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Factor V Leiden:     more detail

Stroke after Marijuana smoking in a teenager with factor V Leiden mutation.(Brief Article): An article from: Southern Medical Journal by Mark A. Marinella, 2001-12-01 Estrogen use with factor V Leiden not advised.(Women's Health)(Clinical report): An article from: Internal Medicine News by Colin Nelson, 2006-08-15 Factor V Leiden genetic variant in an American Indian population.(COMMUNICATIONS--PROFESSIONAL): An article from: Proceedings of the North Dakota Academy of Science by Melanie Nadeau, Sheri T. Dorsam, et all 2007-04-01 Genetic Polymorphisms: Single Nucleotide Polymorphisms, 5-Httlpr, Factor V Leiden, Rs6265, Rs6313, Rs6295, Rs5569, Rs6311, Rs6314, Rs7997012 Single Nucleotide Polymorphisms: Factor V Leiden, Rs6265, Rs6313, Rs6295, Rs5569, Rs6311, Rs6314, Rs7997012, Rs1805054, Rs4680, Rs1801133 Factor V Leiden thrombophilia: An entry from Thomson Gale's <i>Gale Encyclopedia of Genetic Disorders, 2nd ed.</i> by Dawn, MS, CGC Jacob, 2005 Blood Proteins: Hemoglobin, Hemocyanin, Glycated Hemoglobin, Haptoglobin, Human Serum Albumin, Fibrin, Factor V Leiden Factor V Leiden as a common genetic risk factor for venous thromboembolism.(Genomics to Health): An article from: Journal of Nursing Scholarship by McDonald K., III Horne, Donna Jo McCloskey, 2006-03-22 Factor V Leiden

lists with details

1. Factor V Leiden / Thrombophilia Support Page - Living With Thrombophilia
   This site contains information for those living with Thrombophilia or factor v leiden. FVL Thrombophilia Support Page.  
   http://www.fvleiden.org/
   
   Extractions: The prothrombin 20210 mutation is the second most common inherited clotting abnormality. It is more common than protein S and C deficiency and Antithrombin deficiency combined; 2% of the general population is heterozygous. It is only a mild risk factor for clots, but together with other risk factors (such as oral contraceptives, surgery, trauma, high blood pressure, obesity, smoking, etc) or combined with other clotting disorders (like Factor V Leiden), the risk of clotting increases dramatically.

2. Factor V Leiden Thrombophilia
   factor v leiden Thrombophilia. Table 1. Molecular Genetic Testing Used in factor v leiden. % of Patients. Genetic Mechanism. Test Type. Test Availability.  
   http://www.geneclinics.org/profiles/factor-v-leiden/details.html

3. Factor V Leiden
   Patient Resources factor v leiden. Associated with factor v leiden, APCR was first described in 1993; factor v leiden was subsequently discovered in 1994.  
   http://www-admin.med.uiuc.edu/hematology/PtFacV2.htm
   
   Extractions: University of Illinois - Urbana/Champaign Carle Cancer Center Hematology Resource Page Patient Resources Factor V Leiden Home Factor V Leiden Antiphospholipid Syndrome General Clotting Information ... Protein S deficiency Factor V Leiden is a genetically acquired trait that can result in a thrombophilic (hypercoaguable) state resulting in the phenomenon of activated protein C resistance (APCR) as described below. Associated with factor V Leiden, APCR was first described in 1993; factor V Leiden was subsequently discovered in 1994. Over 95% of patients with APCR have factor V Leiden. Factor V Leiden's overall impact on the coagulation cascade described below. Mechanism of Action of Factor V Leiden: Factor V Leiden is seen more commonly in the northern European populations. About 4-7% of the general population is heterozygous (see * below) for factor V Leiden. About 0.06 to 0.25% of the population is homozygous (see * below) for factor V Leiden. The factor V Leiden mutation is relatively uncommon in the native populations of Asia, Africa and North America. In contrast, in Greece and southern Sweden, rates above 10% have been reported.

4. Factor V Leiden
   Informatieve site over alles wat met trombose en (erfelijke)stollingsafwijkingen te maken heeft, toegespitst op faktor V Leiden.  
   http://www.factorvleiden.nl/
   
   Extractions: Links Z oek je al lang naar begrijpelijke informatie over Factor V Leiden? Heb je vragen over Factor V Leiden waarop je arts geen antwoord weet? Zoek je informatie over Factor V Leiden of andere stollingsafwijkingen of ben je gewoon op zoek naar Je hebt het goede adres gevonden. Zelf zochten wij het internet af op zoek naar begrijpelijke informatie. Omdat wij niets konden vinden, hebben we zelf een site gebouwd. Wij zijn geen artsen, maar ervaringsdeskundigen. Wijs geworden door het uitwisselen van kennis en ervaringen op onze mailinglist . Wij actualiseren deze site maandelijks met informatie vanuit de mailinglist, informatie uit medische artikelen en informatie die wij van artsen krijgen. Deze kennis willen wij graag met je delen, omdat we het belangrijk vinden dat er meer bekendheid komt over Factor V Leiden. Daarnaast willen we op deze manier bijdragen aan een juiste behandeling van "dragers" van deze stollingsafwijkingen. Het begon eigenlijk met de stollingsafwijking Factor V Leiden, maar dit is gaandeweg uitgebreid naar verschillende

5. Factor V Leiden
   factor v leiden (thrombophilia) factor v leiden profile, GeneClinics, University of Washington  
   http://www.kumc.edu/gec/support/leiden.html
   
   Extractions: Factor V Leiden profile , GeneClinics, University of Washington Deep Vein Thrombosis , DVT.net DeepVein Thrombosis (DVT), Spotlight Health Thrombophilia Support Page (FVL), by individual Factor V Leiden and Venous Thrombosis Case Study , Centers for Disease Control (CDC) Hereditary Thrombophilia , Contact a Family, Canada contact a family in your area

6. Pregnancy, Clotting, And Factor V Leiden: An Overview
   Pregnancy, Clotting, and factor v leiden An Overview. The past 10 years have brought new understanding of and explanations why. some women clot on birth control pills and during pregnancy. Research  
   http://www.naturalchildbirth.org/natural/resources/prebirth/prebirth35.htm

7. Factor V Leiden / Thrombophilia Support Page - Links
   This site contains information for those living with Thrombophilia or factor v leiden. FVL Thrombophilia Support Page. Home Mailing  
   http://www.fvleiden.org/links.html
   
   Extractions: Nine years ago when I searched the web for the first time, I only found one article on fvl. Now if you search, you'll find many articles. This list is not exhaustive or comprehensive. It is a sampling of what's out there. If you want to read up on APC, APS, FVL or any other clotting disorder go to one of the major search engines and put in a variety of spellings for the disorder. You will find many articles.

8. Factor V Leiden
   factor v leiden is a genetically acquired trait that can result in a thrombophilic (hypercoaguable) state with APCR have factor v leiden. factor v leiden's overall impact on  
   http://www.med.uiuc.edu/hematology/PtFacV2.htm

9. GeneReviews: Factor V Leiden Thrombophilia
   Your browser does not support HTML frames so you must view factor v leiden Thrombophilia in a slightly less readable form. Please follow this link to do so.  
   http://www.geneclinics.org/profiles/factor-v-leiden/

10. Predictive Genetics And DNA Diagnostics - Home
    Offers home collection kits for a range of tests including angiotensinogen, factor v leiden, hereditary hemochromatosis, periodontal disease and prothrombin.  
    http://www.pgd2.com/

11. Page Not Found
    Discusses the thrombotic risks of this inherited disorder, also known as factor v leiden.  
    http://www.beckmancoulter.com/Coulter/Techpubs/coagulation/APC.asp
    
    Extractions: If so, please send us your comments . On the comments form, select "Contact the WebMaster" under the column "Non Product Related Questions". If available, please specify the name of the link you clicked, the URL of the page, or how you navigated there. Home Products Customer Support Resource Center ...

12. Genaissance - Solutions - GLP Genotyping - Factor V Leiden
    Available Assays. Choose an Assay A2M. APOE. COMT. CYP2A6. CYP2C9. CYP2C19. CYP2D6. CYP3A4. CYP3A5. DRD2. F2. factor v leiden. FLT3. MDR1. NAT2. PPAR. SDC4. UGT1A1. 5HTT with a 50 to 100fold  
    http://www.genaissance.com/products_services/capabilitiesServices_cg_factor.asp
    
    Extractions: Available Assays Choose an Assay... APOE COMT Factor V Leiden PPAR This mutation is in the initial cleavage site of Factor Va, which disrupts the recognition by Activate Protein C. The result is a long-lived Factor Va and a hypercoaguable state. The clinical name for this condition is Activate Protein C resistance (APCR), which is the most common inherited risk factor in venous thrombosis. Heterozygosity for this mutation is associated with a 5 to 10-fold increased risk of developing a venous thrombitic episode while homozygosity for this mutation is associated with a 50 to 100-fold increased risk. Factor V Leiden is associated with the following complications: venous thrombosis (blood clots, especially with oral contraceptives); deep vein thrombosis (DVT, blood clots in veins); unexplained miscarriage; blood clots in the lungs or anywhere else; gall bladder dysfunction; preeclampsia and/or eclampsia (toxemia while pregnant); and stroke and/or heart attack. Phenotype expression: Blood clots Therapeutic significance: Cardiovascular disease What gene codes for: Clotting factor Indications for use of assay: Concern of thrombophilia

13. Factor V Leiden
    factor v leiden is not a disease, it is the presence of a particular gene that is passed on from your parents. The factor v leiden. Reviewed  
    http://www.netdoctor.co.uk/diseases/facts/factorv.htm
    
    Extractions: NetDoctor.co.uk Home News and features News Newsletter Features Encyclopaedia Diseases Examinations Medicines Premium services SMS services StayQuit thediet Health centres Allergy and asthma Children's health Depression Eczema ... All health centres Discussion and support Discussion forums Support groups Services Ask the doctor Find a hospital Search Medline Test yourself Information About NetDoctor Commercial opportunities NetDoctor.com Factor V Leiden Dr Patrick Davey , cardiologist Dr Rachel Green , consultant haematologist

14. C
    factor v leidenDeborah L. Ornstein, MD; Mary Cushman, MD, MScYou may have been tested for the gene for factor V. factor v leiden is an abnormal ver  
    http://circ.ahajournals.org/cgi/reprint/107/15/e94.pdf

15. MSRGSNet/Genetic Drift/Venous Thrombosis And The Factor V (Leiden) Mutation
    Venous Thrombosis and the Factor V (Leiden) Mutation. The factor V mutation (factor v leiden) is the most common genetic cause of venous thrombosis.  
    http://www.mostgene.org/gd/gdvol14b.htm
    
    Extractions: Who should be tested? Indications include: Deep vein thrombosis and pulmonary embolism Venous thrombosis and pulmonary embolism pose a serious health problem. In this country half a million people are hospitalized each year and 50,000-100,000 deaths occur due to venous thrombosis which is also a leading cause of maternal death. The incidence of symptomatic venous thrombosis cases is approximately 1 in 1000 people per year. Venous thrombosis is a multifactorial condition caused by a combination of genetic, aquired or environmental influences. Natural anticoagulant systems (the protein C system and antithrombin III) are in place to keep coagulation in check. Excess clotting occurs when there is a disturbance in one of the coagulation inhibitor mechanisms or in natural lysis of clots.

16. MoSt GeNe/Genetic Drift/Prenatal Diagnosis
    20 Fall 2002. Heterozygote Counseling for the factor v leiden Mutation. factor v leiden is not known to be associated with arterial thrombosis.  
    http://www.mostgene.org/gd/gdvol20g.htm
    
    Extractions: Factor V Leiden Mutation Heterozygosity for the Leiden mutation in the factor V gene (FVL) is common and may present complex counseling issues. Heterozygosity refers to the situation when an individual has a single Leiden mutation in one of the factor V genes (see Adult Thrombotic Disorders in this issue of the Genetic Drift ). It is inherited in an autosomal dominant manner, and may be identified in individuals with thrombosis or their family members. Heterozygotes exhibit reduced penetrance, meaning some individuals will never exhibit symptoms. In this condition, at least 90% of heterozygotes are asymptomatic throughout their lives. The heterozygote frequency is 5% of the Caucasian US population, 2% of Hispanic Americans, about 1% of African Americans and Native Americans, and less than 1% of Asian Americans. In the general population, deep vein thrombosis occurs in 1 in 1,000 individuals. Heterozygosity for FVL confers a 4-8-fold increased lifetime risk for deep vein thrombosis, yet thrombosis occurs with other circumstantial factors in 50% of cases. Twenty to sixty percent of Caucasians with thrombosis have FVL, and most of these are heterozygotes. Homozygotes, or those with two copies of the faulty gene, have up to an 80-fold risk for thrombosis. For genetic counseling of heterozygotes, it is useful to divide the asymptomatic from the symptomatic, and adults from children.

17. Factor V Leiden Syndrome
    The term "factor v leiden" refers to specific G to A substitution at nucleotide 1691 in the no clinical features specific for factor v leiden the primary clinical manifestation  
    http://www.ibis-birthdefects.org/start/f5fact.htm
    
    Extractions: Extracts from the Seminole paper by R. M. Bertina et al, Nature, 369: 64, 1994. "... single point mutation in the gene for the blood coagulation protein factor V ... 20 percent of all patients with venous thrombosis as well as 4 percent of the general population are carriers of this mutations ..."

18. Case Study: Factor V Leiden And Venous Thrombosis Overview
    Your browser does not support script. factor v leiden and Venous Thrombosis Case Study, Educational Objectives. factor v leiden and Venous Thrombosis Overview.  
    http://www.cdc.gov/genomics/hugenet/CaseStudy/FVL/FVLview.htm
    
    Extractions: Your browser does not support script Factor V Leiden and Venous Thrombosis Case Study Educational Objectives After reading this case study you should be able to: Factor V Leiden and Venous Thrombosis Overview V enous thrombosis is an important cause of morbidity; incidence is low in young people but increases with age to 1% per year in the elderly. In a small proportion of cases, venous thrombosis leads to pulmonary embolism, which can be fatal. Persons with an initial venous thrombosis are at increased risk for recurrence; however, long-term use of anticoagulant prophylaxis can result in major hemorrhagic complications. This challenging clinical problem has received new attention since the discovery of certain genetic variants that increase susceptibility to venous thrombosis.

19. E-joiurnal: Factor V Leiden Mutation And The Risk Of Venous Thrombolembolism In
    April 17, 2002. factor v leiden Mutation and the Risk of Venous Thrombolembolism in Pregnant Women. EJournal Club abstraction form on Tormene D et al.  
    http://www.cdc.gov/genomics/hugenet/ejournal/fvlmutation.htm
    
    Extractions: Emory University Past issues The Health Outcome Pregnancy and puerperium are known risk factors for venous thromboembolism (VTE) (1, 2). The incidence of VTE during pregnancy is 1 per 1,000 (1,2) and increases 10 times during the postpartum period (3). These incidence rates are considerably higher than those reported for women of similar age in the general population. A variety of clotting inhibitor deficiencies have been identified as risk factors for VTE in pregnancy, but most of these defects are rare. Factor V Leiden mutation is relatively common: it affects 5% of the southern European population. The Finding The purpose of the study by Tormene et al. (4), therefore, was to assess the relation between factor V Leiden mutation and incidence of pregnancy-related VTE using a case-control design. Study subjects were

20. Entrez PubMed
    The role of factor v leiden mutation in recurrent pregnancy loss. ClearyGoldman J, Nakhuda GS, Zimmermann RC, Sauer MV. Department  
    http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=1

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