81. JAMA & Archives -- Topic Collections : Bone Marrow Transplantation Full text. Past content. Contempo Updates Chronic graftvs-host disease Vikas Bhushan and Robert H. Collins, Jr JAMA 2003; 290 2599-2603. Full text. http://pubs.ama-assn.org/cgi/collection/bone_marrow_transplantation

Select Journal or Resource JAMA Archives of Dermatology Facial Plastic Surgery Family Medicine (1992-2000) General Psychiatry Internal Medicine Neurology Ophthalmology Surgery Student JAMA For The Media Classified Ads Meetings Peer Review Congress Bone Marrow Transplantation Contributing journals to this collection: JAMA Archives Journals Citations 1-10 of 15 total displayed. Most recent content Neurological Reviews Do Bone Marrow Cells Generate Neurons? David C. Hess, William D. Hill, James E. Carroll, and Cesar V. Borlongan Arch Neurol 2004; 61: 483-485. [Extract] [Full text] Past content Contempo Updates Chronic Graft-vs-Host Disease Vikas Bhushan and Robert H. Collins, Jr JAMA 2003; 290: 2599-2603. [Extract] [Full text] Review Articles Allogeneic Hematopoietic Stem Cell Transplantation: Complications and Results Imad A. Tabbara, Kathy Zimmerman, Connie Morgan, and Zeina Nahleh Archives of Internal Medicine 2002; 162: 1558-1566. [Abstract] [Full text] Studies Sclerodermatous Graft-vs-Host Disease: Clinical and Pathological Study of 17 Patients Arch Dermatol 2002; 138: 924-934.

82. Graft-vs.-host Disease Elicits Expression Of Class I And Class II Histocompatibi Proc Natl Acad Sci US A. 1987 April; 84 (7) 2082 2086 graftvs.-host disease elicits expression of class I and class II histocompatibility antigens and the http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=304589

83. Anatomic Pathology Anatomic Pathology / Original Article. graftvs-host disease After Solid Organ Transplant. Key Words graft-vs-host disease; Solid organ; Transplant; Chimerism. http://www.ajcp.com/previews/abstracts/202247artoc.html

Anatomic Pathology / Original Article Graft-vs-Host Disease After Solid Organ Transplant H. Evin Gulbahce, MD, Charlotte A. Brown, PhD, FACMG, Myra Wick, PhD, Miriam Segall, PhD, and Jose Jessurun, MD Key Words: Graft-vs-host disease; Solid organ; Transplant; Chimerism DOI: 10.1309/395BX683QFN6CJBC Abstract DOI: 10.1309/395BX683QFN6CJBC We value your input... If you have something to say regarding this article or ajcp.com in general, share it with us. If you would like to give us feedback on the Web site in general or make corrections to this article, click on Feedback below. To write correspondence to the editor regarding this article, please click on Comment below. Thank you. more abstracts Order full text article Comment American Society for Clinical Pathology 2100 West Harrison Street Chicago, Illinois 60612 Last Modified: April 14, 2004

84. Immunologic Diseases - NIAID Net News Articles targeted at the general public on allergy and asthma, graftvs-host diseases, immune deficiency diseases and autoimmune diseases. Also contains information on how the immune system works. http://www.niaid.nih.gov/final/immds/immds.htm

50 YEARS NIAID HOMEPAGE INFECTIOUS DISEASES IMMUNOLOGIC DISEASES AIDS ... NIH HOMEPAGE T he immune system is a complex network of specialized cells and organs. When it malfunctions, it can cause a wide array of problems. Scientists are making great strides in detecting, treating, and preventing disorders of the immune system. T Cell The healthy T cell is one type of immune system cell. How does the immune system work? Allergy and Asthma Usually harmless substances can provoke inappropriate immune responses. Graft-vs-Host Disease Transplantation of tissues and organs can cause life-threatening reactions in recipients. Immune Deficiency Diseases These diseases can be inherited or result from infections or drug treatments. Autoimmune Diseases The immune system mistakenly attacks the body. Looking Ahead Gene therapy and improved transplant procedures.

85. Transplant Rejection Averted By Simple Light Exposure In Stanford Animal Study Stanford University School of Medicine researchers now have a better grasp of this phenomenon, known as graftversus-host disease, or GVHD, and have proposed a http://www.eurekalert.org/pub_releases/2004-05/sumc-tra050504.php

Public release date: 5-May-2004 Contact: Ruthann Richter richter1@stanford.edu Stanford University Medical Center Transplant rejection averted by simple light exposure in Stanford animal study STANFORD, Calif. - One of the unfortunate side effects of bone marrow and stem cell transplantation is that the newly implanted cells often stage an internal attack against the patient they're intended to help. Stanford University School of Medicine researchers now have a better grasp of this phenomenon, known as graft-versus-host disease, or GVHD, and have proposed a possible method of prevention: simple ultraviolet light. In a new animal study, researchers identified the principal culprit in GVHD: an immune cell in the skin known as a Langerhans cell. These cells normally function as flag posts for the immune system, signaling infection-fighting T-cells to come to a particular site to fight off a virus or bacteria. But in transplants, they do patients a great disservice, alerting T-cells to attack the patient's own tissues, researchers found. The researchers were able to effectively eliminate the Langerhans cells in transplanted mice by exposing them to ultraviolet light, giving the animals mild sunburn. Transplanted mice that received this treatment experienced no GVHD while those that didn't showed severe signs of the disease, said Edgar Engleman, MD, professor of pathology and medicine and senior author of the study.

86. SkinGVHD CUTANEOUS LESIONS IN graft VERSUS host disease, Also visit Soft Tissue Tumour OnlineIndia GI Path Online-India, Dr. Sampurna Roy MD, http://www.geocities.com/sampyroy2000/SkinGVHD.html

CUTANEOUS LESIONS IN GRAFT VERSUS HOST DISEASE Also visit: Soft Tissue Tumour Online-India GI Path Online-India Dr. Sampurna Roy M.D. Graft versus host disease occurs in any situation in which immunologically competent cells or their precursors are transplanted into immunologically compromised recipients (Eg- In leukemia, aplastic anaemia and certain immunodeficiency states). GVHD is one of the most important causes of morbidity and mortality following bone marrow transplantation. Early acute phase: Acute phase begins between 7 and 21 days after transplantation. Besides the cutaneous lesions, the patient also complains of vomiting,diarrhoea and hepatic dysfuncton. Cutaneous lesions include: - Erythematous macular rash. - Occasionally follicular papules resembling folliculitis. - In severe cases, erythematous to violaceous scaling lesions and even blisters. -Very rarely toxic epidermal necrolysis may occur. Late chronic phase: Chronic phase develops between several months to a year after the transplantation. 1. Early lichenoid phase (both oral and cutaneous lesions clinically resemble lichen planus.

87. Small Bowel - Graft Versus Host Disease graft versus host disease. Because small intestine is rich in lymphoid tissue, it is not surprising to see that the incidence of http://tpis.upmc.edu/tpis/smbowel/SGVHDa.html

Graft versus Host Disease The morphological features of intestinal GVHD and acute rejection share a great degree of similarity. Both conditions histologically manifest with crypt cell apoptosis and pathogenetically show increased expression of TNF-a and involvement of cytotoxic T lymphocytes. The morphological similarities are most striking with latter instances of acute rejection in which apoptosis tends to be more pronounced and cellular infiltration is often less severe. In the latter episodes of acute rejection, the allograft gut-associated lymphoid tissue has been replaced by recipient lymphoid cells and immune reactivity is directed primarily at epithelial and stromal cells. Therefore, the overall immunologic process in latter episodes of acute rejection are more akin to GVHD. Obviously, because of the morphological similarities between acute rejection and GVHD, histological diagnosis of intestinal GVHD can only be made in the biopsies obtained from the native intestine. REFERENCES 1. Abu-Elmagd K, Reyes J, Todo S, Rao A, Lee R, Irish W, Furukawa H, Bueno J, McMichael J, Fawzy AT, Murase N, Demetris J, Rakela J, Fung JJ, Starzl TE. Clinical intestinal transplantation: New Perspectives and immunologic considerations. J Am Coll Surg 1998; 186:512-527.

88. Immunopathology The graft versus host disease here is marked by yellowbrown collections of bile in the canaliculi, as well as chronic inflammatory cells within the liver http://medlib.med.utah.edu/WebPath/IMMHTML/IMM034.html

The graft versus host disease here is marked by yellow-brown collections of bile in the canaliculi, as well as chronic inflammatory cells within the liver parenchyma.

89. Immunopathology Microscopically, graft versus host disease is one of the best examples of a process called apoptosis or single cell necrosis. http://medlib.med.utah.edu/WebPath/IMMHTML/IMM032.html

Microscopically, graft versus host disease is one of the best examples of a process called "apoptosis" or single cell necrosis. There is vacuolization and dissolution of epidermal cells along the basal layer, along with lymphocytes. At the arrow is a rounded pink apoptotic body.

90. The Sidney Kimmel Comprehensive Cancer Center At Johns Hopkins We're very sorry! The page you requested can not be found. If you typed the URL yourself, please make sure that the spelling is correct. If you clicked on a link to get here, there may be a problem http://www.hopkinskimmelcancercenter.org/clientpages/gvhdinformation.cfm

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