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         Factor V Leiden:     more detail
  1. Stroke after Marijuana smoking in a teenager with factor V Leiden mutation.(Brief Article): An article from: Southern Medical Journal by Mark A. Marinella, 2001-12-01
  2. Estrogen use with factor V Leiden not advised.(Women's Health)(Clinical report): An article from: Internal Medicine News by Colin Nelson, 2006-08-15
  3. Factor V Leiden genetic variant in an American Indian population.(COMMUNICATIONS--PROFESSIONAL): An article from: Proceedings of the North Dakota Academy of Science by Melanie Nadeau, Sheri T. Dorsam, et all 2007-04-01
  4. Genetic Polymorphisms: Single Nucleotide Polymorphisms, 5-Httlpr, Factor V Leiden, Rs6265, Rs6313, Rs6295, Rs5569, Rs6311, Rs6314, Rs7997012
  5. Single Nucleotide Polymorphisms: Factor V Leiden, Rs6265, Rs6313, Rs6295, Rs5569, Rs6311, Rs6314, Rs7997012, Rs1805054, Rs4680, Rs1801133
  6. Factor V Leiden thrombophilia: An entry from Thomson Gale's <i>Gale Encyclopedia of Genetic Disorders, 2nd ed.</i> by Dawn, MS, CGC Jacob, 2005
  7. Blood Proteins: Hemoglobin, Hemocyanin, Glycated Hemoglobin, Haptoglobin, Human Serum Albumin, Fibrin, Factor V Leiden
  8. Factor V Leiden as a common genetic risk factor for venous thromboembolism.(Genomics to Health): An article from: Journal of Nursing Scholarship by McDonald K., III Horne, Donna Jo McCloskey, 2006-03-22
  9. Factor V Leiden

61. Re Group For Factor V Leiden And PG
Highrisk Pregnancy Discussion Board. Re Group for factor v leiden and PG In Response To Group for factor v leiden and PG (Kitty).

62. International Factor V Leiden Meetup Day
International factor v leiden Meetup Day. in 16 Days. WHAT. Meetup with other Join other factor v leiden Sufferers near you! factor v leiden
@import url(""); @import url(""); @import url(""); @import url(""); Your Meetup now has its own Message Boards! Join the conversation! Meetup Home Discuss Sign In ... Factor V Leiden
International Factor V Leiden Meetup Day
in 7 Days
WHAT Meetup with other local people who have been diagnosed with Factor V Leiden (FVL). Offer support and advice on how to deal with difficulties, such as DVTs, pregnancy, fertility, passing on the genetic condition to our children, stroke risks, etc. WHEN Wednesday, June 9 @ 7:00PM
(2nd Wednesday of every month.) WHO Factor V Leiden Sufferers Worldwide (and friends.) So far, have signed up. AGENDA T.B.D. More info.
Join other Factor V Leiden Sufferers near you!
Factor V Leiden Meetups can happen in up to 646 cities worldwide on the same day. Enter your location to find the one near you: writeForm("horiz") US Residents, enter your

63. August 1995 - The Factor V Leiden Mutation
August, 1995. THE factor v leiden MUTATION. A MAJOR RISK FACTOR FOR INHERITED THROMBOSIS. Franklin A. Bontempo, MD, Medical Director, Coagulation Services.
August, 1995
THE FACTOR V LEIDEN MUTATION A MAJOR RISK FACTOR FOR INHERITED THROMBOSIS Franklin A. Bontempo, M.D., Medical Director, Coagulation Services Andrea Cortese Hassett, Ph.D., Scientific Director, Coagulation Services Introduction
Traditional attempts to identify an underlying cause of familial thrombosis by testing for antithrombin III, protein C, and protein S have only rarely provided an explanation for a patient's thrombotic event. Recently, studies have clearly shown that a mutation of clotting factor V is highly associated with thrombosis and may account for as much as 25% of all cases of venous thrombosis of unknown cause. In addition, knowledge that a patient carries this gene may have significant clinical implications. Pathophysiology The factor V mutation, named factor V Leiden after the site of its discovery, is due to a point mutation in the normal factor V molecule. This mutation renders the factor V molecule insensitive to the action of activated protein C, a natural anticoagulant. This in turn appears to shift the patient's overall hemostatic balance toward thrombosis, especially venous thrombosis. Incidence The incidence of the factor V Leiden mutation in both European and American populations has been repeatedly found to be surprisingly high. The best evidence from the United States indicates that 6% of the population carries the gene for the factor V mutation. The reason for its persistence at such a high level is unclear.

64. Factor V Leiden, Factor V, V Leiden, Coagulation, Thrombosis, Genetic, Molecular
factor v leiden testing by ProADN. For the analysis of factor v genetic mutations, chose ProADN s home factor v leiden tests for canada, montreal, quebec.
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For any comment about this site: New Predisposition Factors The Factor V Leiden The factor V gene mutation, known as the factor V Leiden, is present in 3 to 8% of the Caucasian population in the heterozygous state. The prevalence of this gene in the homozygous state is only about 0.1%. While only 8% of the individuals with a heterozygous gene present with a thrombosis, nearly all those with the homozygous gene will have a thrombotic manifestation during their lifetime. Moreover, this mutation has also been identified in significant numbers in women who have had obstetrical complications (preeclampsia, placental detachment, still-born baby, intra-uterine growth retardation, repeated pregnancy losses). For young women with the factor V Leiden mutation, taking birth control pills increases the risk of thrombosis thirty-five (35) times compared with the risk of those who are not carriers of this mutation. Indications for Investigation of Genetic Factors for Thrombophilia
  • Venous thromboembolism (thrombophlebitis, pulmonary embolism)

65. Factor V Leiden - MSU Tissue Typing Laboratory
factor v leiden. Staff Directory John Gerlach, Ph.D., dipABHI Peggy J. Bull, CHS.
Factor V Leiden
Staff Directory : John Gerlach, Ph.D., dipABHI
Peggy J. Bull, CHS

66. Kimball Genetics - Factor V Leiden DNA Test
factor v leiden DNA Test. Indications for factor v leiden DNA Testing Venous thrombosis. Pulmonary embolism. Transient ischemic attack or premature stroke.
Factor V Leiden DNA Test
The factor V Leiden mutation is the most common genetic risk factor for venous thrombosis, a serious health problem affecting approximately 1 in 1000 people and leading to 50,000 deaths annually in this country. This mutation is involved in 20-40% of venous thrombosis cases and is present in 5% of the general population. The Factor V Leiden DNA Test should be included in the evaluation of all patients with thrombosis or a family history of this condition. Venous thrombosis is multigenic, and up to a third of individuals affected with inherited thrombosis have two or more genetic defects. The Factor V Leiden mutation is also associated with multiple pregnancy loss and intrauterine fetal demise. Kimball Genetics provides the Factor V Leiden DNA Test separately, in combination with the Prothrombin (Factor II) DNA Test, or as part of panels including coagulation tests for other inherited hypercoagulability disorders.
Indications for Factor V Leiden DNA Testing:
Venous thrombosis Pulmonary embolism Transient ischemic attack or premature stroke Peripheral vascular disease, particularly lower extremity occlusive disease

67. Kimball Genetics - The Factor V R2 DNA Test
factor v leiden is the most common genetic risk factor for venous thrombosis and pulmonary embolism, present in 5% of the Caucasian population and in 2040% of
The Factor V R2 DNA Test
Factor V Leiden is the most common genetic risk factor for venous thrombosis and pulmonary embolism, present in 5% of the Caucasian population and in 20-40% of individuals with a history of venous thromboembolism. Not all individuals who are heterozygous for factor V Leiden will experience a thrombotic event. The presence of the R2 polymorphism in the factor V gene increases the likelihood of thrombosis in factor V Leiden heterozygotes. The R2 polymorphism is very common, occurring in 1 in 10 individuals. This polymorphism is part of the HR2 haplotype, a collection of polymorphisms always inherited together.
Factor V Leiden heterozygotes are at a 7-fold increased risk for venous thrombosis. Co-existence of the R2 polymorphism with factor V Leiden increases that risk by an additional 3-fold through a further increase in APC resistance. The average age of the first thrombotic event is six years younger in individuals who are heterozygous for both factor V Leiden and the R2 polymorphism than in those with factor V Leiden alone. Because of this dramatic increase in the risk for a life-threatening event, it is important to identify those factor V Leiden heterozygotes who also have the R2 polymorphism.
The R2 polymorphism is identified by PCR amplification and restriction enzyme digestion followed by gel electrophoresis. Within 24 hours of the detection of factor V Leiden, the presence of the additional risk factor, R2, can be determined without an additional blood draw, and an appropriate plan of care can then be initiated.

68. World History: Fabian Doles Thru Facsimile
Return to Main Article Index Home Page. INDEX OF ARTICLES Fabian Steinheil thru factor v leiden. Factor price equalization, factor v leiden. Bringing History to Life
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69. Factor V Leiden And Contraception
factor v leiden is a fairly new phenomenon. From Journal of Midwifery Women s Health. factor v leiden and Contraception. Posted 01/27/2004.

factor v leiden ASSAY (1380006). SYNONYMS Protein C resistance, APC Resistance, Activated Protein C resistance. CPT 4 CODE 83890, 83894, 83905.
A B C D ... LSG Home FACTOR V LEIDEN ASSAY (138-0006) SYNONYMS: Protein C resistance, APC Resistance, Activated Protein C resistance CPT 4 CODE: Test Order Mnemonic: FACT5 PCR Applies to: Mutation of Factor V clotting factor Lab: Molecular Diagnostic Laboratory Request Form: Must be manually written on any of the available Laboratory Request Forms Collection: Routine Venipuncture Storage Instructions: Whole Blood Refrigerated Causes for Rejection: Serum, heparin collection tube Availability: Samples are accepted Monday-Friday, 8:00 AM to 5:00 PM, Room 5.212, John Sealy Annex, or any time in Specimen Receiving, McCullough 5.136 Special Instructions: Do not Centrifuge or separate specimen Specimen: Whole blood with EDTA (purple) or Sodium Citrate (blue) Volume: 5 mls. Minimum Volume: 1 ml Container: EDTA (purple) or sodium citrate (blue) Reviewed by Dr. Payne/ S. Seifert 2/00 A B C D ... Statewide Search
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71. Factor V Leiden :: Index
Het is nu Ma Mei 24, 2004 901 am factor v leiden Forum Index, Bekijk onbeantwoorde berichten. Forum, Onderwerpen, Berichten, Laatste Bericht. factor v leiden,

72. Health Library -
support groups. factor v leiden / Thrombophilia. Self Help Clearinghouse. factor v leiden Mailing List and Digest. Online.Mailing

73. Liver Information: Budd-Chiari Syndrome Associated With Factor V Leiden Mutation
Liver Transplant Program and Center for Liver Disease. BuddChiari Syndrome Associated with factor v leiden Mutation A Report of 6 patients. Hoffman R et al.
Budd-Chiari Syndrome Associated with Factor V Leiden Mutation: A Report of 6 patients. Hoffman R et al. Liver Transplantation and Surgery 1999;5:96-100 , (Haifa, Israel)
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factor v leiden , BLOOD. NOTE An “Activated Protein C Resistance” (p. 34), must be performed before this test can be ordered.
FACTOR V LEIDEN , BLOOD NOTE: An “Activated Protein C Resistance” (p. 34), must be performed before this test can be ordered. Reflex test if APCR is Abnormal. Test Performed At: Barnes Jewish Hospital Molecular Diagnostics Lab Specimen Required: Draw blood in a lavender-top (EDTA) tube(s). DO NOT CENTRIFUGE. (CLOTTED BLOOD OR AN INSUFFICIENT SPECIMEN IS NOT ACCEPTABLE.) Send 3.0 mL of EDTA whole blood. Place specimen on wet ice and forward promptly to St. Louis Children’s Hospital. Avoid hemolysis. NOTE: Draw a discard tube prior to lavender-top (EDTA) tube(s). Tube(s) should remain stoppered. Laboratory Notes: Reference Values: Interpretative report Day(s) Test Set Up: Monday through Friday Turn Around Time: CPT Code: 83901 x 2 - amplification 83894 x 2 - electrophoresis 83891 - extraction 83912 - interpretation and report Care Manager Order Name: Factor V Leiden Cerner Order Name: Factor V Leiden

75. 11 March 2004
Carriers of factor v leiden or prothrombin gene mutations have double the risk of experiencing two or more miscarriages Recurrent pregnancy loss is a
Carriers of Factor V Leiden or prothrombin gene mutations have double the risk of experiencing two or more miscarriages
Recurrent pregnancy loss is a significant clinical problem. Recently, thrombophilias have been implicated as a possible cause. Factor V Leiden and prothrombin gene (G20210A) mutations are the most common types of hereditary thrombophilias, but are usually undiagnosed because most carriers are asymptomatic. The relationship between Factor V Leiden, prothrombin gene (G20210A) mutations, and recurrent pregnancy loss has been investigated with conflicting results. Thus US researchers analyzed existing data to determine whether an association exists in a systematic review of the literature. 16 studies were selected for the Factor V Leiden meta-analysis and 7 for the prothrombin gene (G20210A) mutations analysis.
The researchers concluded: "Carriers of Factor V Leiden or prothrombin gene mutations have double the risk of experiencing 2 or more miscarriages compared with women without thrombophilias. Hereditary thrombophilias may be an unrecognized cause of recurrent pregnancy loss. We recommend testing for these mutations in women with recurrent pregnancy loss." Arch Intern Med . 2004;164:558-563. March 8, 2004

76. Specialty Laboratories We Help Doctors Help Patients
Print View. factor v leiden and Activated Protein C Resistance A single adenineto-guanine point mutation at base 1691 in the gene coding for factor V causes

77. Factor V (Leiden), DNA Analysis
factor v leiden, DNA Analysis Back Use your browser s Back button to return to the previous page. x2. Test ID, FVLC. SMS Browse, factor v leiden DNA Analysis.
Factor V Leiden, DNA Analysis Back Use your browser's "Back" button to return to the previous page Tube Type Test ID FVLC SMS Browse FACTOR V LEIDEN DNA Analysis CPT Code Laboratory Molecular Genetics Tube Station Specimen Routine: 3 mL blood/Yellow Top (ACD) Tube or Lavender top EDTA tube micro: 1.0 mL Blood/3x EDTA Pediatric Bullet/ Lavender Top Availability Routine: Weekly; no weekends or holidays STAT: N/A Turnaround Time 10 days Reference Range Normal: No evidence of mutation Comments Additional information: Factor V Leidin Associated with Inherited Predisposition to Venous Thrombosis pdf file PDF files may be read using the free Acrobat Reader from Adobe Systems Incorporated, which may be downloaded from the Adobe site ( After installation of the Acrobat Reader, memos may be retrieved by clicking on the Subject. Return to Test Information Introduction Page
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78. BioMed Central | Full Text | The Relationship Of The Factor V Leiden Mutation Or
The relationship of the factor v leiden mutation or the deletiondeletion polymorphism of the angiotensin converting enzyme to postoperative thromboembolic
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PubMed record ... Related articles in PubMed Search PubMed For Della Valle CJ Issack PS Baitner A Steiger DJ ... Di Cesare PE Key E-mail Corresponding author Research article The relationship of the factor V Leiden mutation or the deletion-deletion polymorphism of the angiotensin converting enzyme to postoperative thromboembolic events following total joint arthroplasty Craig J Della Valle MD Paul S Issack Avi Baitner David J Steiger Carrie Fang and Paul E Di Cesare Musculoskeletal Research Center, Room 1500 NYU-Hospital for Joint Diseases Department of Orthopaedic Surgery, 301 East 17th Street New York, USA Department of Medicine New York University-Hospital for Joint Diseases 301 East 17th Street New York, USA BMC Musculoskeletal Disorders The electronic version of this article is the complete one and can be found online at: Received Accepted Published Outline Abstract Abstract Background Patients and Methods Statistical Analysis ... Pre-publication history Background Although all patients undergoing total joint arthroplasty are subjected to similar risk factors that predispose to thromboembolism, only a subset of patients develop this complication. The objective of this study was to determine whether a specific genetic profile is associated with a higher risk of developing a postoperative thromboembolic complication. Specifically, we examined if the Factor V Leiden (FVL) mutation or the deletion polymorphism of the angiotensin-converting enzyme (ACE) gene increased a patient's risk for postoperative thromboembolic events. The FVL mutation has been associated with an increased risk of idiopathic thromboembolism and the deletion polymorphism of the ACE gene has been associated with increased vascular tone, attenuated fibrinolysis and increased platelet aggregation.

79. ARUP Factor V Leiden, Fetal
search. factor v leiden, Fetal, , Allergens, A, B, C, D, E, F, G, H, I, J, K, L, M, N, O, P, Q, R, S, T, U, V, W, X, Y, Z, Search UG. ARUP s User s Guide. 0050766 factor v leiden, Fetal.

80. Hematology: Heparin Reverses Preeclampsia In A Factor V Leiden Mutation Carrier, Banner Ad (468x60). Heparin reverses preeclampsia in a factor v leiden mutation carrier. Hematology May 6, 2004 2004 MAY
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