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         Lipoid Nephrosis:     more detail

61. Roche Lexikon Medizin (4. Aufl.) - Lipoidnephrose
Translate this page Lipoid nephrose. engl. lipoid nephrosis. glomeruläre Minimalveränderungen.Verwandte Themen. glomeruläre Minimalveränderungen.
http://www.gesundheit.de/roche/ro22500/r22648.html
Lipoid nephrose engl.: lipoid nephrosis
Verwandte Themen

62. AllRefer Health - Minimal Change Disease (Idiopathic Nephrotic Syndrome Of Child
Minimal Change Disease (Idiopathic Nephrotic Syndrome of Childhood, lipoid nephrosis,Minimal Change Nephrotic Syndrome, Nil Disease) information center covers
http://health.allrefer.com/health/minimal-change-disease-info.html
AllRefer Channels :: Yellow Pages Reference Health Home ... Contact Us Quick Jump ADD/ADHD Allergies Alzheimer's Disease Arthritis Asthma Back Pain Breast Cancer Cancer Colon Cancer Depression Diabetes Gallbladder Disease Heart Attack Hepatitis High Cholesterol HIV/AIDS Hypertension Lung Cancer Menopause Migraines/Headaches Osteoporosis Pneumonia Prostate Cancer SARS Stroke Urinary Tract Infection 1600+ More Conditions Alternative Medicine Health News Symptoms Guide Special Topics ... Medical Encyclopedia
You are here : AllRefer.com Health Minimal Change Disease
Minimal Change Disease
Definition Prevention
Treatment

Expectations or Prognosis
...
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Alternate Names : Idiopathic Nephrotic Syndrome of Childhood, Lipoid Nephrosis, Minimal Change Nephrotic Syndrome, Nil Disease Definition Minimal change disease is a kidney disorder. Each kidney is made of more than a million units called nephrons. Each nephron has a tuft of blood vessels called a glomerulus, which is surrounded by a Bowman's Capsule. Together, these structures filter blood and form urine. Minimal change disease is one cause of nephrotic syndrome . It is named "minimal change" because the nephrons appear totally normal under a light microscope. The condition is only seen using an electron microscope, which reveals changes in the Bowman's Capsule in the nephrons. Urine tests also show high protein levels.

63. Nefrología. Volumen 4/99 Casos Clínicos. Art.2
Translate this page Kidney Int 33 708-715, 1998. ·4.·Kashgarian M lipoid nephrosis and focal sclerosisdistinct entities or spectrum of disease. Nephron 13 105-108, 1974.
http://www.aulamedica.es/nefrologia/nefro2-00/comunic1e.htm
Volumen XX. Número 2. 2000 INDICE Factores de pronóstico en la evolución de la glomeruloesclerosis segmentaria y focal D. Moreno*, Ch. Magrans**, H. Gómez Barry*** y A. M. Gámez****
*Especialista de Segundo Grado en Nefrología. Jefe del Servicio de Trasplantes del Instituto de Nefrología. **Profesor de Nefrología del Instituto de Nefrología. ***Profesor de Patología. Jefe del Departamento de Patología del Instituto de Nefrología. ****Nefróloga Hospital Julio Trigo.
Instituto de Nefrología "Abelardo Puch López". La Habana. Cuba. Recibido: 14-VI-99.
En versión definitiva: 28-XII-99.
Aceptado: 28-XII-99. Correspondencia: Dr. Darío Moreno Vega
Servicio de Trasplantes
Instituto de Nefrología «Abelardo Buch López»
Ave. 26 y Rancho Boyeros
Plaza Habana, 6
Cuba Subir Las nefropatías glomerulares primarias ocupan un lugar preponderante en el campo de la Nefrología, ya que constituyen una de las primeras causas de insuficiencia renal crónica . Aunque la etiología de este conjunto de enfermedades no está totalmente dilucidada en el momento actual, su patogenia, aun siendo compleja, está mejor conocida, ya que a partir de los trabajos de Brenner han emergido un conjunto de conocimientos sobre los mecanismos de lesión glomerular, desencadenados a partir de estímulos, sean éstos de carácter inmunológico o de otra índole, que conllevan en última instancia la pérdida de unidades funcionales

64. Arch Intern Med -- Abstracts: Hulter And Bonner 140 (3): 403
lipoid nephrosis appearing as acute oliguric renal failure. HN Hulterand EL Bonner Jr. Acute oliguric renal failure previously was
http://archinte.ama-assn.org/cgi/content/abstract/140/3/403
Select Journal or Resource JAMA Archives of Dermatology Facial Plastic Surgery Family Medicine (1992-2000) General Psychiatry Internal Medicine Neurology Ophthalmology Surgery Student JAMA For The Media Classified Ads Meetings Peer Review Congress
Vol. 140 No. 3, March 1, 1980 Featured Link E-mail Alerts ARTICLE Article Options Send to a Friend Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Hulter HN Bonner EL Articles that cite this article Contact me when this article is cited
Lipoid nephrosis appearing as acute oliguric renal failure
H. N. Hulter and E. L. Bonner Jr
Acute oliguric renal failure previously was reported to develop in patients with preexisting idiopathic nephrotic syndrome in association with clinical evidence of vascular volume depletion. We describe an 81-year-old man without recent proteinuria or evidence of preexisting nephrotic syndrome in whom acute oliguric renal failure developed. Renal biopsy disclosed minimal change disease. Nephrotic range proteinuria without severe hypoalbuminemia

65. SUNY Stony Brook Pathology Department HBP310 Urinary And Male Genital Outline
n Several types. Nephrotic Diseases. w Many causes. n lipoid nephrosis (nil disease).n Membranous glomerulonephritis. n Focal segmental sclerosis. w Several causes.
http://www.path.sunysb.edu/coursemat/hbp310urinarymg_outline.htm
URINARY AND MALE GENITAL OUTLINE
Male Genitourinary System
w Dual function
w Congenital abnormalities common
w Infertility
n Cryptorchidism
Infection
w Urethritis
n Chlamydia
n Gonorrhea
w Ulcers and condyloma
n Herpes
n Syphilis
w Hematocoele
w Torsion
w Varicocoele
Tumors
w Germ cell origin
w Parallel to ovarian tumors
w Cryptorchidism
w Seminoma most common
w Self diagnosis
Prostate
w Hyperplasia
n Hormonal
n Obstructive
w Symptoms
w Cancer
n Most common, 3rd in fatality
n Age related
n Cause unknown
n Spread to bone
Bladder
w Cancer
n Environmental
w Infection
n Instrumentation
n Colonization
n Reflux
n Flora
Renal Syndromes
w Acute renal failure
n Oliguria
n Anuria
w Nephritic
w Nephrotic
w Isolated Hematuria
w Tubular Defects (metabolic)
Kidney
w Infection
n Acute
w Ascending
n Own flora n Reflux
w Hematogenous
n Chronic
n Stones
Tumors
w
w Adenocarcinoma
w
w Horseshoe kidney
w Dysplasia
w Autosomal Dominant Adult Polycystic Kidney Disease (ADPK)
n Cysts lead to renal failure
n Hypertension
n Berry aneurysm
w Other cysts
Nephritic and Nephrotic Syndrome
w Nephrotic
n Loss of protein
n Decreased serum albumin
n Edema
w Nephritic
n Oliguria
n Azotemia (Urea, Creatinine)

66. Conventional Therapy
(MCGN, minimal proliferative intercapillary glomerulonephritis with nephrotic syndrome(MPI + NS), glomerular minimal changes, lipoid nephrosis, minimal change
http://www.uni-tuebingen.de/uni/kmp/cgts5e.htm
Next Back Home
Conventional Therapy
Patient Recruitment Stopped
Primary Glomerulonephritis
General information
Objective
Conventional therapy protocols are assigned to a patient by random numbers in the case of several available treatment modalities for one and the same form of glomerulonephritis. The treatment effect is compared by a randomized, controlled, open trial. The randomization is done on-site at each centre by a computer program (random number generator).
Inclusion criteria:
The inclusion criteria for the treatment following the underneath mentioned therapy protocol are: For all glomerular diseases except IgA-Nephropathy: 1. Proteinuria over 3.5 g/die, measured three times in series IgA-Nephropathy: Histologically proven diagnosis independently of serum creatinine or proteinuria. If inclusion criteria are not given and the patient is suffering from biopsy proven glomerulonephritis patients data and follow-up are nevertheless registered in the central data bank. Analysis on this subgroup of patients will be performed separately.
Study endpoint
The study endpoint is determined five years after inclusion. An intermittent analysis of the data will be done after six months.

67. Laboratory #3 - Renal
Supplement No Glass Slides lipoid nephrosis; Focal Segmental Glomerulosclerosis;Membranoproliferative Glomerulonephritis (MPGN).
http://www.umdnj.edu/pathnweb/syspath/syslab_3/syslab_3.htm
Lab 3 Slides 70 Slides 51 Supplement ... Slides 28 Welcome to the Systemic Pathology Laboratory #3: Renal, Urinary Disease, and Male GU Please select a slide group to view by clicking on the appropriate slide button or text found on the left or top navigation bars. The contents of the each slide group are listed in the index below. Alternatively, you can click on the continue text below to proceed to the first slide group. To return to the Pathology Course Menu select from the text navigator bar below. Slides 70: Glomerulonephritis, Rapidly Progressive Slides 51: Membranous Glomerulopathy Supplement : No Glass Slides
  • Lipoid Nephrosis Focal Segmental Glomerulosclerosis Membranoproliferative Glomerulonephritis (MPGN)
Slide 41: Acute Proliferative Glomerulonephritis, with Early Crescents Slides 40: Polycystic Disease (Adult, Autosomal Dominant) Slides 28: Prostate: Adenocarcinoma Continue to Slide 70 Lab 3 Slides 70 Slides 51 ... UMDNJ
Please direct comments and questions about the Pathology Medical Student pages to izaguial@umdnj.edu

68. Nephrology Terms
entry LifeSite™ hemodialysis venous access system see MT Desk glossary entryline of Toldt lipoid nephrosis minimal-change disease lithium Lithospec
http://www.mtdesk.com/lstneph.shtml

Andrews School for Medical Transcription
NEPHROLOGY TERMS Back to MT DESK Back to Index of Terminology A-M N-Z Back to Word List Index Nephrology Terms
Tc-DMSA
Tc-DTPA
Tc-labeled HAG3 [investigational imaging agent]
Tc-labeled MAG3
Tc-MAG3
access blood flow
access flow measurement
access recirculation (AR)
ACE (angiotensin-converting enzyme)
ACE inhibitor ACEI (angiotensin-converting enzyme inhibitor) acid-base homeostasis acidification acquired cystic disease [dialysis-associated cystic disease] acquired renal insufficiency Acucise® endopyelotomy [balloon] catheter - Applied Medical Resources Acucise endopyelotomy acute nephritic syndrome acute poststreptococcal glomerulonephritis (ASPGN) acute renal failure (ARF) acute tubular necrosis (ATN) ADPKD (autosomal dominant polycystic kidney disease) adrenal gland adult-onset medullary cystic disease aerobic bacteria air embolism Aksys PHD™ system [investigational hemodialysis system for home use] - see MT Desk glossary entry Alcaligenes xylosoxidans ALG (antilymphocyte globulin) Allen-Brown shunt allergic interstitial nephritis allogenic renal transplant allograft dysfunction AlloMune™ system - see MT Desk glossary entry allopurinol (Zyloprim®) alpha -antitrypsin Alport posttransplant anti-GBM disease Alport syndrome Altra Flux® hemodialyzer - Minntech, Inc.

69. X. DISEASES OF THE GENITOURINARY SYSTEM NEPHRITIS, NEPHROTIC
glomerulonephritis mixed membranous and proliferative glomerulonephritis 581.3 Withlesion of minimal change glomerulonephritis lipoid nephrosis Minimal change
http://www.nber.org/mortality/1995/docs/ch10.txt

70. Surgery Of Modern Warfare
There is a disease of the kidneys that used to be called lipoid nephrosis, andit causes children to present with swelling all over their bodies and renal
http://www.surgeryofmodernwarfare.com/archives/july2103/july2103.html
Surgery of Modern Warfare
A poem about a pathologist
and a cadaver poem
by Surgery's very first surgeon-contributor,
Noah Raizman
The Pathologist Discusses His Life The foot processes fuse and
efface themselves, despite this being a disease of minimal
change, the nil disease, wherein the binding substance leaves the blood,
drawing with it the body1s water; and without the binder,
pressure forces the water out, so little left in the blood,
we hold it back, puff ourselves out, a disobedient child holding his breath and refusing to move we classify this entity as a single point in our cartography of suffering, this disease of minimal change, the patients of stasis, young and bedridden they are the worst kind, but some are like me, that is to say old and sclerosed, a lifetime of deposition, microscopically obliterated but if instructed to look closer, with the advantage of stains and metals the movement is ultrastructural which is perhaps true of everything but what can be said of that visible only to electrons? Only this: we see it through their starry, charged eyes.

71. NEPHRITIC And NEPHROTIC SYNDROMES
Age Group Affected, Treatment and Outcome. NEPHROTIC SYNDROME Proteinuria, Hypoalbuminemia,Edema, Hyperlipidemia. Minimal Change Disease (lipoid nephrosis).
http://www.student.med.umn.edu/class2001/nephrotic-nephritic.htm
NEPHRITIC and NEPHROTIC SYNDROMES Disease Most Frequent Clinical Presentation Pathogenesis Light Microscope F.M. (Fluorescence Microscope) E.M. (Electron Microscope) Age Group Affected Treatment and Outcome NEPHROTIC SYNDROME Proteinuria, Hypoalbuminemia,
Edema, Hyperlipidemia Minimal Change Disease (Lipoid Nephrosis)
  • Selective proteinuria (Albumin)
Loss of foot processes Loss of GBM polyanionic sites Appearance of villi on epithelial cells Normal Lipid in tubules F.M. = negative E.M. = loss of foot processes, lipid vacuoles #1 cause of Nephrotic Syndrome in children, esp. boys younger than 6 yrs. old. Responds well to corticosteroids. No progression into chronic renal failure Focal Segmental Glomerular Sclerosis
  • Non-selective proteinuria Hypertension Microscopic hematuria
Idiopathic Lower renal mass (in obese) causes: heroin use, HIV Focal and segmental sclerosis Hyalinosis Hypercellular mesangium Thick B.M.

72. Answer List 1, 1996
Tumor L Lactic Dehydrogenase Laparoscopy Legionella Pneumophila Leiomyoma LeiomyosarcomaLeukotrienes Lichen Sclerosus lipoid nephrosis Lupus Nephritis Lymph
http://www.kumc.edu/instruction/medicine/pathology/ed/exams/ans1_96.html
Answer List #1, 1996
Click on a circle to insert into currently selected question A-
Abrasion
Abscess
Acid Phosphatase
Acute Interstitial Nephritis
Acute Tubular Necrosis
Adenovirus
Adult Polycystic Kidney Disease
Adult Respiratory Distress Syndrome (ARDS)
Alkaline Phosphatase
Allergic Acute Interstitial Nephritis Allergic Bronchopulmonary Aspergillosis Alpha-1-Antitrypsin Alpha Fetoprotein Alport Syndrome Amyloidosis Analgesic Nephropathy Anorchia Anthracosis Anuria ARDS Artery Arthritis Asbestos Asbestos Body Asbestosis Aspergillosis Aspiration Of Gastric Contents Asthma: Allergic Asthma: Infectious Asthma: Occupational Asthma: Exercise Induced Asthma: Drug Induced Atelectasis Autosomal Dominant Polycystic Kidney Autosomal Recessive Polycystic Kidney Azoospermia B- Bacteremia Benign Familial Hematuria Berger's Disease Bilateral Cortical Necrosis Bladder, Urinary Blastomycosis Bone Bordetella Pertussis Bowen Disease Bowman Capsule Bronchiectasis Bronchiole Bronchiolitis Obliterans-Organizing Pneumonia (B00P) Bronchitis: Acute Bronchitis: Chronic Bronchogenic Cyst Bronchopleural Fistula Bronchopneumonia Bronchoscopy C- Calcium Carbonate Calcium Chloride Calcium Hydroxyapatite Calcium Oxalate/Phosphate Calculus Carcinoid Carcinoma: Adenocarcinoma Carcinoma: Bronchioloalveolar Carcinoma: Carcinoma In Situ Carcinoma: Embryonal Carcinoma Carcinoma: Endometrioid Carcinoma Carcinoma: Infiltrating Duct Carcinoma Carcinoma: Lobular Carcinoma: Medullary Carcinoma: Mucinous Carcinoma: Oat Cell Carcinoma Carcinoma: Prostatic Carcinoma: Renal Cell

73. Katholieke Universiteit Leuven: Research
The heparan sulfate proteoglycans are missing from the glomerular wall in congenitallipoid nephrosis, and accumulate in excessive amounts in the amyloid
http://www.kuleuven.ac.be/research/keydomains/medicine/diagnostic_agents1/med1_2
Home Research activities Research groups Key domains ... Nederlands Research - Key Domain Medicine - Development of new diagnostic agents Monoclonal antibodies
Monoclonal anti-heparan sulfate and heparan sulfate proteoglycan antibodies
Research topics Heparan sulfate proteoglycans are proteins that carry one or more complex carbohydrate chains of the heparan sulfate type. They are expressed at the cell surface and in the extracellular matrix in the immediate vicinity of the cells. The heparan sulfate chains bind and activate a specific group of extracellular matrix components, growth factors, enzymes and enzyme inhibitors, but also repel a variety of components. The heparan sulfate proteoglycans are involved in cell-cell and cell-matrix adhesion, modulate cellular responses to growth and differentiation factors, and are important determinants of the anticoagulant and filtrating properties of the vascular wall. The heparan sulfate proteoglycans are missing from the glomerular wall in congenital lipoid nephrosis, and accumulate in excessive amounts in the amyloid plaques of Alzheimer's disease and other neurodegenerative syndromes. A panel of monoclonal antibodies has been developed that traces three of the cell surface bound heparan sulfate proteoglycans (fibroglycan, glypican and syndecan) and the major heparan sulfate proteoglycan from basement membranes or pericellular matrices. In addition, monoclonal antibodies have been developed against epitopes in native heparan sulfate chains and against neo-epitopes produced by the enzyme heparitinase, allowing to trace and purify any heparan sulfate proteoglycan or heparitinase generated core protein preparation.

74. DAHAN K
NIAUDET P. lipoid nephrosis in childhood. Rev. Prat., 53 (18), 20272032,2003 ; (Facteur d Impact 2001 X) (Services cités Néphrologie
http://www.necker.fr/irnem/Unites 2003/03nephroped.htm
DAHAN K., DEVUYST O., SMAERS M., VERTOMMEN D., LOUTE G., POUX J.M., VIRON B., JACQUOT C., GAGNADOUX M.F., CHAUVEAU D., BUCHLER M., COCHAT P., COSYNS J.P., MOUGENOT B., RIDER M.H., ANTIGNAC C., VERELLEN-DUMOULIN C., PIRSON Y.
A cluster of mutations in the UMOD gene causes familial juvenile hyperuricemic nephropathy with abnormal expression of uromodulin.
J. Amer. Soc. Nephrol., ; (Facteur d'Impact 2001 :
(Services cités : U507, Néphrologie Pédiatrique, Néphrologie Adulte, U574
DUREAU P., BROYER M., DUFIER J.L.
Evolution of ocular manifestations in nephropathic cystinosis: A long-term study of a population treated with cysteamine.
J. Ped. Ophthalmol. Strab., ; (Facteur d'Impact 2001 : X
(Services cités : Ophtalmologie, Néphrologie Pédiatrique
FAKHOURI F., BOCQUET N., TAUPIN P., PRESNE C., GAGNADOUX M.F., LANDAIS P., LESAVRE P., CHAUVEAU D., KNEBELMANN B., BROYER M., GRUNFELD J.P., NIAUDET P.
Steroid-sensitive nephrotic syndrome: from childhood to adulthood.
Amer. J. Kidney Dis., ; (Facteur d'Impact 2001 : (Services cités : Néphrologie Pédiatrique, Néphrologie Adulte, Biostatistique

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76. Blackwell Synergy - Cookie Absent
Clin Nephrol 47 289297, 1997 MEDLINE Abstract ISI Abstract. 15. Matsumoto KDecreased release of IL-10 by monocytes from patients with lipoid nephrosis.
http://www.blackwell-synergy.com/links/doi/10.1111/j.1523-1755.2004.00392.x/enha
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77. Blackwell Synergy - Cookie Absent
3, R ICH AR A hitherto undescribed vulnerability of the juxtamedullary glomeruliin lipoid nephrosis. Bull Johns Hopkins Hosp 100173 186, 1957.
http://www.blackwell-synergy.com/links/doi/10.1046/j.1523-1755.1998.00845.x/enha
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78. International Classification Of Diseases, Icd-9, Code
581.3 With lesion of minimal change glomerulonephritis. lipoid nephrosisMinimal change glomerular disease glomerulitis nephrotic syndrome
http://www.centralx.com/diseases/icd511.htm
I NTERNATIONAL C LASSIFICATION OF D ISEASES This data is published to be used by HiDoctor'98 users
HiDoctor'98: The New Generation of Medical Software PREVIOUS NEXT MAIN PAGE HiDoctor'98 Home
581 Nephrotic syndrome
581.0 With lesion of proliferative glomerulonephritis
581.1 With lesion of membranous glomerulonephritis
Idiopathic membranous glomerular disease
581.2 With lesion of membranoproliferative glomerulonephritis
Nephrotic syndrome with lesion (of):
hypocomplementemic persistent glomerulonephritis
lobular glomerulonephritis
mesangiocapillary glomerulonephritis
mixed membranous and proliferative glomerulonephritis
581.3 With lesion of minimal change glomerulonephritis
Lipoid nephrosis
Minimal change:
glomerular disease
glomerulitis
nephrotic syndrome
581.8 With other specified pathological lesion in kidney
581.9 Unspecified
Nephrosis NOS
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79. Intra-Arterial Administration Of Agents Using ALZET Osmotic Pumps
P4004 Garin EH, Laflam P, Chandler L. Antiinterleukin 8 antibody abolishes effectsof lipoid nephrosis cytokine. Pediatric Nephrology 1998; 12381-385.
http://www.alzet.com/bibliography/bib_pages/ia.htm
Home Page References on the Intra-Arterial Administration of Agents Using ALZET Osmotic Pumps (IA-Q4-2003) 2ML1; 1 week; Controls received mp w/ vehicle; functionality of mp verified by residual volume; dose-response (p. 181, 183); cardiovascular; peptides. 2ML1; 24 hours; Cardiovascular; peptides; MCP-1 is monocyte chemoattractant protein 1. 2ML2; 3,7 days; Study proliferation of collateral arteries and capillaries. 2ML2; 18 days; Controls received mp w/ vehicle. 1007D; Controls received mp w/ vehicle; peptides. Yang HT, Yan Z, Abraham JA, Terjung RL. VEGF 2002; 14 days; Controls received mp w/ vehicle; cardiovascular; peptides; vehicle was PBS w/ 10% sodium citrate and 1.6% glycerol; sodium citrate used to prevent coagulation; glycerol used to enhance protein stability. 2002; 14 days; controls received mp w/ vehicle; dose response; peptides, cardiovascular; recomb. human FGF used; sodium citrate used to maintain catheter patency; glycerol used to stabilize protein. 2002; 2 weeks; controls received mp w/vehicle; functionality of mp verified by residual volume; peptides; human. recomb fibroblast growth factor used. 2ML4; 28 days; pump diagram (p. 888); 22 g intravenous catheter was inserted into tygon tubing; "implantable osmotic pumps offer an alternative simple delivery method, providing a constant release of soluble agents in a steady-state fashion." (p. 892); controls received mp w/vehicle; good methods (pp. 887-888); cardiovascular; peptides.

80. Nephrosis, Lipoid - Medical Dictionary Definitions Of Popular Medical Terms
MedicineNet Home MedTerms medical dictionaryAZ List nephrosis, lipoid. Advanced Search.
http://www.medterms.com/script/main/art.asp?articlekey=24101

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