101. HealthCentral - General Encyclopedia - Atherosclerosis General Health Encyclopedia, atherosclerosis. Causes, incidence, and risk factors atherosclerosis is a common disorder of the arteries. http://www.healthcentral.com/mhc/top/000171.cfm

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102. Atherosclerosis NewJour Home NewJour A Search Prev Next atherosclerosis. Cynthia Reid wrote From Cynthia Reid CynthiaReid@worldnet.att http://gort.ucsd.edu/newjour/a/msg03076.html

NewJour Home NewJour: A Search [Prev] ... [Next] Atherosclerosis http://www.elsevier.nl/inca/publications/store/5/2/2/7/9/0/ Atherosclerosis brings together from all sources papers concerned with research and investigation on atherosclerosis, its complications, and related diseases, including: lipoprotein metabolism, arterial and vascular biology and disease, thrombosis, inflammation, disorders of lipid transport, diabetes and hypertension as related to atherosclerosis, and cardiovascular risk factors. The editors are also interested in clinical papers dealing with case studies of specific or general interest, new or unusual lipid syndromes, and the genetic basis and familial incidence of atherosclerosis and related diseases. High quality reports of controlled clinical trials of drugs or diets will be considered provided the paper deals with the mechanism of action of the drug or diet. ScienceDirect subscribers have access to full-text articles by logging in at http://www.sciencedirect.com . Editor: E.J. Schaefer and J. Shepherd Email: usinfo@sciencedirect.com NewJour Home NewJour: A Search [Prev] ... [Next]

103. Manbir Online ... Atherosclerosis atherosclerosis. atherosclerosis is a common form of hardening of the arteries due to plaque deposition. Risk factors for atherosclerosis. http://www.manbir-online.com/cardiac/anthero.1.htm

Home Medi News Medical Tidbits Interesting Topics ... Ask a Question Atherosclerosis Home Medi News Medical Tidbits Interesting Topics ... Alpha lipoic acid Atherosclerosis is a common form of hardening of the arteries due to plaque deposition. The arteries are the vessels that carry oxygen and other nutrients from heart to the other parts of body. Plaques are patches of fatty tissue that collect in the arteries and damage artery walls. As the fatty deposits accumulate, they reduce the elesticity of the blood vessels and narrow the passageway, thus interfering with the blood flow. The Atherosclerotic vascular disease leads to decreased circulation in the blood vessels of the brain, heart and extremeties. Signs and symptoms of Atheroslerosis Symptoms are often absent until atherosclerosis reaches advanced stages. Symptoms depend on which part of the body has decreased blood flow, and the extent of the disease. Presentations of atherosclerosis:- Angina pectoris (Cardiac pain) Myocardial infarction (Heart attacks) Transient ischaemic attacks (TIA) Strokes (Brain attacks) Intermittent claudication which may progress to gangrene of the organ / site involved.

104. What Is Atherosclerosis atherosclerosis (ather-o-skle-RO-sis) is the hardening and narrowing of the arteries. What is atherosclerosis? atherosclerosis http://www.nhlbi.nih.gov/health/dci/Diseases/Atherosclerosis/Atherosclerosis_Wha

DCI Home Atherosclerosis : What Is ... What Is ... Other Names Causes Who Is At Risk ... Links What is Atherosclerosis? Atherosclerosis (ath-er-o-skle-RO-sis) is the hardening and narrowing of the arteries. It is caused by the slow buildup of plaque (plak) on the inside of walls of the arteries. Arteries are blood vessels that carry oxygen-rich blood from the heart to other parts of the body. Plaque is made up of fat, cholesterol, calcium, and other substances found in your blood. As it grows, the buildup of plaque narrows the inside of the artery and, in time, may restrict blood flow. Plaque can be: Hard and stable, or Soft and unstable. Hard plaque causes artery walls to thicken and harden. Soft plaque is more likely to break apart from the walls and enter the bloodstream. This can cause a blood clot that can partially or totally block the flow of blood in the artery. When this happens, the organ supplied by the blocked artery starves for blood and oxygen. The organ's cells may either die or suffer severe damage. Atherosclerosis is a slow, progressive disease that may start in childhood. It can affect the arteries of the brain, heart, kidneys, and the arms and legs. As plaque builds up, it can cause serious diseases and complications. These include:

105. Hospital Practice: Atherosclerosis atherosclerosis An Infectious Disease? In most of these, casecontrol odds ratios have suggested a relationship between infection and atherosclerosis. http://www.hosppract.com/issues/1999/0901/benitez.htm

Atherosclerosis: An Infectious Disease? R. MICHAEL BENITEZ University of Maryland h Traditional risk factors do not fully account for atherogenesis. There is increasing evidence that infectious pathogens, such as Helicobacter pylori, cytomegalovirus, and Chlamydia pneumoniae, may promote the atherosclerotic cascade. Proposed mechanisms include macrophage transformation, endothelial injury, chronic inflammation, and thrombosis. Specific antibiotic therapy may affect outcome. Dr. Benitez is Assistant Professor, Division of Cardiology, University of Maryland School of Medicine, Baltimore. Atherosclerotic coronary artery disease (CAD) causes more than 1.5 million myocardial infarctions and contributes to 33% of all deaths in the United States each year. Age, gender, and a family history of CAD are recognized risk factors for the development of atherosclerosis, along with the modifiable risks of smoking, diabetes mellitus, hypertension, and hypercholesterolemia. Although treatment of modifiable risk factors is imperative, it is equally important that we continue to search for other contributors to the evolution of the atherosclerotic process. Considering the ubiquitous nature of infectious pathogens in human disease, it is not incongruous to ask whether there is a link between chronic infection and atherosclerotic disease. In the 1970s, experimental work suggested that such a link may exist. At that time, investigators reported that typical atherosclerosis developed in an avian model after the birds were infected with gallid herpesvirus (Marek's disease virus). Since then, seroepidemiologic investigations, microscopic examination of atherosclerotic plaques, and animal and human studies have suggested a possible link between the

106. Atherosclerosis MAIN SEARCH INDEX. atherosclerosis. (Illustration by Hans Cassady.). Definition. atherosclerosis is the build up of a waxy plaque on the inside of blood vessels. http://www.ehendrick.org/healthy/000162.htm

MAIN SEARCH INDEX Atherosclerosis Definition Description Causes and symptoms Diagnosis ... Resources The progression of atherosclerosis. Definition Atherosclerosis is the build up of a waxy plaque on the inside of blood vessels. In Greek, athere means gruel , and skleros means hard. Atherosclerosis is often called arteriosclerosis. Arteriosclerosis (from the Greek arteria, meaning artery ) is a general term for hardening of the arteries. Arteriosclerosis can occur in several forms, including atherosclerosis. Description Atherosclerosis, a progressive process responsible for most heart disease, is a type of arteriosclerosis or hardening of the arteries. An artery is made up of several layers: an inner lining called the endothelium, an elastic membrane that allows the artery to expand and contract, a layer of smooth muscle, and a layer of connective tissue. Arteriosclerosis is a broad term that includes a hardening of the inner and middle layers of the artery. It can be caused by normal aging , by high blood pressure, and by diseases such as diabetes. Atherosclerosis is a type of arteriosclerosis that affects only the inner lining of an artery. It is characterized by plaque deposits that block the flow of blood. Plaque is made of fatty substances, cholesterol, waste products from the cells, calcium, and fibrin, a stringy material that helps clot blood. The plaque formation process stimulates the cells of the artery wall to produce substances that accumulate in the inner layer. Fat builds up within these cells and around them, and they form connective tissue and calcium. The inner layer of the artery wall thickens, the artery's diameter is reduced, and blood flow and oxygen delivery are decreased. Plaques can rupture or crack open, causing the sudden formation of a blood clot (thrombosis). Atherosclerosis can cause a

107. Children With DIABETES - Diabetes Dictionary: A See also atherosclerosis. atherosclerosis One of many diseases in which fat builds up in the large and medium-sized arteries. http://www.childrenwithdiabetes.com/dictionary/a.htm

Advertisement E-mail Page Print Format Donate Diabetes Dictionary: A Jump to a new section [ Getting Around ] Home Page What's New User's Guide Index Search Site Map [ People ] Parents Kids Grads Grandparents Adults Friends IDYA Tidbits Parent humor Art Polls Surveys Message Board Chat cwdMail Mailing Lists Jobs Events Quilt for Life Rufus [ Clinic ] Ask the Diabetes Team Diabetes Basics Diabetes at School Sample 504 Plans Diabetes Camps Studies Products Insulin Pumps la Bomba de Insulina Nutrition Alternate Site Testing Diabetes Dictionary Presentations Warning Signs Marketplace [ Sources ] News and Information Press Releases Advocacy On-Line Links Support Groups Regional Info Services [ Feedback ] About Letters to Editor Contribute Advertising Online Store Logos and Linking Awards A B C D ... X Acanthosis Nigricans A dermatological disorder present in association with endocrine diseases (i.e., diabetes, obesity, pituitary tumors, or Cushing's disease). It is characterized by brown, velvety hyperkeratotic patches in the body folds. Acarbose A drug used as a treatment for Type 2 (noninsulin-dependent) diabetes ; belongs to a class of drugs called alpha-glucosidase inhibitors See also: Oral hypoglycemic agents ACE Inhibitor A type of drug used to lower blood pressure . Studies indicate that it may also help prevent or slow the progression of kidney disease in people with diabetes. ACE is an acronym for Angiotensin Converting Enzyme.

108. Homocysteine: A New Risk Factor For Atherosclerosis / October 15, 1997, AFP Homocysteine A New Risk Factor for atherosclerosis. The accumulating evidence for the role of homocysteine as a risk factor for atherosclerosis is persuasive. http://www.aafp.org/afp/971015ap/fallest.html

Advanced Search Articles Departments Patient Information This article exemplifies the AAFP 1997-98 Annual Clinical Focus on prevention and management of cardiovascular disease. Homocysteine: A New Risk Factor for Atherosclerosis PATRICIA C. FALLEST-STROBL, PH.D., DAVID D. KOCH, PH.D., JAMES H. STEIN, M.D., and PATRICK E. MCBRIDE, M.D., M.P.H. The accumulating evidence for the role of homocysteine as a risk factor for atherosclerosis is persuasive. A high plasma homocysteine concentration induces pathologic changes in the arterial wall and thus is strongly associated with an increased risk of atherosclerosis, manifested as cardiovascular, cerebrovascular and peripheral vascular events. Studies are being conducted to determine whether lowering homocysteine levels prevents occlusive events. At present, testing for elevated homocysteine concentrations should be considered in patients with premature atherosclerosis or a strong family history of atherosclerosis, since hyperhomocysteinemia is a common risk factor in these patients. Treatment of hyperhomocysteinemia is straightforward and associated with minimal risk. This disorder is usually correctable with vitamin supplements containing folic acid. An elevated plasma level of the amino acid homocysteine has been identified as an independent risk factor for atherosclerosis, including coronary artery disease, cerebrovascular disease, peripheral vascular disease and venous thromboembolism.

109. Anger Causes Atherosclerosis To Speed Up 12/10/00 atherosclerosis, the buildup of plaques in the arteries that can lead to a heart attack, progresses more quickly in people who express a lot of anger and in http://www.mercola.com/2000/dec/10/atherosclerosis_anger.htm

Dr. Joseph Mercola Author of the Total Health Program Enter your e-mail address below to subscribe to my free newsletter: Previous Issues Essential Info Health Blog My Vision My Qualifications #1 Natural Health Site ... More... Health Resources Nutrition Plan Fewer Grains/Sugars More Omega-3 More Water ... Issue 183 December 10, 2000 Some Drugs Can Increase Risk of Birth Defects Sluggish Thyroid Linked to Miscarriage Insulin Resistance Rampant Among Overweight Children Estrogen No Cure For Bone Loss ... Print this Page document.write ( "E-mail to a Friend" ); document.write ( "" ); Anger Causes Atherosclerosis to Speed Up Atherosclerosis, the build-up of plaques in the arteries that can lead to a heart attack, progresses more quickly in people who express a lot of anger and in those who have a weak social support system, new research suggests. Previous studies have linked anger, hostility and a weak social support system to artery problems, but this is the first study to examine the effects of these psychological factors on the progression of atherosclerosis. Researchers followed 150 people with diseased arteries.

110. NEJM -- From Asthma To Atherosclerosis -- 5-Lipoxygenase, Leukotrienes, And Infl Perspective from The New England Journal of Medicine From Asthma to atherosclerosis 5Lipoxygenase, Leukotrienes, and Inflammation. http://content.nejm.org/cgi/content/short/350/1/4

HOME SEARCH CURRENT ISSUE PAST ISSUES ... HELP Please sign in for full text and personal services Previous Volume 350:4-7 January 1, 2004 Number 1 Next Raffaele De Caterina, M.D., Ph.D., and Antonella Zampolli, Ph.D. Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings. Full Text PDF PDA Full Text Add to Personal Archive ... PubMed Citation Eicosanoids are lipid mediators of inflammation; they include a variety of compounds (prostaglandins, thromboxanes, leukotrienes, hydroxy- and epoxy-fatty acids, lipoxins, and isoprostanes) that are derived from the ubiquitous 20-carbon atom arachidonate (20 in Greek is "eicosa") and a few similar polyunsaturated fatty acids. These fatty acids are esterified in the phospholipids of biologic membranes and then released in response to a variety of stimuli to become substrates for metabolizing enzymes. More than 20 years ago, Samuelsson and coworkers identified leukotrienes as a class of eicosanoids that are derived through the action of 5-lipoxygenase. This enzyme, which is selectively expressed

111. NEJM -- Prevalence And Correlates Of Accelerated Atherosclerosis In Systemic Lup Original Article from The New England Journal of Medicine Prevalence and Correlates of Accelerated atherosclerosis in Systemic Lupus Erythematosus. http://content.nejm.org/cgi/content/short/349/25/2399

HOME SEARCH CURRENT ISSUE PAST ISSUES ... HELP Please sign in for full text and personal services Previous Volume 349:2399-2406 December 18, 2003 Number 25 Next Prevalence and Correlates of Accelerated Atherosclerosis in Systemic Lupus Erythematosus Mary J. Roman, M.D., Beth-Ann Shanker, A.B., Adrienne Davis, A.B., Michael D. Lockshin, M.D., Lisa Sammaritano, M.D., Ronit Simantov, M.D., Mary K. Crow, M.D., Joseph E. Schwartz, Ph.D., Stephen A. Paget, M.D., Richard B. Devereux, M.D., and Jane E. Salmon, M.D. Full Text PDF PDA Full Text Perspective ... Chapters at Harrison's ABSTRACT Background Although systemic lupus erythematosus is associated with premature myocardial infarction, the prevalence of underlying atherosclerosis and its relation to traditional risk factors for cardiovascular disease and lupus-related factors have not Methods In 197 patients with lupus and 197 matched controls, we performed carotid ultrasonography, echocardiography, and an assessment for risk factors for cardiovascular disease. The patients were also evaluated with respect to their clinical and serologic features, inflammatory mediators, and disease

112. Atherosclerosis - Cholesterol: Health And Medical Information About High Cholest atherosclerosis A process of progressive thickening and hardening of the walls of mediumsized and large arteries as a result of fat deposits on their inner http://www.medterms.com/script/main/art.asp?articlekey=15018

113. Atherosclerosis Heart Health. atherosclerosis. What is atherosclerosis? atherosclerosis, also atherosclerosis. How to avoid atherosclerosis. Remaining http://www.pfizer.ca/english/your health/heart health/atherosclerosis/default.as

114. GEArray Q Series Human Cardiovascular Disease Gene Array II: Atherosclerosis Gen . The GEArray Q Series Human Cardiovascular Disease Gene Array II is designed to analyze the relative expression of 96 atherosclerosisrelated genes....... http://www.superarray.com/gene_array_product/HTML/HS-038.html

GEArray Q Series Human Cardiovascular Disease Gene Array II: Atherosclerosis: HS-038 Description Need more information about GEArray products? please Go To GEArray Technology Overview Please click here to view gene table. Related Products Related Probe Synthesis Kits Related mRNA Purification Kits Related siRNA Products Functional Gene Grouping Molecules regulating inflammatory response: CCL2 (MCP-1), CCL5 (Rantes), CCL11 (eotaxin), CCL20 (MIP-3 alpha), CCR1, CCR2, CSF1 (M-CSF), CSF2 (GM-CSF), CSF3 (G-CSF), DSCR1, IFNAR2, IFNG, IL1A, IL1B, IL1R1, IL1R2, IL1RL1, IL2, IL3, IL4, IL5, IL6, IL7, IL8, IL13, LIF, NFKB1, NFKBIL2, TNF. Adhesion molecules and extracellular matrix molecules: CD44, CDH5 (VE-cadherin), COL3A1 (collagen), FN1 (fibronectin), ICAM1, ICAM2, ITGA2, ITGA5, ITGAX, ITGB2 (LFA-1), ITGB3, ITGB5, ITGB7, LAMA1 (laminin), MMP1, MMP3, MMP9, SELE (E-selectin), SELL (L-selectin), SELPLG (P-selectin), SERPINB2 (PAI-2), SERPINE1 (PAI-1), SPP1, THBS4, TNC, VCAM1. Cholesterol/lipid transport and metabolism: ABCA1 (ABC-1), ADFP (adipophilin), CD36, MSR1 (SR-A), OLR1 (LOX-1), SCARB1 (SR BI).

115. Nature Medicine Special Focus On Atherosclerosis Archive Search Information Classified Subscribe. top. Home, Copyright 2002 Nature Publishing Group Site Guide Feedback. http://www.nature.com/nm/special_focus/atherosclerosis/

116. Thrombosis Journal | Abstract | 1477-9560-2-5 | MRI Of Coronary Artery Atheroscl MRI of coronary artery atherosclerosis in rabbitsHistopathologyMRI correlation and atheroma characterization Rakesh Sharma and Ram B Singh Thrombosis Journal http://www.thrombosisjournal.com/content/2/1/5

Home Browse articles Search Weblinks ... Volume 2 Viewing options Abstract PDF Other links: E-mail to a friend Download references Post a comment PubMed record ... Related articles in PubMed Search PubMed For Sharma R Singh RB Key E-mail Original basic research MRI of coronary artery atherosclerosis in rabbits:Histopathology-MRI correlation and atheroma characterization Rakesh Sharma and Ram B Singh Thrombosis Journal Abstract (provisional) Background and objectives: We report in vivo magnetic resonance imaging (MRI) characteristics and histopathology correlation of the thrombus formation in atherosclerosis the rabbit animal model. Design and methods: Atherosclerosis was induced in white male rabbits fed with vegetable ghee followed by oxidized diet. Baseline MRI of atherosclerosis-recruited rabbits was done and later animals were used for atheroma histopathology characterization. Contiguous cross-sectional T2-weighted fast spin echo MRI images were compared by coronary histopathology. In all animals, coronary aortic wall thickening and atheroma size was measured using MRI. Results Conclusion The combination of in vivo MRI and comparison with histopathology images of rabbit coronary thrombus may be a research tool for understanding of the pathogenesis of acute coronary plaques.

117. Entrez PubMed Comment in Circulation. 2002 Oct 29;106(18)e1356; disscussion e135-6. Click here to read Inflammation and atherosclerosis. Libby http://www.thrombosisjournal.com/pubmed/11877368

Entrez PubMed Nucleotide Protein ... Books Search PubMed Protein Nucleotide Structure Genome Books CancerChromosomes 3D Domains Domains Gene GEO GEO DataSets HomoloGene Journals MeSH NCBI Web Site OMIM PMC PopSet SNP Taxonomy UniGene UniSTS for Limits Preview/Index History Clipboard ... Text Version Entrez PubMed Overview FAQ Tutorial New/Noteworthy ... E-Utilities PubMed Services Journals Database MeSH Database Single Citation Matcher Batch Citation Matcher ... Cubby Related Resources Order Documents NLM Gateway TOXNET Consumer Health ... PubMed Central Summary Brief Abstract Citation ASN.1 MEDLINE XML UI List LinkOut Related Articles Cited in Books CancerChrom Links Domain Links 3D Domain Links GEO DataSet Links Gene Links Genome Links GEO Links HomoloGene Links Nucleotide Links OMIM Links PMC Links Cited in PMC PopSet Links Protein Links SNP Links Structure Links UniSTS Links Show: Sort Author Journal Pub Date Text File Clipboard E-mail Order Circulation. 2002 Mar 5;105(9):1135-43. Related Articles, Links Comment in: Circulation. 2002 Oct 29;106(18):e135-6; disscussion e135-6. Inflammation and atherosclerosis. Libby P, Ridker PM, Maseri A.

118. FDA Heart Health Online - Atherosclerosis atherosclerosis Illustration. •, View...... Heart Health Home Conditions and Diseases atherosclerosis. atherosclerosis. On http://www.fda.gov/hearthealth/conditions/atherosclerosis.html

FDA Home Page Search FDA Site A-Z Index Contact FDA Powered by Google HOME The Healthy Heart Conditions and Diseases Risk Factors ... Conditions and Diseases Atherosclerosis On this page: Other Names Description Associated Conditions Risk Factors ... Links Other Names: Arteriosclerosis, Hardening of the Arteries Description: View pictures of atherosclerosis (Requires Flash Player - Download now Read text description of the atherosclerosis pictures In atherosclerosis, plaque builds up on the inner walls of arteries, the blood vessels that carry oxygen-rich blood throughout the body. As the artery walls thicken, the pathway for blood narrows. This can decrease or block blood flow through the body. Plaque is formed from cholesterol, fat, calcium, and other substances in the blood. When blood cholesterol levels are high, there is a greater chance that plaque will build up on the artery walls. In most people, this process begins when they are children or teenagers and worsens as they get older. Back to Top Associated Conditions: Coronary Artery Disease Angina Heart Attack (Myocardial Infarction) Stroke (Cerebrovascular Accident)

119. The Atherosclerosis File: Late-Breaking New Treatments Updated regularly over 100 descriptions of the latest treatments for atherosclerosis—from specialists in circulatory disorders worldwide. http://www.lifestages.com/health/atherosc.html

The Atherosclerosis File SM C E N T E R F O R C U R R E N T R E S E A R C H Approved by Physicians' Home Page Medinex Seal of Approval WellnessWeb: The Patient's Network HONcode Principles of the Health On the Net Foundation Partners of CareData.com W Atherosclerosis File . Learn about late-breaking research from recognized experts in circulatory disorders, vascular surgery, neurology, cardiology, and more. Compiled from the National Library of Medicine database at the National Institutes of Health, the Atherosclerosis File Atherosclerosis File informs you about studies at such renowned institutions as the University of Oxford, the Wake Forest University School of Medicine, and Harvard Medical School. The Atherosclerosis File brings you the inside medical story on: Carotid Atherosclerosis Surgery for Atherosclerosis General Treatments for Atherosclerosis Renal Atherosclerosis Genetic Factors in Atherosclerosis Hormone Therapy for Atherosclerosis General Studies in Atherosclerosis Access to Free Full-Text Articles from Postgraduate Medicine, the British Medical Journal, and the New England Journal of Medicine

120. University Of Manitoba: Centre For Research And Treatment Of Atherosclerosis/inf Centre for the Reseach and Treatment of atherosclerosis. atherosclerosis Information, About atherosclerosis. Athersclerosis http://www.umanitoba.ca/faculties/medicine/units/atherosclerosis/information/

C entre for the Reseach and Treatment of A therosclerosis Atherosclerosis Information About Atherosclerosis Athersclerosis is a vascular disease arising from an abnormal thickening and hardening of the arteries. It is caused by the deposit of cholesterol and other fatty substances on the inner lining of blood vessels, especially those in the the heart. This disorder is characterized by a inconsistant thickening of the artery from LDL uptake by cells in the artery. An LDL is essentially a large complex of proteins and lipids (20-25 nm in diameter) with the following structure: Figure 1. LDL structure These accumulations, referred to as "plaques", start from a fatty yellow "streak," to rounded dots or streaks on the artery surface. Fatty streaks are composed of cells which in turn are comprised primarily of cholesterol and lipids (namely triacylglycerides). Click here to see the chemical formula of a triglyceride (triG) in a popup window. Figure 2. The structure of cholesterol. Triacylglycerides, or TriG, are composed of 3 fatty acids esterified to a glycerol backbone.

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