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         Cerebellar Diseases:     more books (29)
  1. Cerebellar Disorders: A Practical Approach to Diagnosis and Management
  2. Handbook of Cerebellar Diseases (Neurological Disease and Therapy)
  3. Cerebellar Infarct Midline Tumors Minimally Invasive Endoscopic Neurosurgery (Advances in Neurosurgery) by Bernhard Ludwig Bauer, M. Brock, 1994-04
  4. Parenchymatous Atrophy of the Cerebellum: A Contribution to the Symptomatology of Intrinsic Cerebellar Disease by La Salle Archambault, 1918-01-01
  5. Comparison of patients with Parkinson's disease or cerebellar lesions in the production of periodic movements involving event-based or emergent timing [An article from: Brain and Cognition] by R.M.C. Spencer, R.B. Ivry, 2005-06-01
  6. Cerebellar functions (Nervous and mental disease monograph series) by André Thomas, 1912
  7. The localisation of cerebral disease: Being the Gulstonian lectures of the Royal College of Physicians for 1878 by David Ferrier, 1879
  8. Cerebellar abscess secondary to ear disease: Illustrated by a case successfully treated by operation : with remarks on diagnosis, and with a table of published cases by Theodore D Acland, 1896
  9. Cerebellar abscess secondary to ear disease, by Charles Alfred Ballance, 1896
  10. Cerebellar Functions. Nervous and Mental Disease Monograph Series No. 12 by Andre Thomas] Andre-Thomas [ie, 1912-01-01
  11. Malignant pleural mesothelioma with scalp, cerebellar, and finger metastases: a rare case.(Disease/Disorder overview): An article from: Southern Medical Journal by Asiye Kanbay, Kivilcim Ipek Oguzulgen, et all 2007-01-01
  12. CURRENT Diagnosis & Treatment in Neurology (LANGE CURRENT Series) by John Brust, 2006-09-14
  13. Speech motor programming in hypokinetic and ataxic dysarthria [An article from: Brain and Language] by K.A. Spencer, M.A. Rogers,
  14. Hemangiomas of cerebellum and retina (Lindau's disease): With the report of a case by Harvey Cushing, 1928

61. Veterinary Information - Neurologic Diseases And Disorders Of Dogs
textbooks for information. cerebellar abiotrophy is a progressivedisease that affects the cerebellum. Dogs with this condition
http://www.vetinfo.com/dneuro.html
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Neurological Diseases and Disorders of Dogs
Neurological problem in Brittany

Auditory-triggered spasms in elderly Sheltie

Treating spinal cord problem

Sudden paralysis in Rottie
...
Pug Encephalitis
also see Meningitis
also see Myasthenia Gravis
also see Head tremors also see seizure control also see White Shaker Dog Syndrome also see Granulometous meningioencephalitis (GME) also see Degenerative Myelopthy also see Epilepsy also see Hepatic Encephalopathy also see Motion and Motor Problems also see Seizure also see Trauma also see Vestibular also see Fibrocartilaginous embolism also see Brain tumor also see MRI Please note: The most recent medical information is at the top of the page to least current at the bottom.
Neurological problem - in Brittany Question: Dear Dr. Mike

62. Cerebellar Degeneration - Diseases & Conditions
cerebellar Degeneration diseases Conditions. diseases Conditions- Index cerebellar Degeneration. cerebellar Degeneration - following
http://www.spiritindia.com/diseases-conditions-47.html

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Cerebellar Degeneration
Cerebellar Degeneration - following article(s) are best informative resources for Cerebellar Degeneration.

63. NEUROLOGICAL DISEASES CAUSED BY VIRUSES
disease typically occurs between the ages of 50 and 65 years. There are two mainmodes of presentation 1. Chronic dementing illness; 2. Progressive cerebellar
http://www.uct.ac.za/depts/mmi/jmoodie/neurol2.html
NEUROLOGICAL DISEASES CAUSED BY VIRUSES
Neurological disease is one of the most serious complications of virus infection.
Clinically, viral neurological disease can be divided into
acute diseases
and chronic syndromes
The pathology may be due either to viral multiplication in the cells of the brain, or due to the (misdirected) immune response of the host - post infectious encephalo-myelitis
Where virus replicates in the brain, virus can usually be isolated from brain tissue or from cerebrospinal fluid. This is not the case with the post infectious syndromes.
Acute neurological syndromes
Virus may reach the brain either by the blood stream or by spread along peripheral nerves. Asymptomatic infection of the brain is common. There are four main syndromes:
Acute viral encephalitis
Viral replication occurs in the brain tissue itself, causing destructive lesions in the grey matter. The main symptoms include: fever, drowsiness, confusion, convulsions

64. Prion Diseases
it occurs typically in the 4th5th decade, characterised by cerebellar ataxia andconcomitant motor problems, dementia less common and disease course lasts
http://www-micro.msb.le.ac.uk/3035/prions.html
: Prions Updated: January 20, 2004 Search See: Principles of Molecular Virology: Chapter 8 Amazon.co.uk ...
Instructors Version
In addition to all the material on the Standard Version CD, the Instructor's CD also contains all the figures from the book in electronic form and a PowerPoint slide set with complete lecture notes to aid course preparation. ( Amazon.co.uk
Prion Diseases
Latest: BSE in the USA
This document describes infectious agents which (almost certainly) do not have a nucleic acid genome. It seems that a protein alone is the infectious agent. The infectious agent has been called a prion . A prion has been defined as " small proteinaceous infectious particles which resist inactivation by procedures that modify nucleic acids ". The discovery that proteins alone can transmit an infectious disease has come as a considerable surprise to the scientific community.
Prion diseases are often called spongiform encephalopathies because of the post mortem appearance of the brain with large vacuoles in the cortex and cerebellum. Probably most mammalian species develop these diseases. Specific examples include:
  • Scrapie : sheep TME (transmissible mink encephalopathy): mink CWD (chronic wasting disease): muledeer, elk

65. Demyelinating Diseases
It is not primarily a periventricular process, but as the disease progresses, the Brainstem and cerebellar lesions are found in about onethird of patients
http://spinwarp.ucsd.edu/NeuroWeb/Text/br-840.htm
DEMYELINATING DISEASES OF THE BRAIN John R. Hesselink, MD, FACR
MR imaging is exquisitely sensitive for detecting brain abnormalities. Particularly in the evaluation of white matter diseases, MR far outperforms any other imaging technique. Lesions that may be quite subtle or even invisible on CT are often clearly seen on the MR scan. The MR signal characteristics of white matter lesions are similar and relatively nonspecific, but other distinguishing features are often present to assist in diagnosis, such as the pattern of the abnormality, location, and enhancement features. The white matter is affected by many disease processes. The primary demyelinating disease is multiple sclerosis, but many other metabolic and inflammatory disorders result in deficient or abnormal myelination. Histologically, myelin abnormalities are either demyelinating or dysmyelinating. Demyelination implies destruction of myelin. Dysmyelination refers to defective formation or maintenance of myelin resulting from dysfunction of the oligodendrocytes. Most of the dysmyelinating disorders are caused by metabolic defects that present in infancy. White matter diseases in older children and adults are generally demyelinating or a combination of the two processes.
NORMAL WHITE MATTER The white matter of the brain is located in the central and subcortical regions of the cerebral and cerebellar hemispheres and accounts for about 60 % of the total brain volume. The white matter includes the major commissural tracts, the cortical association fibers, and all the cortical afferent and efferent fibers. Histologically, the white matter contains nerve fibers, supporting cells, interstitial space, and vascular structures. White matter consists mostly of axons with their envelope of myelin, along with two types of neuroglia - oligodendrocytes and astrocytes. Axons are extensions of neurons that reside within the gray matter of the brain, spinal cord, and ganglia. The myelin is produced and maintained by oligodendrocytes. Myelin functions as an insulator of the axons, and its structure facilitates rapid transmission of impulses.

66. Disease - Spinocerebellar Ataxia (SCA)
SearchTerm, Joseph s disease, Hereditary cerebellar ataxia, Spinocerebellar degeneration,Marie s ataxia, Inherited olivopontocerebellar atrophy, Familial
http://www.rehabinfo.net/resources/diseases/list/disease.asp?id=27

67. ORPHANET - Rare Diseases - Orphan Drugs
Printing version, DISEASE cerebellar ataxia and hypogonadism, Synonym(s)Luteinizing hormone releasing hormone deficiency of with ataxia,
http://www.orpha.net/consor/cgi-bin/OC_Exp.php?Lng=GB&Expert=1173

68. Neurological Disorders: Brain Diseases: Cerebellar
Neurological Disorders Brain diseases cerebellar.
http://www.puredirectory.com/Health/Conditions-and-Diseases/Neurological-Disorde
Neurological Disorders: Brain Diseases: Cerebellar
Home Health Conditions and Diseases Neurological Disorders ... Brain Diseases : Cerebellar google_ad_client = 'pub-3272565765518472';google_ad_width = 336;google_ad_height = 280;google_ad_format = '336x280_as';google_color_border = 'FFFFFF';google_color_bg = 'FFFFFF';google_ad_channel ='7485447737';google_alternate_color = 'FFFFFF';google_color_link = '0000FF';google_color_url = '008000';google_color_text = '000000';
Standard Listings
International Paraneoplastic Association
An article about paraneoplastic cerebellar degeneration with an explanation what it is, diagnosis, treatment, research and some notes to care give...
Paraneoplastic Cerebellar Degeneration
An article written by Timothy C. Hain, MD.
The Merck Manual: Disorders Of Movement
Information about cerebellar and spinocerebellar disorders. Includes a short discussion about friedreich's ataxia, cerebellar ataxias and multipl...
Help build the largest human-edited directory on the web. Submit a Site Open Directory Project Become an Editor PureDirectory.com uses a modified version of the Open Directory.

69. Neurobiology Of Disease Journal Entry 9
for a genetic subtype of the type I autosomal dominant cerebellar ataxias, clinically Theminor clinical differences between the two diseases may result from
http://www.neuro.wustl.edu/journal/jrnl9.htm
The gene for Machado-Joseph disease maps to the same 3-cM interval as the spinal cerebellar ataxia 3 gene on chromosome 14q
G. Stevanin: , P. S. Sousa: , G. Cancel: , A. Durr: , 0. Dubourg: , G. A. Nicholson: , J. Weissenbach: , E. Jardim: , Y. Agid: , E. Cassa: , A. Brice: )INSERM U289, Hopital de la Salpetriere, Paris, France, )Departamento de Neurologia, Hospital das Clinicas, Universidade de Sao Paulo, Ribeirao Preto, Sao Paulo, Brasil, )Department of Medicine, Molecular Genetics and Medicine Laboratory, University of Sydney, Concord Hospital, New South Wales, Australia and )Genethon, Evry, France Correspondence: Dr Alexis Brice, INSERM U289, Hopital de la Salpetriere, 47 Bd de I'Hopital, 75651 Parix Cedex 13, France. Fax: 00/33/l/44.24.36.58.
Summary
Machado-Joseph disease (MJD) is an autosomal dominant neurodegenerative disorder in families of Portuguese-Azorean ancestry. The gene responsible for MJD has been assigned to a 29-cM interval on chromosome 14q. A large Brazilian family with MJD was genotyped with six new microsatellite markers spanning 19 cM on chromosome 14q. Linkage analysis and haplotype reconstruction reduced the MJD candidate region to a 3-cM interval between markers D14S280 and D14S81, permitting positional cloning. This interval also contains the spinal cerebellar ataxia 3 (SCA3) gene, responsible for a genetic subtype of the type I autosomal dominant cerebellar ataxias, clinically related to MJD. This result supports the hypothesis that abnormalities in the same gene may be responsible for both disorders. The minor clinical differences between the two diseases may result from allelic heterogeneity.

70. Frequently Asked Questions
Primary neurological changes 11. Is it known why gait and balance are affectedearly in FA but not so much in other cerebellar degenerative diseases?
http://www.fan.asn.au/faq.html
FREQUENTLY ASKED QUESTIONS
1. What is the life expectancy of people with FA?
Cannot be predicted in a specific case and we are finding ‘older cases’ all the time. It is no longer just a disease of childhood.
2. Does FA cause impotence, or effect sexual response in males or females?
There is a peripheral nerve component to the disease but the autonomic nervous system doesn’t seem to be imparied. General physical disability might be a problem however.
3. Does insomnia occur with FA?
There are often sleeping problems associated with FA, which can be treated. These problems are usually only apparrent much later from the original onset of symptoms though.
4. Does it affect your sight/vision and hearing ability in all cases?
Only a small proportion, certainly not all
5. Does fa always cause fatigue?
Any neurological or neurodegenerative disease can cause easy fatigue.
6. Do all FAers have scoliosis? Can be a major problem. 7. Is the age of onset of FA related to the length of survival? To an extent, earlier onset , more severe disease. 8. Does age of onset relate at all to rapidity of speech deterioration?

71. Virtual Hospital: Infectious Diseases Of The Central Nervous System: Parenchymal
kuru, the practice of cannibalism has been stopped and the incidence of the diseasehas abated. Kuru has a slow onset and is manifested by cerebellar signs and
http://www.vh.org/adult/provider/pathology/CNSInfDisR2/Text/PInf.CDE.html
For Providers Infectious Diseases of the Central Nervous System
Parenchymal Infections: Prions
Gary Baumbach, M.D., Department of Pathology, University of Iowa College of Medicine
Peer Review Status: Internally Peer Reviewed Scrapie is a chronic disease of sheep which is transmitted by a filterable particle that is resistant to heat and formalin fixation. The human disease kuru , which is also caused by a filterable particle, is found in the Fore speaking people of New Guinea. The disease is perpetuated through the practice of cannibalism. These people eat the bodies of their own dead. Because only the women and children eat the brains, kuru is usually not found in the adult male population. Having discovered the relationship between cannibalism and kuru, the practice of cannibalism has been stopped and the incidence of the disease has abated. Kuru has a slow onset and is manifested by cerebellar signs and shaking ataxia. Death occurs with 18 to 24 months. Until recently, the infective agents of kuru and Creutzfeldt-Jakob disease in humans, and scrapie in sheep, were presumed to be slow, latent viruses. The latest hypothesis is that these diseases are caused by prions. Prions are composed of protein and are much smaller than viruses. Kuru
The gross findings in kuru are minimal, and consist of congestion of blood vessels, which can be seen on this slide

72. Microsoft Word - Movement Disorders
168DISEASES OF THE CEREBELLUMOBJECTIVES 1. List and be able to identify on tape clinical signs associated with cerebellar disorders.2. Provide a differential diagnosis for disease that affect the
http://education.vetmed.vt.edu/Curriculum/VM8654/move.pdf

73. Treatable Psychiatric Symptoms Common With Degenerative Brain Disease
and his colleagues conducted detailed interviews and analyzed brain images of threegroups 31 patients with degenerative cerebellar disease, 21 patients with
http://www.hopkinsmedicine.org/press/2002/August/020806.htm
August 6, 2002
MEDIA CONTACT: Trent Stockton
PHONE:
E-MAIL: tstockt1@jhmi.edu Treatable Psychiatric Symptoms Common With Degenerative Brain Disease
Potentially treatable psychiatric problems are common in patients with degenerative brain diseases affecting movement and coordination, according to a study by Johns Hopkins scientists. Up to 80 per cent of those with either Huntington's disease or degenerative diseases affecting the cerebellum also suffer from depression, impaired thinking and changes in personality, the study found. "Traditionally, there has been this distinction between neurological and psychiatric disorders, but it is an artificial distinction," said lead author Russell L. Margolis, M.D., associate professor of psychiatry at Hopkins and director of the Laboratory of Genetic Neurobiology. "Indeed, the high rate of psychiatric disorders in these patients suggests that many, if not most, can benefit from treatment, even if the course of the brain disease itself cannot be reversed. Many symptoms can be eased, and the quality of life for these patients and their families can be greatly enhanced," says Margolis. "Our findings of high rates of psychiatric disorders in Huntington's disease confirm the results of previous studies, and we believe we've found the first well-established link between serious psychiatric disorders and the cerebellum, a region at the back of the brain densely packed with nerve cells," notes Margolis.

74. EMedicine - Paraneoplastic Cerebellar Degeneration : Article By Abbas Mehdi, MD
When the condition is associated with Hodgkin lymphoma, patients are usually youngmen, and the cerebellar disease often follows the diagnosis of lymphoma.
http://www.emedicine.com/neuro/topic299.htm
(advertisement) Home Specialties Resource Centers CME ... Patient Education Articles Images CME Patient Education Advanced Search Consumer Health Link to this site Back to: eMedicine Specialties Neurology Neuro-oncology
Paraneoplastic Cerebellar Degeneration
Last Updated: November 12, 2003 Rate this Article Email to a Colleague Synonyms and related keywords: paraneoplastic neurological syndrome, PCD AUTHOR INFORMATION Section 1 of 10 Author Information Introduction Clinical Differentials ... Bibliography
Author: Abbas Mehdi, MD , Clinical Fellow, Clinical Instructor, Department of Neurology, University of Alabama at Birmingham Coauthor(s): David Y Ko, MD , Laboratory Director, Assistant Professor, Department of Neurology, University of Southern California Medical Center Abbas Mehdi, MD, is a member of the following medical societies: American Academy of Neurology American Epilepsy Society , and American Medical Association Editor(s): Frederick M Vincent, Sr, MD , Professor of Clinical Medicine, Psychiatry, and Neurology, Dept of Medicine, Dept of Psychiatry, Dept of Neurology, Michigan State University, College of Human Medicine,College of Osteopat;

75. Gordon Morgan Holmes (www.whonamedit.com)
StewartHolmes symptom Rebound phenomenon seen in cerebellar disease. Stewart-Holmessyndrome Epileptic fits, manifested by jerking movements of one arm.
http://www.whonamedit.com/doctor.cfm/1189.html

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This survey of medical eponyms and the persons behind them is meant as a general interest site only. No information found here must under any circumstances be used for medical purposes, diagnostically, therapeutically or otherwise. If you, or anybody close to you, is affected, or believe to be affected, by any condition mentioned here: see a doctor.
Gordon Morgan Holmes
Irish neurologist, born February 22, 1876, Dublin; died December 29, 1965, Farnham, Surrey.
Associated eponyms: Adie's syndrome A neurological phenomenon in which one or both pupils is dilated and responds slowly or not at all to light and a near stimulus, accompanied by slow constriction and relaxation in the change from near to distant vision, and impaired accommodation. Balint's syndrome A syndrome combining paralysis of visual fixation, optic ataxia, and impairment of visual fixation. Head-Holmes syndrome Sensory changes produced by lesions of the cerebral cortex and other parts of the brain. Holmes' syndrome I Inheritable disease picture with cerebellar ataxia due to a degeneration of cerebellum olivary nucleus.

76. Ataxia, Marie's
Synonyms cerebellar Syndrome; Hereditary cerebellar Ataxia; Nonne s Syndrome;PierreMarie s Disease. Disorder Subdivisions None. General Discussion
http://my.webmd.com/hw/health_guide_atoz/nord403.asp
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You are in Medical Library Choose a Topic Our Content Sources Ask A Question Clinical Trials Health Guide A-Z Health Topics Symptoms Medical Tests Medications ... For a Complete Report Ataxia, Marie's Important It is possible that the main title of the report Ataxia, Marie's is not the name you expected. Please check the synonyms listing to find the alternate name(s) and disorder subdivision(s) covered by this report. Synonyms
  • Cerebellar Syndrome Hereditary Cerebellar Ataxia Nonne's Syndrome Pierre-Marie's Disease
Disorder Subdivisions
  • None
General Discussion Marie's Ataxia is an inherited disorder of impaired muscle coordination usually beginning during young adulthood or middle age. This hereditary form of ataxia is characterized by unsteady walking. Nerve degeneration and muscle atrophy in the legs, head and neck area and arms may occur. Cases that begin later in life may be mild with symptoms that can often be treated successfully. Resources WE MOVE (Worldwide Education and Awareness for Movement Disorders) 204 West 84th Street New York, NY 10024

77. Hartnup Disease,H Disease,Hart Syndrome,Pellagra-Cerebellar Ataxia-Renal Aminoac
Hartnup Disease,H Disease,Hart Syndrome,Pellagracerebellar Ataxia-Renal AminoaciduriaSyndrome,Tryptophan Pyrrolase Deficiency,Hartnup Syndrome,Hartnup
http://www.icomm.ca/geneinfo/hartnup.htm
For Information on Workshops and Seminars for Special Needs Children click here The GAPS INDEX
to Information on the Internet about Genetic Disorders and Birth Defects

Genetic Information and Patient Services, Inc. (GAPS)
HOME
DISORDERS GLOSSARY Hartnup Disease
(as defined by the
National Organization for Rare Disorders

also known as:
H Disease Hart Syndrome
Pellagra-Cerebellar Ataxia-Renal Aminoaciduria Syndrome
Tryptophan Pyrrolase Deficiency
Hartnup Syndrome Hartnup Disorder
Hartnup Disease is a rare metabolic disorder inherited as a recessive trait. It involves an inborn error of amino acid metabolism as well as niacin deficiency. Factors which may precipitate acute attacks of this disorder may include poor nutrition, exposure to sunlight, sulfonamide medications and/or psychological stress. Hartnup Disease may be marked by skin problems, coordination impairment, vision problems, mild mental retardation, grastrointestinal problems, and central nervous system abnormalities. Frequency of attacks usually diminish with age.

78. Cerebellar Ataxia An Autosomol Recessive Genetic Disease Identified In The Spino
Click Here to find out how YOU can Help, cerebellar Ataxia a GeneticDisease identified in the Spinoni Italiani. This year, an inherited
http://www.spinone.com/Health/CA/CA_Article.htm
Cerebellar Ataxia a Genetic Disease identified in the Spinoni Italiani
This year, an inherited disease which manifests itself as a Cerebellar Ataxia, or abnormal gait originating from a problem in a part of the brain, was identified in Spinoni. It was brought to light in Great Britain, when an eagerly awaited and carefully planned litter was born to Christine Bennet and Pat Wilkinson. Initially normal, the puppies began to have problems getting up at about 5 months, and by 11 months, after Christine's puppy had won Best in Show, the puppy was dead, and another no longer could walk and was euthanized. Largely through the sustained efforts of Ms. Wilkinson who was able to enlist the help of a well known geneticist, Dr Bruce Cattanach, and equally known neurologist at the Royal Veterinary College, Dr. Simon Wheeler, much has been identified about this disease through the study of the live animals and their post mortum tissue, as well as intense investigation of the pedigrees of affected animals. Their conclusions are these: 1. This particular Cerebellar Ataxia (henceforth in this article, "CA") is inherited as an autosomal recessive gene. This means that a puppy must have inherited a copy of the abnormal gene from both the sire and the dam for the puppy to display the disease. It means that both parents carried the gene, but did not suffer from the disease. A litter from a mating of two carriers will produce on average 25% puppies who will die of the disease, 50% who are well but are carriers and if mated will pass on the gene to 50% of their offspring, and 25% totally normal pups.

79. National Cancer Institute - Childhood Cerebellar Astrocytoma Treatment
Recurrence may take place in childhood cerebellar gliomas and may develop manyyears after initial treatment. Disease can be at the primary tumor site or
http://www.cancer.gov/cancerinfo/pdq/treatment/child-cerebellar-astrocytoma/heal
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NCI Highlights Some Men with Low PSAs Have Prostate Cancer Energy Balance Trans-HHS Cancer Health Disparities Report Past Highlights General Information This cancer treatment information summary provides an overview of the diagnosis, classification, treatment, and prognosis of childhood cerebellar astrocytomas. The National Cancer Institute provides the PDQ pediatric cancer treatment information summaries as a public service to increase the availability of evidence-based cancer information to health professionals, patients, and the public. These summaries are updated regularly according to the latest published research findings by an Editorial Board of pediatric oncology specialists.

80. Cerebellum Resources
cerebellum diseases; where is the cerebellum located; mood and cerebellum;rabbits + cerebellum; diseases associated with the cerebellum.
http://www.1-2-3-low-fat-cooking-recipes.com/cerebellum.html
cerebellum
CLICK HERE TO ENTER CEREBELLUM RESOURCES
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