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Basal Ganglia Diseases:     more books (74)

l-dopa impairs learning, but spares generalization, in Parkinson's disease [An article from: Neuropsychologia] by D. Shohamy, C.E. Myers, et all The roles of sequencing and verbal working memory in sentence comprehension deficits in Parkinson's disease [An article from: Brain and Language] by J. Hochstadt, H. Nakano, et all 2006-06-01 Flanker compatibility effects in patients with Parkinson's disease: Impact of target onset delay and trial-by-trial stimulus variation [An article from: Brain and Cognition] by X.E. Cagigas, J. Vincent Filoteo, et all 2007-04-01 Advances in Neurology: Basal Ganglia and New Surgical Approaches For Parkinson's Disease, Vol. 74 by C. David Marsden, 1997 Advances in Neurology: Basal Ganglia and New Surgical Approaches For Parkinson's Disease, Vol. 74 by C. David Marsden, 1997-01-01 Developments in research: The works of Professor Hirotaro Narabayashi and his associates by Hirotarō Narabayashi, 1988 Brain Biochemistry and Brain Disorders by Philip G. Strange, 1993-01-07 Age-Related Dopamine-Dependent Disorders: International Symposium on Age-Related Monoamine-Dependent Disorders and Their Modulation by Gene and Gend (Frontiers of Neurology and Neuroscience) by M. Segawa, 1995-05 Backus Strikes Back by Jim Backus, Henny Backus, 1984-05 The neuropsychology of ventral prefrontal cortex: Decision-making and reversal learning [An article from: Brain and Cognition] by L. Clark, R. Cools, et all 2004-06-01 Variability in subcortical aphasia is due to variable sites of cortical hypoperfusion [An article from: Brain and Language] by A.E. Hillis, P.B. Barker, et all 2004-06-01 PARKINSONISM: An entry from Macmillan Reference USA's <i>Encyclopedia of Aging</i> by SULTAN DARVESH, 2002 Corticobasal degeneration: An entry from Thomson Gale's <i>Gale Encyclopedia of Neurological Disorders</i> by Richard Robinson, 2005 Is the unilateral lesion of the left substantia nigra pars compacta sufficient to induce working memory impairment in rats? [An article from: Neurobiology of Learning and Memory] by M.I. Bellissimo, I. Kouzmine, et all 2004-09-01

lists with details

81. HealingWell.com - Parkinson's Disease
    the basal ganglia and the right parietal lobe, that are key to measuring time.They hope the discovery will help them treat Parkinson s disease, Attention  
    http://healingwellparkinsons.subportal.com/health/Diseases_and_Conditions/Brain/
    
    Extractions: HealthScout Reporter THURSDAY, March 1 (HealthScout) They say timing is everything, and now doctors have figured out how your brain keeps its eye on the clock. Using sophisticated brain scans, scientists have identified the two parts of the brain, called the basal ganglia and the right parietal lobe, that are key to measuring time. They hope the discovery will help them treat Parkinson's disease, Attention-Deficit/Hyperactivity Disorder (ADHD) and Huntington's disease, all of which weaken time perception for their sufferers. "Now we have a diagnostic technique that will allow us to measure the activity of basal ganglia as a way, not only to evaluate the effectiveness of existing treatments, but to evaluate novel treatments," says lead investigator Stephen Rao. The results of the study appear in the current issue of Nature Neuroscience . The study was funded by the National Institute of Mental Health. "I think it's a very interesting result, but unexpected," says Dr. Roger Albin, a University of Michigan neurology professor who studies the basal ganglia. Most literature, he says, points to the cerebellum as the site for time perception.

82. Re: Basal Ganglia
    Betty P. The only info I came across was that the disorders of the basal ganglia(extrapyramidaldisorders These symptoms are as those in Parkinson s Disease.  
    http://www.healthboards.com/parkinsons-disease/2603.html
    
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83. Health, Conditions And Diseases, Neurological Disorders, Brain Diseases, Basal G
    HealthConditions and DiseasesRare Disorders. Neuroleptic Malignant Syndrome InformationService Information about the service as well as the disease itself.  
    http://www.combose.com/Health/Conditions_and_Diseases/Neurological_Disorders/Bra

84. HONselect - Parkinson Disease, Secondary
    Browse other medical images in HONmedia? MeSH Broader term(s) Brain diseases. Otherhealth news. MeSH Broader term(s) Nervous System diseases.  
    http://www.hon.ch/HONselect/Selection/C10.228.140.079.862.800.html
    
    Extractions: the word the part of word in MeSH term in MeSH term and description Contents on "Parkinson Disease, Secondary": MeSH hierarchy and definition Research Articles Web resources Medical Images Medical News Medical Conferences Clinical Trials MeSH Hierarchy English French German Spanish Portuguese MeSH Broader term(s) Diseases Parkinson Disease, Secondary MeSH definition Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)

85. CEREBROVASCULAR DISEASES
    Middle cerebral artery supplies the lateral parts of the cerebral hemisphere, basalganglia, and the anterior limb of the HYPERTENSIVE CEREBROVASCULAR DISEASE.  
    http://edcenter.med.cornell.edu/CUMC_PathNotes/Neuropathology/Neuropath_I/isch.h
    
    Extractions: A stroke is defined as a neurologic deficit with sudden onset due to vascular disease. It is clinical (not pathological) definition. Cerebral ischemia is a decrease in cerebral blood flow with resultant decrease in cerebral oxygenation. The brain is totally dependent upon aerobic metabolism. It receives 15% of the resting cardiac output and accounts for 20% 0f the oxygen consumption. Hypoxia, regardless of cause, produces a constant pattern of brain injury; however, the injury may be localized or generalized - resulting in two patterns of Acute Ischemic Injury: Ischemic (hypoxic) Encephalopathy - generalized (global) hypoxic injury Cerebral Infarction - localized hypoxic injury with necrosis When blood flow is decreased, followed by subsequent reperfusion, one may see reversible or irreversible brain damage. Reversible Ischemia Focal Ischemia Transient Ischemic Attacks (TIA's) Focal neurologic deficits lasting less than 24 hours. No permanent brain damage results. Theoretically, very severe or prolonged focal ischemia can cause infarction:

86. Smith Lab Projects And Grants
    and electrophysiologic effects of these receptors on various populations of basalganglia neurons in normal monkeys and animal models of Parkinsons s disease.  
    http://www.emory.edu/NEUROSCIENCE/Smith/LabProjects.htm

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