Healthfinder® Glomerular Diseases Overview of kidney function and glomerular diseases and their causes, symptoms, and diagnosis. glomerular diseases. internet URL. http://www.healthfinder.gov/Scripts/ShowDocDetail.asp?doc=6507&lang=1
CIN'2003. Wedeen. Occupational Renal Diseases The contribution of environmental toxin exposure to the development of lupus erythematosus and glomerular diseases remains to be determined. http://www.uninet.edu/cin2003/conf/wedeen/wedeen.html
Extractions: East Orange, New Jersey. USA wedeen@umdnj.edu Occupational renal diseases are important because they are entirely preventable. In addition, renal disease produced by heavy exposure to industrial toxins by relatively few workers provides a clinical basis for understanding the consequences of low-dose exposure to the general population from environmental pollution. The known toxins that produce kidney disease in the workplace are limited to the heavy metals plus silica and certain organic compounds, particularly halogenated hydrocarbons, employed as industrial solvents. The absence of information on the adverse renal effects of other industrial pollutants does not prove that no such effects occur, but rather that epidemiologic studies are lacking. It is probable that as the genetic basis of individual susceptibility unfolds, the contribution of toxins to renal disease in the general population will become clearer. The renal diseases due to heavy metals are listed in the Table. Lead, cadmium, mercury arsenic, chromium, and uranium are widely recognized as nephrotoxins in humans. Given appropriate chemical forms, dosage, and route of administration, acute tubular necrosis (ATN) is consistently produced by these metals. ATN occurs because of the selective accumulation of metals in the proximal tubule. When the appropriate oxidative form is present in sufficient quantity, the metals induce irreversible cellular injury. Following intense occupational exposure, and in animal experiments, the cause of ATN is obvious. When recovery is incomplete, chronic tubular-interstitial nephritis (TIN) may persist as a residual. On the other hand, the role of heavy metals in the induction of immunologically mediated glomerular disease is by no means obvious.
Glomerular Diseases Glomerular. Diseases. Educational Computing Curriculum General Pathology General Pathology Courseware Hippocrates Project http://endeavor.med.nyu.edu/courses/systemicpathology/courseware/renalpath/r1/gl
Glomerular Diseases II glomerular diseases II. Click here to start. Table of Contents. glomerular diseases II. Renal Disease associated with Diabetes Mellitus (p569). http://pathophysiology.uams.edu/Spring03/Renal/Renal4/
Glomerular Diseases: Part A & B II. glomerular diseases A. Etiologic types 1. Immunologic glomerular diseases. 2. Metabolic diseases (Diabetic Mellitus, Amyloidosis). http://www.umdnj.edu/pathnweb/syspath/lec_1/gd_ab/gd_ab.htm
Extractions: Home G.D.: C II. Glomerular Diseases: A. Etiologic types: B. Pathogenic mechanisms of immunologic glomerular diseases: ii.Antigen endogenous (e.g., DNA, Ig, tumor antigens) b.In-situ immune complex formation: (Fig: 5A) Fig 5A i.Visceral epithelial cell GP330 antigen (Exp. Model: Heyman nephritis) (Fig 5B) Fig 5B ii.Planted (trapped) antigens: (Fig:5C)
Glomerular Diseases: Part D,E & F injury. 3. End stage disease. E. Classification of glomerular diseases 1. Primary glomerular diseases, eg, Postinfectious, Drugs, etc. http://www.umdnj.edu/pathnweb/syspath/lec_1/gd_def/gd_def.htm
DVM - Glomerulonephritis Remains An Important Cause Of Renal It can be very difficult to differentiate nonimmunologically from immunologically mediated glomerular diseases with the limited diagnostic techniques that are http://www.dvmnewsmagazine.com/dvm/article/articleDetail.jsp?id=88239
Glomerular Diseases - National Institutes Of Health (NIH) glomerular diseases Health Information from National Institutes of Health (NIH). Skip Over Navigation Links. glomerular diseases. Resources. NIDDK. http://health.nih.gov/result.asp/1058
Extractions: NIDDK Directory of Kidney and Other Urologic Diseases Organizations Glomerular Diseases ... Your Urinary System and How It Works Related Topics: Kidney Diseases (General) Kidney and Urinary System Up to Top Look up "Glomerular Diseases" in: MEDLINEplus - health resources from US government agencies and other credible organizations Clinical Trials Database - research studies in which you can participate This page was last reviewed on Tuesday, February 24, 2004. Career Opportunities Visitor Information FOIA
MED On Line has not been previously determined) and to identify any associated infections, renal biopsies were performed in 85 patients with glomerular diseases who were http://www.medonline.com.br/med_ed/med8/miami1.htm
Canine Glomerular Disease - WSAVA 2002 Use of the urine protein/creatinine ratio to identify and quantitate proteinuria has greatly facilitated diagnosis of these glomerular diseases in veterinary http://www.vin.com/proceedings/Proceedings.plx?CID=WSAVA2002&PID=2706
Karger Publishers Urinary Sediment Podocalyxin in Children with glomerular diseases Katsue Kanno a , Hiroshi Kawachi d , Yoshiaki Uchida c , Masanori Hara b , Fujio Shimizu d http://content.karger.com/ProdukteDB/produkte.asp?Doi=74322
Glomerular Kidney and Urologic Diseases Information Clearinghouse A detailed discussion about the kidneys and how glomerular diseases interfere with their functions. http://www.thenewhealthfind.com/Health/ConditionsandDiseases/UrologicalDisorders
Extractions: RESULTS: A total of 1629 consecutive percutaneous renal biopsies of native kidneys showed glomerular disease in 1413 cases. The most common clinical indication for renal biopsy was persistent proteinuria (n=735; 52.0%), while the most frequently found glomerular lesion was immunoglobulin A nephropathy (n=338; 23.9%). Minimal-change nephrotic syndrome (n=125; 8.8%) and immunoglobulin M nephropathy (n=11; 0.8%), were the most common glomerular diseases that presented with nephrotic syndrome. The male to female ratio for lupus nephritis was 1:14 (n=290), whereas for minimal-change nephrotic syndrome, the ratio was 1.8:1 (n=125). Immunoglobulin A nephropathy and membranous glomerulonephritis (n=117) affected approximately equal numbers of male and female patients. Familial fibrillary glomerulonephritis, a disease hitherto unknown in Hong Kong, was diagnosed in two siblings.
Kidney Disease glomerular diseases glomerular diseases are those that attack the blood filtering units of the kidneys. Diabetes and high blood http://www.labtestsonline.org/understanding/conditions/kidney-2.html
Extractions: TESTS Test not listed? A/G Ratio ACE ACT ACTH AFB Culture AFP Maternal AFP Tumor Marker Albumin Aldosterone Allergies ALP Alpha-1 Antitrypsin ALT Amylase ANA Antibody Tests Apo A Apo B ApoE Genotyping aPTT AST Autoantibodies Bilirubin Blood Culture Blood Gases Blood Smear BMP BNP Bone Markers BRCA BUN C-peptide CA-125 CA 15-3 CA 19-9 Calcium Cardiac Risk CBC CEA Celiac Disease Tests CF Gene Mutation Chlamydia Chloride Cholesterol CK CK-MB CMP CMV Coagulation Factors Complement Levels Cortisol Creatinine Creatinine Clearance CRP CRP, high-sensitivity Cystatin C D-dimer DHEAS Differential DLDL EGFR Electrolytes Electrophoresis ESR Estrogen Estrogen Receptors Factor V Leiden Fecal Occult Blood Ferritin fFN Fibrinogen Flu Tests Folate Fructosamine FSH Genotypic Resistance GFR GGT Glucose Gonorrhea Gram Stain Growth Hormone H-pylori hCG HDL Hematocrit Hemoglobin Hemoglobin Variants Hepatitis A Hepatitis B Hepatitis C Her-2/neu Herpes HIV Antibody HLA-B27 Home Tests Homocysteine HPV hs-CRP IGF-1
Complementary Medicines First Name Last Name email Note you can unsubscribe any time you wish. glomerular diseases. How Do glomerular diseases Interfere With Kidney Function? http://www.complementarymedicines.com/ailments/ailments_display.php?id=36
Blackwell Synergy - Cookie Absent Complement regulatory proteins in glomerular diseases. Masaomi Nangaku. Complement regulatory proteins in glomerular diseases. http://www.blackwell-synergy.com/links/doi/10.1046/j.1523-1755.1998.00130.x/full
Extractions: Home An Error Occurred Setting Your User Cookie A cookie is a small amount of information that a web site copies onto your hard drive. Synergy uses cookies to improve performance by remembering that you are logged in when you go from page to page. If the cookie cannot be set correctly, then Synergy cannot determine whether you are logged in and a new session will be created for each page you visit. This slows the system down. Therefore, you must accept the Synergy cookie to use the system. What Gets Stored in a Cookie? Synergy only stores a session ID in the cookie, no other information is captured. In general, only the information that you provide, or the choices you make while visiting a web site, can be stored in a cookie. For example, the site cannot determine your email name unless you choose to type it. Allowing a web site to create a cookie does not give that or any other site access to the rest of your computer, and only the site that created the cookie can read it. Please read our for more information about data collected on this site.
Blackwell Synergy - Cookie Absent PERSPECTIVES IN BASIC SCIENCE. Progression of glomerular diseases Is the podocyte the culprit? Progression of glomerular diseases Is the podocyte the culprit? http://www.blackwell-synergy.com/links/doi/10.1046/j.1523-1755.1998.00044.x/enha
Extractions: Home An Error Occurred Setting Your User Cookie A cookie is a small amount of information that a web site copies onto your hard drive. Synergy uses cookies to improve performance by remembering that you are logged in when you go from page to page. If the cookie cannot be set correctly, then Synergy cannot determine whether you are logged in and a new session will be created for each page you visit. This slows the system down. Therefore, you must accept the Synergy cookie to use the system. What Gets Stored in a Cookie? Synergy only stores a session ID in the cookie, no other information is captured. In general, only the information that you provide, or the choices you make while visiting a web site, can be stored in a cookie. For example, the site cannot determine your email name unless you choose to type it. Allowing a web site to create a cookie does not give that or any other site access to the rest of your computer, and only the site that created the cookie can read it. Please read our for more information about data collected on this site.
Berger Disease - Medstudents - Neprhology are increased in the serum of 5095% of patients, and its measurement is extremely useful in differentiating IgA nephropathy from other glomerular diseases. http://www.medstudents.com.br/nefro/nefro2.htm
Extractions: Medstudents' Homepage Also known as primary IgA nephropathy, glomerulonephritis with IgA and IgG deposits and IgA mesangial nephropathy, Berger disease has a wide pattern of distribution and is believed to be the most common form of primary glomerular disease throughout the world. Although highly prevalent, geographic variations occur, which could be due to genetic as well as enviromental influences. Extensive mesangial IgA deposits (the characteristic pathologic feature of Berger disease), may also occur in a variety of multisystem, neoplastic and infectious diseases; when this occurs the term Secondary IgA Nephropathy should be applied. Examples of the most common causes are: Multisystem disease Schönlein-Henoch purpura In spite of the extremely varied clinical presentation, the most common feature leading to the diagnosis, by renal biopsy, is recurring episodes of macroscopic hematuria (which is generally the initial complaint). More than 80% of patients are between the ages of 16 to 35 years at the time of diagnosis. However, the disorder may be found at any age, although being uncommon before the age of 10 and after the age of 50. Thus, it should be considered a disease of children and young adults. There is a predilection for males (2:1), which disappears when affecting black persons (this disease is uncommon in blacks).
Committee On Appropriations Mr. Chairman, FSGS is one of a cluster of glomerular diseases that attack the one million tiny filtering units contained in each human kidney. http://appropriations.house.gov/index.cfm?Fuseaction=Hearings.Testimony&HearingI
Entrez PubMed Renal function in proteinuric glomerular diseases correlates to the changes in urine IgM excretion but not to the changes in the degree of albuminuria. http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=1
