81. Bayer Biologicals Jerry Winkelstein, MD, a renowned immunologist in the treatment of primary immune deficiencies, will lead the IDF Center at Johns Hopkins. http://www.bayerbiologicals.com/News_Center/Press/2001/20011105.asp

Immune Defiency Foundation Opens Center for Primary Immunodeficiencies at Johns Hopkins University Hospital "The Immune Deficiency Foundation Center for Primary Immune Deficiency will be a valuable asset to patients with primary immune deficiencies. The center will provide patients with the consultative services of renowned immunologists Dr. Jerry Winkelstein and Dr. Howard Lederman, regardless of ability to pay," said Marcia Boyle, co-founder of IDF. "We are grateful to be associated with a team that includes Bayer, whose generous gift made this center a reality, Johns Hopkins University, and Drs. Winkelstein and Lederman. I personally credit Dr. Winkelstein with saving my son's life, and providing essential medical guidance to IDF, which has helped us positively impact the lives of patients around the world." Primary immune deficiency diseases are disorders in which part of the body's immune system is missing or does not function properly. There are a wide variety of primary immune deficiencies. The World Health Organization recognizes approximately 85 primary immune deficiency diseases. Some disorders, such as selective IgA Deficiency, can be quite common, occurring as often as 1/500 to 1/1000 individuals. Others may be as rare as one individual affected per million. In addition to consultative services, the Immune Deficiency Foundation Center at Johns Hopkins will provide appropriate laboratory tests, access to therapy, and specialists, as well as providing public and professional education services.

82. Primary Immune Deficiencies - An Overview Primary immune deficiencies An Overview Primary immune deficiencies are inborn genetic defects in the immune system. It is estimated http://www.mindbranch.com/listing/product/R313-4440.html

Primary Immune Deficiencies - An Overview Published by Datamonitor Published on October 2002 Product Code: R313-4440 Ordering and More Information Price and Delivery Options Abstract Introduction: Primary immune deficiencies are inborn genetic defects in the immune system. It is estimated that at least half a million people worldwide suffer from at least one of these disorders. These patients suffer from recurrent and persistent infections. Gene therapy offers the promise of a potential cure, obliterating the need for antibiotic and immunoglobulin therapy and bone marrow transplants. Scope: Examines disease etiology, symptoms and treatment options available to patients Looks at the prevalence and diagnosis of some of the more common and defined conditions and discusses the prognosis for these individuals Assesses unmet needs in the treatment of primary immune deficiencies and future opportunities, particularly in the area of gene therapy. Report Highlights: Prompt administration of antibiotics at the first signs of infection is vital and those with deficient antibody levels require monthly immunogobulin infusions. Bone marrow transplantation has provided complete remission to some suffering from very severe conditions, such as severe combined immunodeficiency disease (SCID), but global success rates over the past thirty years are only between 6070%. Gene therapy is beginning to show some promise as a viable treatment alternative. The obvious benefit in curing these debilitating illnesses will have to be balanced with the potential risks posed by this new form of treatment. Although entry into the commercial arena is not expected for another few years, the market and growth for gene therapy products will be unprecedented, since application of this technology will have far-reaching implications for a number of other clinical conditions.

83. STEP: The Immune System -- An Overview Such an understanding may help comprehend the root of immune deficiencies, and perceive potential avenues that the immune system can be modulated in the case http://www.thebody.com/step/immune.html

The Immune System An Overview November, 1993 A Fact Sheet from the Seattle Treatment Education Project by Paul A. Linnemeyer The immune system is composed of many interdependent cell types that collectively protect the body from bacterial, parasitic, fungal, viral infections and from the growth of tumor cells. Many of these cell types have specialized functions. The cells of the immune system can engulf bacteria, kill parasites or tumor cells, or kill viral-infected cells. Often, these cells depend on the T helper subset for activation signals in the form of secretions formally known as cytokines, lymphokines, or more specifically interleukins. The purpose of this article is to review the organs, cell types and interactions between cells of the immune system as a commentary on their importance and interdependence on the T helper subset. Such an understanding may help comprehend the root of immune deficiencies, and perceive potential avenues that the immune system can be modulated in the case of specific diseases. The Organs of the Immune System Bone Marrow All the cells of the immune system are initially derived from the bone marrow. They form through a process called hematopoiesis. During hematopoiesis, bone marrow-derived stem cells differentiate into either mature cells of the immune system or into precursors of cells that migrate out of the bone marrow to continue their maturation elsewhere. The bone marrow produces B cells, natural killer cells, granulocytes and immature thymocytes, in addition to red blood cells and platelets.

84. Allergy, Asthma, And Immunology immune deficiencies. immune deficiencies. There are many different immune system deficiencies that require clinical care by a physician http://www.rush.edu/rumc/page-P01698.html

Allergy and Immunology Immune Deficiencies Immune Deficiencies There are many different immune system deficiencies that require clinical care by a physician or other healthcare professional. Listed in the directory below are some, for which we have provided a brief overview. If you cannot find the condition in which you are interested, please visit the Allergy, Asthma, and Immunology Online Resources page in this Web site for an Internet/World Wide Web address that may contain additional information on that topic. Immune Disorders Allergy, Asthma, and Immunology Home Page Immune Deficiencies Common Variable Immunodeficiency (CVID) DiGeorge Syndrome Severe Combined Immunodeficiency (SCID) X-linked Agammaglobulinemia ... Site Map

85. Modells Fund Research On Immune Diseases Children with immune deficiencies have a high risk of contracting pneumocystis pneumonia, which is lifethreatening only to people with impaired resistance to http://www.news.harvard.edu/gazette/1997/10.16/ModellsFundRese.html

[an error occurred while processing this directive] October 16, 1997 SEARCH THE GAZETTE Modells Fund Research on Immune Diseases By William J. Cromie Gazette Staff Confused with AIDS Replacing What's Missing pneumonia. They bind molecules from these organisms with so-called B cells, which then produce immunoglobulins to neutralize the invaders.

86. Allergy, Asthma, And Immunology - Immune Deficiencies immune deficiencies. There are many different immune system deficiencies that require clinical care by a physician or other healthcare professional. http://www.musckids.com/health_library/allergy/oidhub.htm

Home About Us Departments Send an e-Card ... Your Hospital Visit 171 Ashley Ave. Charleston, SC 29425 800-424-MUSC Print Version Allergy, Asthma, and Immunology Immune Deficiencies There are many different immune system deficiencies that require clinical care by a physician or other healthcare professional. Listed in the directory below are some, for which we have provided a brief overview. If you cannot find the condition in which you are interested, please visit the Allergy, Asthma, and Immunology Online Resources page in this Web site for an Internet/World Wide Web address that may contain additional information on that topic. Severe Combined Immunodeficiency (SCID) Common Variable Immunodeficiency (CVID) DiGeorge Syndrome X-linked Agammaglobulinemia ... Contact Us

87. PCOS Messages For January, 2002: Re: To Barbra Re: Lupus, Immune Deficiencies Et Re To Barbra re lupus, immune deficiencies etc. (Was re test results) ; Next in thread Panacea Re To Barbra re lupus, immune deficiencies etc. http://forums.obgyn.net/pcos/PCOS.0201/0191.html

-VISIT OUR OTHER FORUMS- Breast Health Forum Endo@OBGYN.net Mujer (en español) PCOS Forum PCOS Diet Forums PCOS Medication Forum Vrouw en Gezondheid (nederland) Women's Health Forum Young Women's Health Forum s earch this forum: Re: To Barbra re: lupus, immune deficiencies etc. (Was re: test results) From: Belle anonymous@medispecialty.com Mon, 7 Jan 2002 22:54:18 -0600 (CST) Messages sorted by: [ date ] [ thread ] [ subject ] [ author ] Next message: Panacea: "Re: In regards to Barbara" Previous message: Dona: "Re: In regards to Barbara" In reply to: Sonnet: "To Barbra re: lupus, immune deficiencies etc. (Was re: test results)" Next in thread: Panacea: "Re: To Barbra re: lupus, immune deficiencies etc. (Was re: test results)" Reply: Panacea: "Re: To Barbra re: lupus, immune deficiencies etc. (Was re: test results)" Reply: Barbra Camara: "Re: To Barbra re: lupus, immune deficiencies etc. (Was re: test results)" Because of the connection with insulin and the genetic links, many endocrinologists do consider PCOS to be auto-immune even though it has not officially been classified as such yet. As we know, if you get one auto-immune disorder, you are more likely to get another. Mononucleosis has been linked with auto-immune disorders in itself. Just last week (Thursday or Friday?) research was completed and a recommendation was sent out alerting doctors that if a client has a history of mono, they are more likely to have MS. It has been widely known that if you had mono, you are more likely to have an auto-immune disorder.

88. SafeScience, Inc. Researchs and develops products to improve plant health, enhance antibody production, and treat human cancer, hiv, and diseases related immune system deficiencies. (Nasdaq SAFS). http://www.safesci.com/

89. Interactive Fly, Drosophila The newly generated deficiencies were analyzed for complementation of the imd 1 mutation by monitoring the immune inducibility of the gene encoding the http://sdb.bio.purdue.edu/fly/dbzhnsky/imd1.htm

immune deficiency Biological Overview Evolutionary Homologs Regulation Developmental Biology ... References Gene name - immune deficiency Synonyms Cytological map position - Function - signal transduction Keywords - immune response Symbol - imd FlyBase ID: Genetic map position - Classification - death domain protein Cellular location - cytoplasmic NCBI and GadFly links: Precomputed BLAST LocusLink UniGene GadFly Fly GRID Imd genetic and physical interactions BIOLOGICAL OVERVIEW In the mid 1990s, it became apparent that two distinct pathways control the antifungal and antibacterial responses (Lemaitre, 1995 and 1996). Indeed, it was found that the dorsoventral regulatory gene cassette Toll cactus directs the potent antifungal response in Drosophila adults, whereas the antibacterial defense is largely independent of these genes (Lemaitre, 1996). A mutation was discovered at that time, and referred to as imd immune deficiency ) in which the antibacterial, but not the antifungal response, was compromised (Lemaitre, 1995). Since the initial description of the imd mutation, four additional genes have been shown to participate in the antibacterial defense of Drosophila: (1)

90. Secondary Deficiencies Acquired immune Deficiency Syndrome. Protein and/or calorie malnutrition affects production of leukocytes which eventually induces immune deficiency. http://imcip.gsm.com/integrated/immu/im/immuno/ch12/secndary.htm

Secondary Deficiencies Acquired Immune Deficiency Syndrome Acquired immune deficiency syndrome (AIDS) results from destruction of the Th cells by infection by the human immunodeficiency virus (HIV-1). The binding site for HIV-1 is the CD4 molecule that is found preferentially on Th cells. Since both cellular and humoral responses require the participation of Th cells, both types of immunity are deficient (above figure). Patients present with multiple bacterial, viral, fungal, and parasitic infections including P. carinii . They are also subject to a greatly increased incidence of a particular skin cancer, Kaposi's sarcoma. Drugs such as AZT that limit viral replication have had some success in slowing down the progression of the disease and combination therapy with some of the new protease inhibitors show some real promise. However, replacement of the Th cells would not have any long term benefit unless one first eliminates the virus. Otherwise, the new cells are simply infected and destroyed similar to the original infection. Other secondary immunodeficiencies Temporary immunosuppression results from several diseases including cancer, measles, influenza, diabetes, tuberculosis, and leprosy. Treatment of the underlying condition usually restores immune function. However, during the time of immunosuppression, the patients are at increased risk of opportunistic and viral infections. Some people have theorized that inopportune immunosuppression from viral diseases could be the factor that allows cancer growth to get ahead of

91. Hiv (Immune Deficiency) Treatment - Print Version : Online Reference For Health of zinc, HIV patients should be monitored for zinc deficiencies and supplemented zinc levels are also associated with severe decline in immune status (Rousseau http://www.lef.org/protocols/prtcls-txt/t-prtcl-059.html

HIV and AIDS A BRIEF HISTORY OF HIV/AIDS The United Nations named HIV and acquired immune deficiency syndrome (AIDS) a major threat to developing countries. In this time period, the disease orphaned 13.2 million children, infected 60 million people worldwide, and killed a worldwide total of almost 22 million people, rivaling the 25 million death count from the Black Death (bubonic plague) of the 14th century (Gottlieb 2001; Sepkowitz 2001; Steinbrook 2001). In the last 20 years, we have seen waves of nearly mass hysteria about HIV, long periods of dashed hopes regarding treatment, resurgence of optimism with the development of the newer treatments of highly active antiretroviral therapy (HAART), frustrated efforts at vaccine development, and global rates of infection at crisis proportions, especially involving transmission from mothers to children. AIDS activists made definite helpful reforms regarding the availability of newer drugs and the drug approval process. In 1986, it took an average of 34.1 months to push a drug through the FDA approval process. In 1999, this time frame decreased to an average of 12.6 months. Rather than following the established rules of lobbying, other novel approaches were used, such as drug buyers' clubs, civil disobedience, and telephone "zaps" during which the telephone switchboard of a targeted company was incapacitated by a barrage of incoming coordinated calls. Because of AIDS activism, patients are now routinely consulted in drug study design. Community-based research efforts are conducted all across the country, and new drugs are made available more quickly.

92. IMMUNE DEFICIENCY FOUNDATION Celebrating Primary immune Deficiency Awareness Week April 1824, QUICK NEWS Get the latest news right here. IGIV Recall Gammar ® -PIV April 1, 2004. NEW! http://www.primaryimmune.org/

QUICK NEWS Get the latest news right here IGIV Recall : Gammar -PIV April 1, 2004 NEW! 2004 IDF Regional Retreats Information Now Available! Research Grant Opportunity: Call for Concept Research Proposals on Primary Immune Deficiency Diseases (PIDD) Announcement , Deadline: November 1, 2004 USIDNET 3) To submit a Concept Proposal using proposalCENTRAL, click here New! IDF Brochure April 23, 2004 Blue Jeans for Healthy Genes Help support IDF while implementing a casual dress day in your workplace. The Clinical Presentation of Primary Immune Deficiency Diseases Continuing Medical Education (CME) course Call for Submissions for Fellowship Opportunities: Deadline November 1, 2004 2004 Fellowship Winner: Sean McGhee, M.D. INDUSTRY NEWS OCTAGAM Receives FDA Approval for PID May 24, 2004 CSL Limited, the parent company of ZLB Bioplasma, acquires Aventis Behring April 1, 2004 OUR LATEST NEWSLETTER Winter 2004 OUR SPONSORS Funding for this site provided by Manufacturers of IDF Core Services supported by Many files on this site are Adobe Acrobat PDFs. You will need the Adobe

93. Immune Deficiency immune deficience resources, national and international support groups, clinics with genetic counselors and geneticists immune Deficiency Conditions. PID (Primary immune Deficiency) and SCID (Severe immune Deficiency Foundation. Courthouse Square, 4565 Ellicott Mills http://www.kumc.edu/gec/support/immune.html

Immune Deficiency Conditions PID (Primary Immune Deficiency) and SCID ( Severe Combined Immunodeficiency) Immune Deficiency Foundation Courthouse Square, 4565 Ellicott Mills Dr., Unit B2, Ellicott City, MD 21043 Phone: 800.296.4433, Fax: 410.461.5292 Patient / Family Handbook for the Primary Immune Deficiency Diseases Our Immune System The Jeffrey Modell Foundation 747 Third Avenue, 34th Floor, New York, New York 10017 National Hotline:1.800.JEFF.844 E-mail: info@jmfworld.org Web site: www.jmfworld.com/ International Patient Organization for Primary Immunodeficiencies Patient /Family Handbook German - Spanish - English - Swedish - French - Italian Clinical Presentation of the Primary Immunodeficiency Diseases Clinical Updates , USA International Groups Australia, Canada, Chile, Cyprus, Denmark, Estonia, Finland, France, Germany, Greece, Hungary, India, Ireland, Italy, Netherlands, New Zealand, Norway, Poland, Russia, South Africa, Spain, Sweden, Switzerland, United Kingdom, Yugoslavia, other) Primary Immunodeficiency Association (PIA), United Kingdom

94. Primary Immune Deficiency, NIAID Fact Sheet March 2003. Primary immune Deficiency. Contents. What is Primary immune Deficiency? Selective WHAT IS PRIMARY immune DEFICIENCY? When http://www.niaid.nih.gov/factsheets/pid.htm

March 2003 Primary Immune Deficiency Contents What is Primary Immune Deficiency? Selective IgA Deficiency Common Variable Immunodeficiency (CVI) X-Linked Agammaglobulinemia (XLA) ... More Information WHAT IS PRIMARY IMMUNE DEFICIENCY? When people are born with a faulty immune system, they are said to have a primary immune deficiency or immunodeficiency. Unlike people with AIDS, caused by the human immunodeficiency virus or HIV, people with primary immunodeficiency (PI) diseases have inherited abnormal changes in the cells of their immune systems . Between 25,000 and 50,000 people suffer from the most serious forms of PI diseases in the United States, but experts believe that many more have milder disease that is not yet diagnosed. Each type of immune system cell has its own special function and must work together with other types to fight disease effectively. Because there are many different types of cells that make up the immune system, an error in any one of them can disrupt our immune defenses. Depending on the cell and the type of error that occurs, more than 80 different forms of PI diseases are possible. Some are severe, while others cause few or no symptoms. Having any of them makes it easier to get infections and other medical conditions. More boys than girls have PI, and first symptoms often begin in infancy or later in childhood.

95. Cd5 - Cd19 PID Immunology at Cleveland s Rainbow Babies and Children s Hospital, made a remarkable break through in the discovery of an astonishing Primary immune Deficiency. http://www.scid.net/cd5cd19pid/

What is the Cd5 - Cd19 PID? In 1997, Dr. Robert W. Hostoffer DO, pediatric immunologist from Cleveland, Ohio, made a remarkable break through in the discovery of an astonishing Primary Immune Deficiency . The syndrome, which was given the name Cd5 - Cd19 PID , involves a complete and total lack of response to polysaccharide antibodies and an extremely high expression of Cd5+ B-cells. It has been documented that the lack of antibody response is due to an insufficient number of Cd5- cells in these patients. At this time, it is also suspected that the defect is T-cell related. Presently, there are only 7 boys in the world diagnosed with this very rare syndrome. Although research continues, to date, no females or adults have been identified with this immune deficiency. The following is from a case study written by Peter M. Antall, MD, Howard Meyerson, MD, David Kaplan, MD, John Venglarcik, MD, and Robert W. Hostoffer, DO Cleveland and Youngstown, Ohio, and published in The Journal of Allergy and Clinical Immunology, April 1999, pages 637-641, Volume 103, Number 4 "Primary humoral deficiencies vary from complete absence of B cells and/or serum immunoglobulin to lacunar deficits involving specific antibody responses to polysaccharides." Only a short abstract of this article is available online. It is recommended that anyone researching this subject obtain the complete article.

96. Update On Acquired Immune Deficiency Syndrome (AIDS) Among Patients December 10, 1982 / 31(48);6446,652 Update on Acquired immune Deficiency Syndrome (AIDS) among Patients with Hemophilia A. In July http://www.cdc.gov/epo/mmwr/preview/mmwrhtml/00001202.htm

December 10, 1982 / 31(48);644-6,652 Update on Acquired Immune Deficiency Syndrome (AIDS) among Patients with Hemophilia A Case 1: A 55-year-old severe hemophiliac from Alabama developed anorexia and progressive weight loss beginning in September 1981. He had developed adult-onset diabetes mellitus in 1973, which had required insulin therapy since 1978. He had had acute hepatitis (type unknown) in 1975. In March 1982, he was hospitalized for herpes zoster and a 17-kg weight loss. Hepatosplenomegaly was noted. The absolute lymphocyte count was 450/mm((3)). Liver enzymes were elevated; antibodies to hepatitis B core and surface antigens were present. A liver biopsy showed changes consistent with persistent hepatitis. Evaluation for an occult malignancy was negative. The zoster resolved following 5 days of adenosine arabinoside therapy. In early June, he was readmitted with fever and respiratory symptoms. Chest x-ray showed bibasilar infiltrates. No causative organism was identified, but clinical improvement occurred coincident with administration of broad spectrum antibiotics. Laboratory studies as an outpatient documented transient thrombocytopenia (63,000/mm((3))) and persistent inversion of his T-helper/T-suppressor ratio (TH/TS =0.2). He was readmitted for the third time in early September with fever, chills and nonproductive cough. His cumulative weight loss was now 47 kg. Chest x-ray demonstrated bilateral pneumonia, and open lung biopsy showed infection with P. carinii. He responded to sulfamethoxazole/trimethoprim (SMZ/TMP). His T-cell defects persist.

97. Chronic Fatigue And Immune Deficiency Syndrome - Taking A New Perspective On CFI Chronic Fatigue and immune Deficiency Syndrome ( CFIDS ) or Chronic Fatigue Syndrome ( CFS ) Taking a New Perspective on CFIDS - Dr. Larry Sharp. http://www.cfidsdoctor.com/CFIDSfaqs.asp

1002 Montgomery Street, Fort Worth, Texas 76107 - Tel. 817-732-2878 Frequently Asked Questions regarding CFIDS What is Chronic Fatigue and Immune Dysfunction Syndrome? Can it be treated? Is CFIDS the same thing as Fibromyalgia? Here are just a few of the most commonly asked questions regarding CFIDS. Home About the Doctor Office Information CFIDS F.A.Q.s ... More about CFIDS What is Chronic Fatigue and Immune Dysfunction Syndrome? Chronic Fatigue and Immune Dysfunction Syndrome, CFIDS or CFS, is a serious and complex illness. Chronic Fatigue Syndrome is associated with systemic and cognitive symptoms and with several immune abnormalities. It affects numerous body systems such as the central nervous system, endocrine, immune and gastrointestinal to name a few. According to the CFS case definition published in the Dec. 15, 1994 issue of the Annals of Internal Medicine , diagnosing CFIDS requires a thorough medical history, physical and mental status examinations and laboratory tests to identify underlying or contributing conditions that require treatment. Chronic fatigue can be classified as Chronic Fatigue and Immune Dysfunction Syndrome if the patient meets both the following criteria: Clinically evaluated, unexplained persistent or relapsing chronic fatigue that is of new or definite onset (i.e., not lifelong) is not the result of ongoing exertion, is not substantially alleviated by rest, and results in substantial reduction in previous levels of occupational, educational, social or personal activities.

98. AEGiS-MMWR: Update On Acquired Immune Deficiency Syndrome (AIDS) Among Patients Click here to return to CDC MMWR main menu Update on Acquired immune Deficiency Syndrome (AIDS) among Patients with Hemophilia A http://www.aegis.com/pubs/mmwr/1982/MM3148.html

Important note: Information in this article was accurate in December 1982. The state of the art may have changed since the publication date. Update on Acquired Immune Deficiency Syndrome (AIDS) among Patients with Hemophilia A MMWR Weekly, December 10, 1982 / 31(48);644-6,652 Centers for Disease Control and Prevention Case 1: A 55-year-old severe hemophiliac from Alabama developed anorexia and progressive weight loss beginning in September 1981. He had developed adult-onset diabetes mellitus in 1973, which had required insulin therapy since 1978. He had had acute hepatitis (type unknown) in 1975. In March 1982, he was hospitalized for herpes zoster and a 17-kg weight loss. Hepatosplenomegaly was noted. The absolute lymphocyte count was 450/mm((3)). Liver enzymes were elevated; antibodies to hepatitis B core and surface antigens were present. A liver biopsy showed changes consistent with persistent hepatitis. Evaluation for an occult malignancy was negative. The zoster resolved following 5 days of adenosine arabinoside therapy. In early June, he was readmitted with fever and respiratory symptoms. Chest x-ray showed bibasilar infiltrates. No causative organism was identified, but clinical improvement occurred coincident with administration of broad spectrum antibiotics. Laboratory studies as an outpatient documented transient thrombocytopenia (63,000/mm((3))) and persistent inversion of his T-helper/T-suppressor ratio (TH/TS =0.2). He was readmitted for the third time in early September with fever, chills and nonproductive cough. His cumulative weight loss was now 47 kg. Chest x-ray demonstrated bilateral pneumonia, and open lung biopsy showed infection with P. carinii. He responded to sulfamethoxazole/trimethoprim (SMZ/TMP). His T-cell defects persist.

99. MedWebPlus Web Site ID 22988 http://medwebplus.com/obj/22988

Main About MWP Contribute to MWP Contact Us A service of Flexis, Inc. Welcome to MedWebPlus 2.3! A free service to help you find health sciences information quickly and easily. adam.com Diseases and Conditions Immune Deficiencies http://www.adam.com/dir/Diseases_and_Conditions/Immune_System/Immune_Deficiencies/Immune_Deficiencies.htm Location: North America United States Georgia Atlanta Subjects: All Diseases Immunologic Diseases Sexually Transmitted Diseases Diseases and Conditions ... Privacy statement Powered by y-Base

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