Autoimmune Disorder It is also called acute inflammatory demyelinating polyneuropathy, acute idiopathicpolyradiculneuritis, acute idiopathic polyneuritis and Landry s ascending http://www.fact-index.com/a/au/autoimmune_disorder.html
Extractions: Main Page See live article Alphabetical index An autoimmune disorder arises from an overactive immune response of the body against substances and tissues normally present in the body. The causes of autoimmune disorders are still obscure, and most of these disorders are probably the result of multiple circumstances: for example, a genetic predisposition triggered by an infection. Women tend to be affected more often by autoimmune disorders, nearly 79% of autoimmune disease patients in the USA are women . It is not known why this is the case, although hormone levels have been shown to affect the severity of the disease See also : Immune system. Below is a listing of actual and suspected autoimmune disorders, with brief descriptions and pointers to full articles. Guillain-Barré syndrome (GBS), is an acquired immune-mediated inflammatory disorder of the peripheral nervous system (e.g. not the brain and spinal column). It is also called acute inflammatory demyelinating polyneuropathy, acute idiopathic polyradiculneuritis, acute idiopathic polyneuritis and Landry's ascending paralysis. Lupus erythematosus is an is a chronic (long-lasting) autoimmune disease where the immune system, for unknown reasons, becomes hyperactive and attacks normal tissue. This attack results in inflammation and brings about symptoms.
Extractions: Mono neuritis multiplex is a form of (damage to nerve s outside the brain and spinal cord). callurl('http://www.nlm.nih.gov/medlineplus/ency/article/000782.htm'); CreateTd(1) Mono neuritis multiplex is a form of (damage to nerve s outside the brain and spinal cord). It involves isolated damage to at least 2 separate nerve areas. Mono neuritis multiplex is a group of symptoms, not a distinct disease entity. callurl('http://www.umm.edu/ency/article/000782.htm'); CreateTd(2) Mono neuritis Multiplex
Extractions: Home About Mayo Clinic Contact Us Mayo Clinic Locations: Arizona Florida Minnesota Mayo Clinic ... Medical Services (Synonyms: GBS, Landry-Guillain-Barré syndrome, Landry's ascending paralysis, acute idiopathic polyneuritis, infectious polyneuritis, acute inflammatory demyelinating polyneuropathy) While most people with GBS regain normalcy, the length and degree of recovery varies. About 5 to 15 percent have serious, lifelong disabilities. Treatment Options Treatment programs are tailored to each individual and take into account the needs of the patient and family, while providing state-of-the-art care and access to new developments and appropriate clinical trials. Mayo Clinic offers comprehensive, coordinated care for patients at any stage of GBS, including: Plasmapheresis (plasma exchange) In this treatment, the blood is "cleansed" of damaging antibodies. A small, thin tube called a catheter is inserted into the vein of one arm; another tube is inserted into the opposite hand or foot. Blood is withdrawn from one tube and sent to a "cell separator" device, which separates the red blood cells from the plasma (the liquid part of the blood). The blood cells are then combined with replacement fluids and returned to the body through the other tube. Intravenous immunoglobulin (IVIg)
Extractions: (advertisement) Synonyms, Key Words, and Related Terms: acute idiopathic polyneuritis, acute inflammatory demyelinating polyneuropathy, ascending paralysis, Guillain-Barré syndrome, Guillain-Barré-Strohl syndrome, AIDP Background: Acute inflammatory demyelinating polyneuropathy (AIDP) is an autoimmune process that is characterized by progressive weakness and mild sensory changes. Many variants exist. In the West, the most common presentation is a subacute ascending paralysis. This is associated with distal paresthesias and loss of deep tendon reflexes. The condition usually plateaus after about 2-3 weeks before slowly improving. In 1859, Landry described 10 cases characterized by ascending paralysis and sensory changes. During World War I, Guillain, Barré, and Strohl described a series of patients with a similar presentation and decreased or absent deep tendon reflexes. They also described albuminocytologic dissociation in the cerebrospinal fluid (CSF), ie, increased CSF protein in the absence of increased WBCs. This allowed them to differentiate AIDP from poliomyelitis, the most common acute paralytic syndrome of that era. (AIDP often is referred to as Guillain-Barré syndrome [GBS]). Myelin breakdown and axonal degeneration were observed in nerve biopsies from patients with AIDP by Haymaker and Kernohan in 1949. An allergic etiology was suggested by Krucke in 1955 after he observed lymphocytic infiltrates within biopsy specimens. An autoimmune process was supported by Waksman and Adams when they created the experimental allergic neuritis model by injecting peripheral nerve tissue into rodents.
Extractions: (advertisement) Synonyms, Key Words, and Related Terms: GBS, acute idiopathic polyneuritis Background: Classic Guillain-Barré syndrome (GBS) is an acute, ascending, and progressive neuropathy characterized by weakness, paresthesias, and hyporeflexia. In severe cases, muscle weakness may lead to respiratory failure. Severe labile autonomic dysfunction also may occur. Maximal weakness typically occurs 2 weeks after the initial onset of symptoms but may evolve early and abruptly. In the early 1900s, Guillain, Barré, and Strohl first described the syndrome in 2 patients who spontaneously recovered from a progressive ascending motor weakness with areflexia, paresthesias, sensory loss, and an elevated level of cerebrospinal fluid (CSF) protein. Since the polio vaccine came into widespread use, GBS has become the most common remaining cause of acute neuromuscular paralysis. Pathophysiology: Most patients with GBS exhibit absent or profoundly delayed conduction in action nerve fibers. This aberrant conduction results from demyelination of nerve cell axons. Peripheral nerves and spinal roots are the major sites of demyelination, but cranial nerves also may be involved. GBS is believed to result from an autoimmune response triggered by an antecedent illness or by any of a long list of medical conditions. The autoimmune response seems to have both humoral and cell-mediated components. In most patients with GBS, symptoms result from injury to the myelin sheath. In a subset of patients with GBS, axonal damage results from a direct cellular immune attack on the axon itself.
Extractions: printer friendly home more about us in your area ... how you can help search this site Please use the Index below to access the condition on which you require information. If you do not find what you want in the Index then try our search facility in the navigator on the left. Contact a Family also has information on many other specific conditions and rare disorders. If you cannot find the information you require in The Contact a Family Directory Online , you may wish to use our Contact a Family Helpline service. aAA see Acquired Aplastic Anaemia
Neurology Table Of Contents (August 1 1976, 26, [8]) 800801. S Stolzberg Letter acute idiopathic polyneuritis no residuaafter steroid treatment? Neurology 1976 26 801-802. To see http://www.neurology.org/content/vol26/issue8/index.shtml
ADVANCE For Health Information Professionals Editorial difficulties. The patient was diagnosed as having acute respiratory failureand acute idiopathic polyneuritis (GuillainBarre syndrome). The http://www.advanceforhim.com/common/editorial/editorial.aspx?CC=23671
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MedlinePlus Drug Information: Mercaptopurine many types of autoimmune diseases such as systemic lupus erythematosus, rheumatoidarthritis, acute idiopathic polyneuritis, acute idiopathic nephrotic syndrome http://www.nlm.nih.gov/medlineplus/druginfo/medmaster/a682653.html
Extractions: Contents of this page: Your doctor has ordered the drug mercaptopurine to help treat your illness. The drug is taken by mouth in tablet form. This medication is used to treat: This medication is sometimes prescribed for other uses; ask your doctor or pharmacist for more information. Mercaptopurine belongs to a group of drugs known as antimetabolites. It resembles a normal cell nutrient needed by cancer cells to grow. The cancer cells take up mercaptopurine which then interferes with their growth. Mercaptopurine also is used to treat many types of autoimmune diseases such as systemic lupus erythematosus, rheumatoid arthritis, acute idiopathic polyneuritis, acute idiopathic nephrotic syndrome, psoriatic arthritis, erythroid aplasia, or myelofibrosis; idiopathic hemolytic anemia; macroglobulinemia; idiopathic thrombocytopenia purpura; idiopathic pulmonary hemosiderosis; multiple sclerosis; myasthenia gravis; uveitis; and ulcerative colitis. Talk to your doctor about the possible risks of using this drug for your condition. Before taking mercaptopurine
MedlinePlus Medical Encyclopedia: Guillain-Barre Syndrome LandryGuillain-Barre syndrome; acute idiopathic polyneuritis; Infectious polyneuritis;Acute inflammatory polyneuropathy Definition Return to top. http://www.nlm.nih.gov/medlineplus/ency/article/000684.htm
Extractions: @import url(http://www.nlm.nih.gov/medlineplus/images/advanced.css); Skip navigation Other encyclopedia topics: A-Ag Ah-Ap Aq-Az B-Bk ... Z Contents of this page: Superficial anterior muscles Alternative names Return to top Landry-Guillain-Barre syndrome; Acute idiopathic polyneuritis; Infectious polyneuritis; Acute inflammatory polyneuropathy Definition Return to top Guillain-Barre syndrome is a disorder caused by nerve inflammation involving progressive muscle weakness or paralysis , which often follows an infectious illness. Causes, incidence, and risk factors Return to top Guillain-Barre syndrome is an acute type of nerve inflammation. The inflammation damages portions of the nerve cell, resulting in muscle weakness or paralysis and sensory loss. The damage usually includes loss of the myelin sheath of the nerve (demyelination ), which slows the conduction of impulses through the nerve. The damage can also cause denervation (kill the axon part of the nerve cell), which stops nerve function entirely. Without the axon, messages cannot be transferred from one nerve cell to another.
Arch Intern Med -- Abstracts: Kabins Et Al. 136 (1): 100 acute idiopathic polyneuritis caused by cytomegalovirus. Cytomegalovirus wasisolated from the PBBC from a patient with acute idiopathic polyneuritis. http://archinte.ama-assn.org/cgi/content/abstract/136/1/100
Extractions: Vol. 136 No. 1, January 1, 1976 Featured Link E-mail Alerts ARTICLE Article Options Send to a Friend Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Kabins S Ali MA Contact me when this article is cited S. Kabins, R. Keller, R. Peitchel and M. A. Ali Cytomegalovirus was isolated from the PBBC from a patient with acute idiopathic polyneuritis. Viremia occurred during the early phase of the neurologic disease, preceding viruria, and was accompanied by specific CMV-IgM antibodies, which indicates a recently acquired infection. Complement fixing antibodies to CMV remained stable throughout the illness at a relatively low titer. Previously, CMV has been associated with acute idiopathic polyneuritis on the indirect evidence of rising or elevated titers of complement fixing antibodies or isolation of virus from urine or saliva or both. Our findings provide direct evidence for the causative role
