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         Glomerular Diseases:     more books (33)
  1. The pathogenesis of glomerular nephritis, (Mellon lecture) by Warfield Theobald Longcope, 1929
  2. Harrison's Nephrology and Acid-Base Disorders by J. Jameson, Joseph Loscalzo, 2010-05-03
  3. Pediatric Nephrology
  4. When to discontinue ACE inhibitors for nephropathy by MD George L. Bakris, 2010-06-22

41. Healthfinder® — Glomerular Diseases
Overview of kidney function and glomerular diseases and their causes, symptoms, and diagnosis. glomerular diseases. internet URL.
http://www.healthfinder.gov/Scripts/ShowDocDetail.asp?doc=6507&lang=1

42. CIN'2003. Wedeen. Occupational Renal Diseases
The contribution of environmental toxin exposure to the development of lupus erythematosus and glomerular diseases remains to be determined.
http://www.uninet.edu/cin2003/conf/wedeen/wedeen.html

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Occupational Renal Diseases
Richard P. Wedeen, M.D.
Veterans Health Administration New Jersey Health Care System
East Orange, New Jersey. USA wedeen@umdnj.edu
Occupational renal diseases are important because they are entirely preventable. In addition, renal disease produced by heavy exposure to industrial toxins by relatively few workers provides a clinical basis for understanding the consequences of low-dose exposure to the general population from environmental pollution. The known toxins that produce kidney disease in the workplace are limited to the heavy metals plus silica and certain organic compounds, particularly halogenated hydrocarbons, employed as industrial solvents. The absence of information on the adverse renal effects of other industrial pollutants does not prove that no such effects occur, but rather that epidemiologic studies are lacking. It is probable that as the genetic basis of individual susceptibility unfolds, the contribution of toxins to renal disease in the general population will become clearer. The renal diseases due to heavy metals are listed in the Table. Lead, cadmium, mercury arsenic, chromium, and uranium are widely recognized as nephrotoxins in humans. Given appropriate chemical forms, dosage, and route of administration, acute tubular necrosis (ATN) is consistently produced by these metals. ATN occurs because of the selective accumulation of metals in the proximal tubule. When the appropriate oxidative form is present in sufficient quantity, the metals induce irreversible cellular injury. Following intense occupational exposure, and in animal experiments, the cause of ATN is obvious. When recovery is incomplete, chronic tubular-interstitial nephritis (TIN) may persist as a residual. On the other hand, the role of heavy metals in the induction of immunologically mediated glomerular disease is by no means obvious.

43. Glomerular Diseases
Glomerular. Diseases. Educational Computing Curriculum General Pathology General Pathology Courseware Hippocrates Project
http://endeavor.med.nyu.edu/courses/systemicpathology/courseware/renalpath/r1/gl
Glomerular
Diseases
Educational Computing Curriculum General Pathology General Pathology Courseware Hippocrates Project, New York University School of Medicine

44. Glomerular Diseases II
glomerular diseases II. Click here to start. Table of Contents. glomerular diseases II. Renal Disease associated with Diabetes Mellitus (p569).
http://pathophysiology.uams.edu/Spring03/Renal/Renal4/
Glomerular Diseases II
Click here to start
Table of Contents Glomerular Diseases II Renal Disease associated with Diabetes Mellitus (p569) Diabetic Nephropathy Diabetic Nephropathy ... Home

45. Glomerular Diseases: Part A & B
II. glomerular diseases A. Etiologic types 1. Immunologic glomerular diseases. 2. Metabolic diseases (Diabetic Mellitus, Amyloidosis).
http://www.umdnj.edu/pathnweb/syspath/lec_1/gd_ab/gd_ab.htm
Home G.D.: C II. Glomerular Diseases: A. Etiologic types:
    1. Immunologic glomerular diseases 2. Metabolic diseases (Diabetic Mellitus, Amyloidosis) 3. Coagulopathic disorders (DIC, Hemolytic-uremic syndrome, and Thrombotic-thrombocytopenic Purpura) (See General Path Syllabus : Thrombosis Lecture 4. Hypertension (benign and malignant) 5. Congenital/Hereditary Diseases 6. Idiopathic
B. Pathogenic mechanisms of immunologic glomerular diseases:
    1.Immune-complex mediated: characterized by granular capillary wall and mesangial immunofluorescent pattern of immune reactants (Ig, complement) and electron dense deposits in EM. (Fig: 3A, 3B) Fig 3A Fig 3b
      a.Deposition of circulating immune complexes: (Fig: 4A, 4B, 4C) Fig 4A Fig 4B Fig 4C
        i.Antigen exogenous (e.g., infections, drugs, foreign proteins)
      ii.Antigen endogenous (e.g., DNA, Ig, tumor antigens)
        b.In-situ immune complex formation: (Fig: 5A) Fig 5A
          i.Visceral epithelial cell GP330 antigen (Exp. Model: Heyman nephritis) (Fig 5B) Fig 5B
            ii.Planted (trapped) antigens: (Fig:5C)

46. Glomerular Diseases: Part D,E & F
injury. 3. End stage disease. E. Classification of glomerular diseases 1. Primary glomerular diseases, eg, Postinfectious, Drugs, etc.
http://www.umdnj.edu/pathnweb/syspath/lec_1/gd_def/gd_def.htm
Home G.D.: C D. Pattern of injury:
    1. Acute and Chronic 2. Diffuse, Focal, Segmental and Global glomerular injury 3. End stage disease
E. Classification of glomerular diseases:
    1. Primary glomerular diseases, e.g., Postinfectious, Drugs, etc. 2. Glomerular diseases associated with systemic diseases, e.g. SLE, Systemic Vasculitis, Coagulopathic Disorders, Neoplasia, Amyloidosis, and Diabetes Mellitus 3. Hereditary
F. Clinical Syndromes:

47. DVM - Glomerulonephritis Remains An Important Cause Of Renal
It can be very difficult to differentiate nonimmunologically from immunologically mediated glomerular diseases with the limited diagnostic techniques that are
http://www.dvmnewsmagazine.com/dvm/article/articleDetail.jsp?id=88239

48. Glomerular Diseases - National Institutes Of Health (NIH)
glomerular diseases Health Information from National Institutes of Health (NIH). Skip Over Navigation Links. glomerular diseases. Resources. NIDDK.
http://health.nih.gov/result.asp/1058

Home
Health Information Browse Health Topics Alphabetically Glomerular Diseases Resources NIDDK Clinical Trials Medline Plus NIH Institute and Center Resources National Institute of Diabetes and Digestive and Kidney Diseases
NIDDK
Directory of Kidney and Other Urologic Diseases Organizations Glomerular Diseases ... Your Urinary System and How It Works Related Topics: Kidney Diseases (General) Kidney and Urinary System Up to Top Look up "Glomerular Diseases" in: MEDLINEplus - health resources from US government agencies and other credible organizations Clinical Trials Database - research studies in which you can participate This page was last reviewed on Tuesday, February 24, 2004. Career Opportunities Visitor Information FOIA
Site Map
... Accessibility National Institutes of Health (NIH)
9000 Rockville Pike
Bethesda, Maryland 20892 Department of Health
and Human Services

49. MED On Line
has not been previously determined) and to identify any associated infections, renal biopsies were performed in 85 patients with glomerular diseases who were
http://www.medonline.com.br/med_ed/med8/miami1.htm
ASN, 2-7 de novembro de 1999. Glomerular Disease in Adults in the Northeast of Brazil: A Clinical and Pathological
Study.
Suzana M. Melo, Lucila V. Lopes, Alina Cavalcante, Alexis Harris, Sandra N. Coelho, Charles Jennette.
Nephrology, Federal University of Pernambuco, Recife PE, Brazil; Pathology, University of North Carolina,
Chapel Hill, NC.
Poster Session: Glomerular and Tubular Disorders: Risk Factors/Outcomes I (9:30 AM-5:00 PM)
Presentation Date: Friday, November 5, 1999, Time: 9:30AM, Room: Halls B and C.
aqui

50. Canine Glomerular Disease - WSAVA 2002
Use of the urine protein/creatinine ratio to identify and quantitate proteinuria has greatly facilitated diagnosis of these glomerular diseases in veterinary
http://www.vin.com/proceedings/Proceedings.plx?CID=WSAVA2002&PID=2706

51. Karger Publishers
Urinary Sediment Podocalyxin in Children with glomerular diseases Katsue Kanno a , Hiroshi Kawachi d , Yoshiaki Uchida c , Masanori Hara b , Fujio Shimizu d
http://content.karger.com/ProdukteDB/produkte.asp?Doi=74322

52. Glomerular
Kidney and Urologic Diseases Information Clearinghouse A detailed discussion about the kidneys and how glomerular diseases interfere with their functions.
http://www.thenewhealthfind.com/Health/ConditionsandDiseases/UrologicalDisorders
Directory Home Health Conditions and Diseases Urological Disorders : Glomerular (54)

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53. Clinical And Pathological Characteristics Of Patients With Glomerular Diseases A
Clinical and pathological characteristics of patients with glomerular diseases at a university teaching hospital 5year prospective review.
http://www.hkmj.org.hk/hkmj/abstracts/v5n3/240.htm
About the HKMJ Subscriptions Reprints Search past issues
Clinical and pathological characteristics of patients with glomerular diseases at a university teaching hospital: 5-year prospective review
KW Chan, TM Chan, IKP Cheng
Department of Pathology, The University of Hong Kong, Queen Mary Hospital, Pokfulam, Hong Kong OBJECTIVE: To examine the prevalence of glomerular disease in Hong Kong.
DESIGN: Prospective review.
SETTING: University teaching hospital, Hong Kong.
PATIENTS: All patients who presented with suspected glomerular disease from 1993 through 1997.
MAIN OUTCOME MEASURES: Histopathological diagnosis from biopsy examination and clinical features of presentation.
RESULTS: A total of 1629 consecutive percutaneous renal biopsies of native kidneys showed glomerular disease in 1413 cases. The most common clinical indication for renal biopsy was persistent proteinuria (n=735; 52.0%), while the most frequently found glomerular lesion was immunoglobulin A nephropathy (n=338; 23.9%). Minimal-change nephrotic syndrome (n=125; 8.8%) and immunoglobulin M nephropathy (n=11; 0.8%), were the most common glomerular diseases that presented with nephrotic syndrome. The male to female ratio for lupus nephritis was 1:14 (n=290), whereas for minimal-change nephrotic syndrome, the ratio was 1.8:1 (n=125). Immunoglobulin A nephropathy and membranous glomerulonephritis (n=117) affected approximately equal numbers of male and female patients. Familial fibrillary glomerulonephritis, a disease hitherto unknown in Hong Kong, was diagnosed in two siblings.

54. Kidney Disease
glomerular diseases glomerular diseases are those that attack the blood filtering units of the kidneys. Diabetes and high blood
http://www.labtestsonline.org/understanding/conditions/kidney-2.html
TESTS Test not listed? A/G Ratio ACE ACT ACTH AFB Culture AFP Maternal AFP Tumor Marker Albumin Aldosterone Allergies ALP Alpha-1 Antitrypsin ALT Amylase ANA Antibody Tests Apo A Apo B ApoE Genotyping aPTT AST Autoantibodies Bilirubin Blood Culture Blood Gases Blood Smear BMP BNP Bone Markers BRCA BUN C-peptide CA-125 CA 15-3 CA 19-9 Calcium Cardiac Risk CBC CEA Celiac Disease Tests CF Gene Mutation Chlamydia Chloride Cholesterol CK CK-MB CMP CMV Coagulation Factors Complement Levels Cortisol Creatinine Creatinine Clearance CRP CRP, high-sensitivity Cystatin C D-dimer DHEAS Differential DLDL EGFR Electrolytes Electrophoresis ESR Estrogen Estrogen Receptors Factor V Leiden Fecal Occult Blood Ferritin fFN Fibrinogen Flu Tests Folate Fructosamine FSH Genotypic Resistance GFR GGT Glucose Gonorrhea Gram Stain Growth Hormone H-pylori hCG HDL Hematocrit Hemoglobin Hemoglobin Variants Hepatitis A Hepatitis B Hepatitis C Her-2/neu Herpes HIV Antibody HLA-B27 Home Tests Homocysteine HPV hs-CRP IGF-1

55. Complementary Medicines
First Name Last Name email Note you can unsubscribe any time you wish. glomerular diseases. How Do glomerular diseases Interfere With Kidney Function?
http://www.complementarymedicines.com/ailments/ailments_display.php?id=36

56. Blackwell Synergy - Cookie Absent
Complement regulatory proteins in glomerular diseases. Masaomi Nangaku. Complement regulatory proteins in glomerular diseases.
http://www.blackwell-synergy.com/links/doi/10.1046/j.1523-1755.1998.00130.x/full
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57. Blackwell Synergy - Cookie Absent
PERSPECTIVES IN BASIC SCIENCE. Progression of glomerular diseases Is the podocyte the culprit? Progression of glomerular diseases Is the podocyte the culprit?
http://www.blackwell-synergy.com/links/doi/10.1046/j.1523-1755.1998.00044.x/enha
 Home An Error Occurred Setting Your User Cookie A cookie is a small amount of information that a web site copies onto your hard drive. Synergy uses cookies to improve performance by remembering that you are logged in when you go from page to page. If the cookie cannot be set correctly, then Synergy cannot determine whether you are logged in and a new session will be created for each page you visit. This slows the system down. Therefore, you must accept the Synergy cookie to use the system. What Gets Stored in a Cookie? Synergy only stores a session ID in the cookie, no other information is captured. In general, only the information that you provide, or the choices you make while visiting a web site, can be stored in a cookie. For example, the site cannot determine your email name unless you choose to type it. Allowing a web site to create a cookie does not give that or any other site access to the rest of your computer, and only the site that created the cookie can read it. Please read our for more information about data collected on this site.

58. Berger Disease - Medstudents - Neprhology
are increased in the serum of 5095% of patients, and its measurement is extremely useful in differentiating IgA nephropathy from other glomerular diseases.
http://www.medstudents.com.br/nefro/nefro2.htm
Nephrology
Bernardo Boaventura Liberato
Medstudents' Homepage
Berger Disease
Also known as primary IgA nephropathy, glomerulonephritis with IgA and IgG deposits and IgA mesangial nephropathy, Berger disease has a wide pattern of distribution and is believed to be the most common form of primary glomerular disease throughout the world. Although highly prevalent, geographic variations occur, which could be due to genetic as well as enviromental influences. Extensive mesangial IgA deposits (the characteristic pathologic feature of Berger disease), may also occur in a variety of multisystem, neoplastic and infectious diseases; when this occurs the term Secondary IgA Nephropathy should be applied. Examples of the most common causes are: Multisystem disease
  • Schönlein-Henoch purpura
  • Celiac Disease
  • Dermatitis Herpetiformis
  • Crohn Disease
    Neoplasia
  • Carcinoma of lungs, larynx, pharynx and pancreas
    Infection
  • Leprosy
  • Toxoplasmosis
  • HIV infection
    Others
  • Chronic liver disease
  • Portosystemic shunts
    In spite of the extremely varied clinical presentation, the most common feature leading to the diagnosis, by renal biopsy, is recurring episodes of macroscopic hematuria (which is generally the initial complaint). More than 80% of patients are between the ages of 16 to 35 years at the time of diagnosis. However, the disorder may be found at any age, although being uncommon before the age of 10 and after the age of 50. Thus, it should be considered a disease of children and young adults. There is a predilection for males (2:1), which disappears when affecting black persons (this disease is uncommon in blacks).
  • 59. Committee On Appropriations
    Mr. Chairman, FSGS is one of a cluster of glomerular diseases that attack the one million tiny filtering units contained in each human kidney.
    http://appropriations.house.gov/index.cfm?Fuseaction=Hearings.Testimony&HearingI

    60. Entrez PubMed
    Renal function in proteinuric glomerular diseases correlates to the changes in urine IgM excretion but not to the changes in the degree of albuminuria.
    http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=1

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