81. Diabetes Mellitus, Hyperinsulinemia, And Coronary Artery Disease - Humana Press Mail humana@humanapr.com, Diabetes Mellitus, hyperinsulinemia, andCoronary Artery Disease. Preventive Cardiology Strategies for http://www.humanapress.com/ChapterDetail.pasp?isbn=1-59259-001-2&ccode=1-59259-0

82. [Control Of Hyperinsulinemia In Essential Hypertension Using The Angiotensin-con 1993;35(2)759. Control of hyperinsulinemia in essential hypertension usingthe angiotensin-converting enzyme inhibitor, lisinopril Article in Czech http://darwinpharmacy.com/zestril-online-rx/zestril-online-rx5.1.html

Zestril online research references Cor Vasa. 1993;35(2):75-9. [Control of hyperinsulinemia in essential hypertension using the angiotensin-converting enzyme inhibitor, lisinopril] [Article in Czech] Umnerova V, Jarolim M, Jindra A, Jachymova M, Peleska J, Savlikova J, Stolba P, Horky K. II. interni klinika a kardiologicka laborator 1. lekarske fakulty Univerzity Karlovy, Praha, Ceska republika. word match zestril online literature Am J Hypertens. 1993 Apr;6(4):148S-154S. Comparative renal hemodynamic effects of lisinopril, verapamil, and amlodipine in patients with chronic renal failure. August P, Lenz T, Laragh JH. Cardiovascular Center, New York Hospital-Cornell Medical Center, New York 10021. word match zestril online literature Circ Res. 1993 Jul;73(1):51-60. Measurement of angiotensin I converting enzyme inhibition in the heart. Kinoshita A, Urata H, Bumpus FM, Husain A. Department of Cardiovascular Biology, Research Institute, OH 44195-5071. word match zestril online literature Arq Bras Cardiol. 1999 Oct;73(4):359-72. Effects of lisinopril on experimental ischemia in rats. Influence of infarct size.

83. Glycemic Index, Hyperinsulinemia, And Breast Cancer Risk Glycemic index, hyperinsulinemia, and breast cancer risk. Key TJ Glycemicindex, hyperinsulinemia, and breast cancer risk Ann Oncol; 12 15071509. http://sci.cancerresearchuk.org/research/ceu/publications/abstracts/2001-9007735

Cancer Research UK Epidemiology Unit Front Page About Us Research Staff ... Contact Details Glycemic index, hyperinsulinemia, and breast cancer risk Key TJ Glycemic index, hyperinsulinemia, and breast cancer risk Ann Oncol Cancer Research UK Epidemiology Unit Gibson Building, Radcliffe Infirmary, Oxford. OX2 6HE Phone +44 (0) 1865 311933 Fax +44 (0) 1865 310545

84. Atkins Nutritionals UK: The Science Behind Atkins: Research Summaries: All Resea The Science Behind Atkins Research Summaries All ResearchSummaries hyperinsulinemia in Hypertension. http://atkins-uk.com/Archive/2002/8/9-976917.html

The Science Behind Atkins Research Summaries All Research Summaries > Hyperinsulinemia in Hypertension Hyperinsulinemia in Hypertension: Associations with Race, Abdominal Obesity, and Hyperlipidemia Reference: Spangler, J.G., Bell, R.A., Summerson, J.H., et al., "Hyperinsulinemia in Hypertension: Associations With Race, Abdominal Obesity, and Hyperlipidemia," Archives of Family Medicine , 7(1), 1998, pages 53-56. Summary: The following information is available at Pub Med and was not written by Atkins professionals. OBJECTIVE: To determine the relative contributions of race, sex, abdominal obesity, and hyperlipidemia to the development of hyperinsulinemia among patients with hypertension. DESIGN: Cross-sectional survey. SETTING: A large family practice ambulatory care unit in Winston-Salem, NC. PATIENTS: MAIN OUTCOME MEASURES: Fasting insulin, lipid, and glucose levels; glycosylated hemoglobin; waist-hip ratio; and resting blood pressure. METHODS: Among 4 patient subgroups (hypertension alone; hypertension and abdominal obesity; hypertension and hyperlipidemia; and hypertension, abdominal obesity, and hyperlipidemia) logistic regression analysis was used to determine correlates of elevated fasting insulin levels. RESULTS: Controlling for age and blood pressure, black males had the highest fasting insulin levels (135 +/- 70 pmol/L [18.8 +/- 9.6 microU/mL] and 265 pmol/L [37.0 +/- 0.0 microU/mL] [mean +/- SD] for obese and nonobese black males, respectively); nonobese white males had the lowest fasting insulin levels (23 +/- 22 pmol/L [3.2 +/- 3.0 microU/mL]). Multivariate logistic regression indicated that the addition of abdominal obesity or hyperlipidemia to pure hypertension more than doubled the risk of hyperinsulinemia (adjusted odds ratio, 2.69; 95% confidence interval, 1.04-6.89; and adjusted odds ratio, 2.62; 95% confidence interval, 0.37-8.6, respectively). The combination of abdominal obesity and hyperlipidemia exerted additive effects among patients with hypertension for elevated insulin levels (adjusted odds ratio, 5.1; 95% CI, 1.59-16.4).

85. PLASMA TOTAL HOMOCYSTEINE LEVELS IN SUBJECTS WITH HYPERINSULINEMIA TEKTRAN. PLASMA TOTAL HOMOCYSTEINE LEVELS IN SUBJECTS WITH hyperinsulinemia.Author(s) BARON H KIDRON M FRIELANDER Y BEN-YEHUDA http://www.nal.usda.gov/ttic/tektran/data/000012/59/0000125941.html

TEKTRAN PLASMA TOTAL HOMOCYSTEINE LEVELS IN SUBJECTS WITH HYPERINSULINEMIA Author(s): BAR-ON H KIDRON M FRIELANDER Y BEN-YEHUDA A SELHUB JACOB ROSENBERG IRWIN H FRIEDMAN G Interpretive Summary: Homocysteine is an amino acid which has important function in the body. How ever, it becomes harmful and results in an increased risk of heart disease when blood levels are elevated. We found that people with high blood levels of insulin, which is a sign of diabetes, also have low homocysteine levels. Although homocysteine is lower than in people with normal insulin levels, high homocysteine levels continue to be a risk for heart disease in these patients. Keywords: folate vitamin b12 b6 pyridoxal-5-phoshate homocysteine homocysteinemia sulphur amino acid b affinity hplc bioavailability dysplasia arteriosclerosis Contact: JEAN MAYER HUMAN NUTR. RE 711 WASHINGTON ST., TUFTS BOSTON MA 02111 FAX: Email: jselhub@hnrc.tufts.edu Approved Date: TEKTRAN United States Department of Agriculture Agricultural Research Service Updated:

86. MIR Teaching File Case Pt018 Diagnosis Effect of hyperinsulinemia on FDG Distribution. Brief history DiagnosisEffect of hyperinsulinemia on FDG Distribution. Full history http://gamma.wustl.edu/pt018te179.html

87. Hyperinsulinemia hyperinsulinemia. This response submitted by on 2/8/99. Answer first, then a question.hyperinsulinemia is too much insulin floating around in your bloodstream. http://neuro-www.mgh.harvard.edu/forum/GeneralNeurologyF/Hyperinsulinemia.html

Hyperinsulinemia This response submitted by on 2/8/99. Email Address: Hi Chris, You've sure been on a bad trip. I'm sorry. Answer first, then a question. Hyperinsulinemia is too much insulin floating around in your bloodstream. It can happen to diabetics who inject insulin or take pills to produce more insulin, then for a number of reasons don't have enough food converted to use the insulin. In a normal, non-diabetic person, eating food signals the pancreas to put out enough insulin to use the food. In a person with hyperinsulinemia, the pancreas overreacts to the signal and puts out too much. It's worst when the person eats carbohydrates, which are very quickly absorbed. The blood sugar (everything is converted to blood sugar) goes up, the pancreas puts out too much to take care of it, and when the blood sugar is processed, the insulin keeps working. Result: Low blood sugar. If you eat more carbs, it will take care of the problem temporarily, then cause a bigger problem, because the pancreas will have put out too much again. My question is whether the blood sugar tests you've had included a five-hour (even two-hour) glucose tolerance test. Otherwise, the blood sugar reading is only for that moment.

88. Hyperinsulinemia Is Common In Family Members Of Women With Polycystic Ovary Synd Fertil Steril. 1996 Dec;66(6)9427. hyperinsulinemia is common infamily members of women with polycystic ovary syndrome. Norman http://hairmillion.com/ref-baldness/baldness-research-abs3.424.html

References: Hair growth and hair loss Fertil Steril. 1996 Dec;66(6):942-7. Hyperinsulinemia is common in family members of women with polycystic ovary syndrome. Norman RJ, Masters S, Hague W. Reproductive Medicine Unit, University of Adelaide, Queen Elizabeth Hospital, Woodville, Australia. OBJECTIVE: To determine whether disorders of insulin secretion are common in male and female family members of subjects with polycystic ovary syndrome (PCOS). DESIGN: Family study of siblings and parents of PCOS subjects (five families). All proband cases met the criteria of polycystic ovaries (PCO) by ultrasound (US) and hyperandrogenism. SETTING: University Reproductive Medicine Unit. PATIENT(S): Family members of PCOS subjects. INTERVENTION(S): Oral glucose tolerance testing (OGTT), insulin, and lipids were measured. Clinical examination including assessment of premature baldness in men and US of ovaries in female members. MAIN OUTCOME MEASURE(S): Insulin, lipids, and clinical parameters. RESULT(S): Hyperinsulinemia (69%) and hypertriglyceridemia (56%) was common in family members as were PCO in 79% of 24 females and premature baldness in men in 88% of eight subjects. CONCLUSION(S): Hyperinsulinemia is a potential metabolic and genetic marker for subjects who may be carriers of a familial tendency for PCO.

89. Psychiatric Diagnoses In Children With Fertil Steril. 1996 Dec;66(6)9427. hyperinsulinemia is common infamily members of women with polycystic ovary syndrome. Norman RJ http://hairmillion.com/ref-alopecia/alopecia-research-abs3.926.html

alopecia areata. Hyperinsulinemia is common in family members of women with polycystic ovary syndrome. Evidence for genetic heterogeneity in monilethrix. References: Hair growth and hair loss J Am Acad Child Adolesc Psychiatry. 1996 Nov;35(11):1518-22. Psychiatric diagnoses in children with alopecia areata. Reeve EA, Savage TA, Bernstein GA. St. Paul-Ramsey Medical Center, St. Paul, MN 55101, USA. OBJECTIVE: To identify psychiatric diagnoses in a sample of 12 children with alopecia Fertil Steril. 1996 Dec;66(6):942-7. Hyperinsulinemia is common in family members of women with polycystic ovary syndrome. Norman RJ, Masters S, Hague W. Reproductive Medicine Unit, University of Adelaide, Queen Elizabeth Hospital, Woodville, Australia. J Invest Dermatol. 1996 Dec;107(6):812-4. Evidence for genetic heterogeneity in monilethrix. Richard G, Itin P, Lin JP, Bon A, Bale SJ. Laboratory of Skin Biology, NIAMS, NIH, Bethesda, MD 20892-2757, USA. Monilethrix is a rare inherited defect of the hair shaft resulting in hair fragility and dystrophic alopecia . In contrast to recent reports mapping monilethrix to the type II epithelial and trichocyte keratin gene cluster on 12q13, we strongly excluded these candidate genes in another family with autosomal dominant monilethrix. Moreover, there was no evidence for linkage of the disease to the keratin gene cluster on chromosome 17q12-q21, thus excluding defects in all known trichocyte and epithelial keratins as the cause of monilethrix in this family. Likewise, several other genes known to play an important role in hair shaft formation (trichohyalin and involucrin, ultra-high sulfur matrix proteins, and transglutaminases 1, 2, and 3) did not provide any evidence for linkage. Our results indicate genetic heterogeneity in monilethrix and suggest that aberrations in at least one other gene result in a similar phenotype.

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