at risk? Should you be concerned for your children? It's time to learn more! A genetic disease that causes the body to absorb and store too much iron , the condition gets its name from "hemo" for blood and "chroma" for color, referring to the characteristic bronze skin tone that iron overload can cause. Someone with hereditary hemochromatosis who has never taken an iron supplement could find out in later years that iron overload is causing serious health problems. Iron is a trace mineral that plays a vital role in the body. Every red blood cell contains iron in its hemoglobin, the pigment that carries oxygen from the lungs to the tissues. We get iron from our diet, and normally the body absorbs approximately 10% of the iron found in foods. People with hemochromatosis absorb double that amount.

78. Hemochromatosis
    Home Browse Resources What s Next Help Comments?. hemochromatosis. Also spelled HAEMOCHROMATOSIS, also called IRON STORAGE  
    http://www.cs.odu.edu/~wild/DiabetesWeb/education/hemochromatosis.html
    
    Extractions: Home Browse Resources What's Next ... Comments? Also spelled HAEMOCHROMATOSIS, also called IRON STORAGE DISEASE, or BRONZE, inborn metabolic defect characterized by an increased absorption of iron, which accumulates in body tissues. The clinical manifestations include skin pigmentation, diabetes, enlargement of the spleen and liver, heart failure, and general weakness and lassitude. In males, the symptoms are usually noted after 35 years of age, and in females, after menopause, when iron is no longer lost through menstruation and pregnancy. Treatment consists of the removal of blood at regular intervals to decrease the concentration of body iron.

79. Hemochromatosis - Information / Diagnosis / Treatment / Prevention
    home genetic disorders hemochromatosis hemochromatosis. Iron Overload Diseases Association Information on hemochromatosis.  
    http://www.healthcyclopedia.com/genetic-disorders/hemochromatosis.html
    
    Extractions: Web Directory: American Hemochromatosis Society This organisation provides information and educates the public about hemochromatosis. They publish a newsletter and have an online support group. Canadian Hemochromatosis Society Information about the symptoms, diagnosis and treatment of hemochromatosis.

80. Hemochromatosis Information
    What is hemochromatosis? Usually, excess iron that the body doesn t need is excreted. But in hemochromatosis, the excess iron is stored in the body.  
    http://www.mamashealth.com/hemo.asp
    
    Extractions: What is Hemochromatosis? Hemochromatosis, (he-mo-kro-ma-toe-sis) also called hereditary hemochromatosis or HHC is a hereditary disorder in which the body absorbs too much iron from food. Usually, excess iron that the body doesn't need is excreted. But in hemochromatosis, the excess iron is stored in the body. Some of the places it is stored are: the pancreas, liver, and skin. The excess iron damages these organs and tissues. What are the Symptoms? Some of the most common symptoms of hemochromatosis are: fatigue, abdominal pain, jaundice (yellowing of skin and eyes), and a change in skin color to bronze or gray. Long Term Affects? Over time, hemochromatosis can cause liver disease, liver failure, liver cancer heart disease diabetes , impotence for men, and infertility and loss of periods for women. How is Hemochromatosis diagnosed? Hemochromatosis is diagnosed through blood tests that measure iron levels. If your doctor thinks liver damage has occured, a liver biopsy can be done to check for the amount of iron in the liver and for liver damage. How is it Treated?

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