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         Reactive Arthritis:     more detail
  1. Psoriatic and Reactive Arthritis: A Companion to Rheumatology by Christopher Ritchlin MD, Oliver FitzGerald, 2007-05-08
  2. Reactive Arthritis by Paavo Toivanen, Auli Toivanen, 1988-08-31
  3. Ankylosing Spondylitis and Reactive Arthritis
  4. Integrative chiropractic treatments for ankylosing spondylitis and reactive arthritis.: An article from: Original Internist by Alex Vasquez, 2006-06-01
  5. Pustular Dermatitis: Reactive arthritis, Eosinophilic folliculitis, Subcorneal pustular dermatosis, Pustulosis, Reiter's disease
  6. Make ARTHRITIS PAIN Disappear - A personal ACTION PLAN!
  7. Best Arthritis Remedies: Triumph Over Arthritis by Bourdin LeBock, 2010-04-29
  8. A Companion to Rheumatology Psoriatic & Reactive Arthritis by Ritchlin, 2007-01-01
  9. Severity, duration of rheumatoid arthritis predict CVD.(RHEUMATOLOGY)(cardiovascular disease)(Clinical report): An article from: Internal Medicine News by Mitchel L. Zoler, 2009-09-01
  10. Acne fulminans in Marfan syndrome.(CASE REPORTS): An article from: Journal of Drugs in Dermatology by Uwe Wollina, Gesina Hansel, et all 2005-07-01

81. Entrez PubMed
Salmonellatriggered reactive arthritis use of polymerase chain reaction, immunocytochemical staining, and gas chromatography-mass spectrometry in the
http://www.biomedcentral.com/pubmed/9920018
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Arthritis Rheum. 1999 Jan;42(1):84-9. Related Articles, Links
Salmonella-triggered reactive arthritis: use of polymerase chain reaction, immunocytochemical staining, and gas chromatography-mass spectrometry in the detection of bacterial components from synovial fluid. Nikkari S, Rantakokko K, Ekman P, Mottonen T, Leirisalo-Repo M, Virtala M, Lehtonen L, Jalava J, Kotilainen P, Granfors K, Toivanen P.

82. MANAGEMENT OF REACTIVE ARTHRITIS IN A JUVENILE GORILLA (
31, No. 4, pp. 539–551. MANAGEMENT OF reactive arthritis IN A JUVENILE GORILLA (GORILLA GORILLA GORILLA) WITH LONGTERM SULFASALAZINE THERAPY.
http://apt.allenpress.com/aptonline/?request=get-abstract&issn=1042-7260&volume=

83. Reactive Arthritis
reactive arthritis. artritis reactive arthritis. Learn More About Living With Osteoarthritis Get information about arthritis pain relief here.
http://www.makali.net/artritis--reactive-arthritis/
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84. Bacterium-specific Synovial Lymphocyte Proliferation In Reactive Arthritis
Longitudinal investigation of bacteriumspecific synovial lymphocyte proliferation in reactive arthritis and lyme arthritis. The
http://www.cfsresearch.org/chlamydia/linkschronic/20nf.htm
The British Journal of Rheumatology, Vol 37, 784-788,
ORIGINAL PAPERS
Longitudinal investigation of bacterium-specific synovial lymphocyte proliferation in reactive arthritis and lyme arthritis
C Fendler, P Wu, U Eggens, S Laitko, H Sorensen, A Distler, J Braun and J Sieper
Deutsches Rheumaforschungszentrum, Department of Nephrology and Rheumatology, Klinikum Benjamin Franklin, Berlin, Germany.
Reprinted with permission of Oxford University Press and the British Society for Rheumatology
Abstract: BACKGROUND: Antigen-specific lymphocyte proliferation of synovial fluid mononuclear cells (SF MNC) has been reported repeatedly in reactive arthritis and Lyme arthritis; however, less information is available on serial investigations of SF MNC in the same patients. METHODS: In this study, the synovial lymphocyte proliferation to Yersinia, Chlamydia, Shigella and Borrelia burgdorferi was investigated sequentially at different time points in 28 patients with reactive arthritis, undifferentiated oligoarthritis or Lyme arthritis responding to one of these bacteria. RESULTS: The same bacterium was always recognized in arthritis triggered by Chlamydia, Shigella or Borrelia, with much variation in the proliferative response. Only the Yersinia-specific responses changed specificity, suggesting that the proliferative response to Yersinia is non-specific in some patients.

85. Entrez PubMed
Click here to read Dietary glycine prevents peptidoglycan polysaccharideinduced reactive arthritis in the rat role for glycine-gated chloride channel.
http://arthritis-research.com/pubmed/11500467
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Infect Immun. 2001 Sep;69(9):5883-91. Related Articles, Links
Dietary glycine prevents peptidoglycan polysaccharide-induced reactive arthritis in the rat: role for glycine-gated chloride channel. Li X, Bradford BU, Wheeler MD, Stimpson SA, Pink HM, Brodie TA, Schwab JH, Thurman RG.

86. Entrez PubMed
Aetiological role of bacteria associated with reactive arthritis in pauciarticular juvenile chronic arthritis. Sieper J, Braun J
http://arthritis-research.com/pubmed/1466598
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Ann Rheum Dis. 1992 Nov;51(11):1208-14. Related Articles, Links
Aetiological role of bacteria associated with reactive arthritis in pauciarticular juvenile chronic arthritis. Sieper J, Braun J, Doring E, Wu P, Heesemann J, Treharne J, Kingsley G.

87. Auto-immune Disease With Formation Of Reactive Arthritis - Medical Illustration
Autoimmune Disease with Formation of reactive arthritis - Medical Illustration, Auto-immune Disease with Formation of reactive arthritis - Medical Illustration.
http://findlaw.doereport.com/generateexhibit.php?ID=430

88. Reactive Arthritis (Reiter's Syndrome)
About YMG. Search YMG. reactive arthritis (Reiter s Syndrome). What is reactive arthritis? Reactive Who is affected by reactive arthritis? Reactive
http://ymghealthinfo.org/content.asp?pageid=P00064

89. Reactive Arthritis Meds Online
reactive arthritis Medications Hydrocodone Lortab Lorcet Norco Zydone Tylenol 3 CodeineVicodin VicoProfen Fioricet Butalbital No Prescription Required.
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    Arthritis is one of the most pervasive diseases in the United States and is the leading cause of disability. According to the Centers for Disease Control and Prevention one out of every three Americans (an estimated 70 million people) is affected by one of the more than 100 types of arthritis. For most people arthritis pain and inflammation cannot be avoided as the body ages. In fact, most people over the age of 50 show some signs of arthritis. Joints naturally degenerate over time. Fortunately, arthritis can be managed through a combination of medication, exercise, rest, weight-management, nutrition, and, in some cases, surgery. Your doctor can tell if you have arthritis through blood tests and x-rays. He or she will then be able to help you decide on the best treatment for your case. Arthritis is a chronic disease that will be with you for a long time and possibly for the rest of your life. Your treatments will probably change over time and medication may be adjusted. Having a positive mental outlook and the support of family and friends will help you live with arthritis and be able to continue to perform your daily activities.

90. The Immunology Of The Carditis In Post-Streptococcal Reactive Arthritis
CRC Number 9502. The Immunology of the Carditis in Post-Streptococcal reactive arthritis. Abstract. Principal Investigator David K. Kurahara, MD.
http://www.pbrc.hawaii.edu/crc/studies/crc95-02.htm
CRC Number:
The Immunology of the Carditis in Post-Streptococcal Reactive Arthritis
Abstract
Principal Investigator: David K. Kurahara, MD Post-Streptococcal Reactive Arthritis (PSRA) is a prolonged arthritis following streptococcal infection that carries a significant risk of carditis in clinical studies here in Hawaii. Despite this finding of carditis in these patients, it is not clear if PSRA is a variant of Acute Rheumatic Fever (ARF), since there are no pathogenic studies of PSRA to date. It is similarly unclear if these patients require antibiotic prophylaxis. Our major aim is to study the pathogenesis of PSRA. Specific Aims and Hypotheses: 1) To document the presence of heart active antibodies in established and acute PSRA patients. 2) To determine the specificity of these heart reactive antibodies and to establish if they are cross-reactive with the Group A streptococcus organism. 3) To describe the incidence of the ARF specific B cell alloantigen which is identified by monoclonal antibody D8/17, in PSRA patients. If cross-reactive heart antibodies are found in PSRA patients and they are found to have the ARF specific B call alloantigen then this would give laboratory evidence that PSRA is indeed a variant of ARF. Post streptococcal reactive arthritis remains a disease that is hardly explored to date. The pathogenesis of the cardiac disease has not been investigated. Results from this study would significantly increase the information about the possible etiology of the cardiac disease. This research has strong clinical application as currently it is unclear if antibiotic prophylaxis should be given to all PSRA patients. Laboratory data and follow up study of these patients would help to strengthen this recommendation, especially if PSRA patients reveal similarities to ARF patients. It is well known how severe the cardiac inflammation in ARF can be, and this can possibly be prevented in PSRA patients by more active studies of this disease.

91. Abstract: Ankylosing Spondylitis, Psoriatic Arthritis, And Reactive Arthritis Sh
Ankylosing Spondylitis, Psoriatic Arthritis, and reactive arthritis Show Increased Bone Resorption, But Differ with Regard to Bone Formation
http://www.jrheum.com/abstracts/abstracts02/1430.html
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Ankylosing Spondylitis, Psoriatic Arthritis, and Reactive Arthritis Show Increased Bone Resorption, But Differ with Regard to Bone Formation JOHANNES GRISAR, PETER M. BERNECKER, MARTIN ARINGER, KURT REDLICH, MARKUS SEDLAK, WOLFGANG WOLOZCSZUK, SUSANNE SPITZAUER, STEPHAN GRAMPP, FRANZ KAINBERGER, WOLFGANG EBNER, JOSEF S. SMOLEN, and PETER PIETSCHMANN
Objective.
To test if markers of bone metabolism are altered in patients with seronegative spondyloarthropathies (SSpA). We studied biochemical markers of bone resorption and bone formation, osteoprotegerin (OPG), and bone mineral density (BMD) in patients with psoriatic arthritis (PsA), ankylosing spondylitis (AS), and reactive arthritis (ReA) and healthy volunteers. The bone resorption markers urinary deoxypyridinoline and crosslinked telopeptide of collagen-I were significantly increased in patients with AS, PsA, and ReA; in PsA they correlated with the acute phase response (C-reactive protein and erythrocyte sedimentation rate). The bone formation markers were divergent: bone-specific alkaline phosphatase was increased in PsA, but not in AS or ReA. Osteocalcin levels were only elevated in AS. Serum levels of OPG were significantly increased in both AS and PsA. Dual energy x-ray absorptiometry (DEXA) measurements of lumbar spine and femoral neck revealed osteopenia in patients with AS, whereas the DEXA distribution was within normal range in PsA. Our data indicate high and, particularly in AS, unbalanced bone turnover in SSpA, consistent with the decrease in BMD found in patients with AS. (J Rheumatol 2002;29:1430-6)

92. Abstract: Reactive Arthritis And Other Sequelae Following Sporadic Salmonella Ty
reactive arthritis and Other Sequelae Following Sporadic Salmonella typhimurium Infection in British Columbia, Canada A Case Control Study
http://www.jrheum.com/abstracts/abstracts02/2154.html
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Reactive Arthritis and Other Sequelae Following Sporadic Salmonella typhimurium Infection in British Columbia, Canada: A Case Control Study JANE A. BUXTON, MURRAY FYFE, SAMARA BERGER, MICHELLE B. COX, KIMBERLY A. NORTHCOTT, and the Multiprovincial Salmonella typhimurium Case-Control Study Group ABSTRACT
Objective
. To describe sequelae occurring in the 3 months after sporadic Salmonella typhimurium (ST) infection in British Columbia (BC), Canada. We compared the incidence of sequelae to similar symptoms in controls; identified risk factors for developing sequelae; identified the incidence of reactive arthritis (ReA) as diagnosed by a rheumatologist, and assessed primary care physician diagnosis of ReA. A questionnaire was administered by telephone to cases of ST occurring in BC between December 1, 1999, and November 30, 2000; and to controls obtained from the BC provincial client registry. Cases reporting symptoms were followed up by a rheumatologist. Thirty-five of 66 (53%) cases reported any symptom, 17 (26%) reported joint symptoms. The Mantel-Haenszel odds ratio (weighted by sex and pediatric/adult) of a salmonella case reporting "any symptom" compared to controls was 5.42; 95% confidence interval (CI) 2.18-16.27; and reporting joint symptoms was 4.40; 95% CI: 1.25-19.53. The sex distribution of cases reporting joint symptoms was not significantly different. No medication taken during the salmonella infection was significantly different between the cases who had joint symptoms and those who did not. Four cases (2 adults, 2 children) were considered by the rheumatologist to have symptoms consistent with ReA, 2 of these had been told by a physician that their symptoms were related to their ST infection.

93. Reactive Arthritis
reactive arthritis.
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94. JW (General) -- Sign In
reactive arthritis MAY BE DUE TO YERSINIA ANTIGENS. reactive arthritis is frank arthritis that develops after infection at another site in the body.
http://general-medicine.jwatch.org/cgi/content/full/1989/203/9

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95. PIER Patient Info - Reactive Arthritis
reactive arthritis. American Academy of Family Physicians reactive arthritis What You Should Know. Arthritis Foundation Reiter s Syndrome (reactive arthritis).
http://www.acponline.org/fcgi/pierpi.pl?module=d355

96. NewYork-Presbyterian Hospital Health A To Z Reactive Arthritis
Health information about reactive arthritis (Reiter s Syndrome) from NewYorkPresbyterian. The University Hospitals of Columbia and Cornell.
http://wo-pub2.med.cornell.edu/cgi-bin/WebObjects/PublicA.woa/2/wa/viewHContent?

97. Entrez PubMed
Detection of Chlamydia trachomatis DNA in joints of reactive arthritis patients by polymerase chain reaction. TaylorRobinson D, Gilroy CB, Thomas BJ, Keat AC.
http://genomebiology.com/pubmed/1352016
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Lancet. 1992 Jul 11;340(8811):81-2. Related Articles, Links
Detection of Chlamydia trachomatis DNA in joints of reactive arthritis patients by polymerase chain reaction. Taylor-Robinson D, Gilroy CB, Thomas BJ, Keat AC.

98. OHSU Health - Reactive Arthritis (Reiter's Syndrome)
Detailed information on reactive arthritis, including causes, symptoms, diagnosis, and treatment. Email. reactive arthritis (Reiter s Syndrome).
http://www.ohsuhealth.com/htaz/arthritis/common/reactive_arthritis_reiters_syndr

Arthritis and Other Rheumatic Diseases Home Page
Common Types of Arthritis and Other Rheumatic Diseases Ankylosing Spondylitis Bursitis ... Raynaud's Phenomenon Reactive Arthritis (Reiter's Syndrome) Rheumatoid Arthritis Scleroderma Systemic Lupus Erythematosus (Lupus) Tendonitis ...
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Reactive Arthritis (Reiter's Syndrome)
What is reactive arthritis?
Reactive arthritis, also known as Reiter's syndrome, is a type of arthritis that occurs as a reaction to an infection somewhere in the body. Most infections that cause the disease originate in the genitourinary tract (the bladder, urethra, penis, or vagina) and are spread through sexual intercourse, a form of the disease called genitourinary Reiter's syndrome, or urogenital Reiter's syndrome. Other infections that can cause reactive arthritis include gastrointestinal infections due to eating contaminated food or handling contaminated substances, a form of the disease called gastrointestinal Reiter's syndrome, or enteric Reiter's syndrome.
Who is affected by reactive arthritis?
Reactive arthritis is characterized by inflamed joints and affects mostly young men, between the ages of 20 and 40. Although researchers are not sure why some people develop reactive arthritis in response to certain infections, a genetic factor (presence of the HLA-B27 gene) seems to increase the risk.

99. Arthritis & Other Rheumatic Diseases - Reactive Arthritis (Reiter's Syndrome)
reactive arthritis (Reiter s Syndrome) What is reactive arthritis? reactive arthritis syndrome. Who is affected by reactive arthritis? Reactive
http://www.muschealth.com/arthritis/reactive.htm
Reactive Arthritis (Reiter's Syndrome) What is reactive arthritis?
Reactive arthritis, also known as Reiter's syndrome, is a type of arthritis that occurs as a reaction to an infection somewhere in the body. Most infections that cause the disease originate in the genitourinary tract (the bladder, urethra, penis, or vagina) and are spread through sexual intercourse, a form of the disease called genitourinary Reiter's syndrome, or urogenital Reiter's syndrome. Other infections that can cause reactive arthritis include gastrointestinal infections due to eating contaminated food or handling contaminated substances, a form of the disease called gastrointestinal Reiter's syndrome, or enteric Reiter's syndrome. W ho is affected by reactive arthritis?
Reactive arthritis is characterized by inflamed joints and affects mostly young men, between the ages of 20 and 40. Although researchers are not sure why some people develop reactive arthritis in response to certain infections, a genetic factor (presence of the HLA-B27 gene) seems to increase the risk. What causes reactive arthritis?

100. MANAGEMENT OF REACTIVE ARTHRITIS IN A JUVENILE GORILLA (
31, No. 4, pp. 539–551. MANAGEMENT OF reactive arthritis IN A JUVENILE GORILLA (GORILLA GORILLA GORILLA) WITH LONGTERM SULFASALAZINE THERAPY.
http://www.bioone.org/bioone/?request=get-abstract&issn=1042-7260&volume=031&iss

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