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         Graft Vs Host Disease:     more books (16)
  1. Graft-Vs.-Host Disease: Immunology, Pathophysiology, and Treatment (Hematology) by Steven J. Burakoff, 1990-07
  2. Gale Encyclopedia of Medicine: Graft-vs.-host disease by J. Ricker Polsdorfer MD, 2002-01-01
  3. Gale Encyclopedia of Cancer: Graft-vs.-host disease by M.S. Jill Granger, 2002-01-01
  4. Graft vs. Host Disease, Third Edition
  5. Graft-vs.-host disease: An entry from Thomson Gale's <i>Gale Encyclopedia of Cancer, 2nd ed.</i> by J., M.D. Polsdorfer, Jill, M.S. Granger, 2006
  6. Immunosuppressive Tx may get boost from adjunctive use of ECP. (Promising for Graft-vs.-Host Disease).(extracorporeal photophoresis ): An article from: Skin & Allergy News by Mitchel L. Zoler, 2003-07-01
  7. Graft vs. Host Disease, Third Edition by James Ferrara, 1980
  8. Graft-Versus-host Disease (Medical Intelligence Unit) by Nelson J. Chao, 1999-03-15
  9. Cutaneous manifestations of systemic disease: sarcoidosis, GVHD, behcet's disease, and pyoderma gangrenosum.(Dermatology Nursing Essentials: Core Knowledge)(Author ... An article from: Dermatology Nursing by Sue Ann McCann, 2007-02-01
  10. Talking Points in Dermatology - I (New Clinical Applications: Dermatology)
  11. Clinical and Diagnostic Pathology of Graft-versus-Host Disease by Berno Heymer, 2002-05-03
  12. Tacrolimus: An entry from Thomson Gale's <i>Gale Encyclopedia of Cancer, 2nd ed.</i> by Diane Calabrese, 2006
  13. Bacterial endotoxin and graft-versus-host-disease by Richard Hugh Moore, 1988
  14. Transfusion-associated graft-versus-host disease in an immunocompetent individual.(Disease/Disorder overview) : An article from: Indian Journal of Critical Care Medicine

41. BioSpace : CCIS : Search Results For Indication = 'Graft Vs. Host Disease'
approved products, please log in above or subscribe. 13 Search Results for Indication = graft vs. host disease . View as worksheet.
http://www.biospace.com/ccis/search.cfm?RXTargetID=57004

42. Graft-vs.-host Disease
graftvs.-host disease is an immune attack on the recipient by cells from a donor. donor and recipient are well matched, graft-vs.-host disease can still occur
http://www.chclibrary.org/micromed/00049690.html

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Definition Description Causes ... Resources
Graft-vs.-host disease
Definition
Graft-vs.-host disease is an immune attack on the recipient by cells from a donor. Description
The main problem with transplanting organs and tissues is that the recipient host does not recognize the new tissue as its own. Instead, it attacks it as foreign in the same way it attacks germs, to destroy it. If immunogenic cells from the donor are transplanted along with the organ or tissue, they will attack the host, causing graft vs. host disease. The only transplanted tissues that house enough immune cells to cause graft vs. host disease are the blood and the bone marrow. Blood transfusions are used every day in hospitals for many reasons. Bone marrow transplants are used to replace blood forming cells and immune cells. This is necessary for patients whose cancer treatment has destroyed their own bone marrow. Because bone marrow cells are among the most sensitive to radiation and chemotherapy , it often must be destroyed along with the cancer. This is true primarily of leukemias, but some other cancers have also been treated this way.
Even if the donor and recipient are well matched, graft-vs.-host disease can still occur. There are many different elements involved in generating immune reactions, and each person is different, unless they are identical twins. Testing can often find donors who match all the major elements, but there are many minor ones that will always be different. How good a match is found also depends upon the urgency of the need and some good luck.

43. Gale Encyclopedia Of Medicine Graft-vs.-host Disease
graftvs.-host disease. The only transplanted tissues that house enough immune cells to cause graft vs. host disease are the blood and the bone marrow.
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Graft-vs.-host disease
by J. Ricker Polsdorfer
Definition
Graft-vs.-host disease is an immune attack on the recipient by cells from a donor.
Description
The main problem with transplanting organs and tissues is that the recipient host does not recognize the new tissue as its own. Instead, it attacks it as foreign in the same way it attacks germs, to destroy it. If immunogenic cells from the donor are transplanted along with the organ or tissue, they will attack the host, causing graft vs. host disease. The only transplanted tissues that house enough immune cells to cause graft vs. host disease are the blood and the bone marrow. Blood transfusions are used every day in hospitals for many reasons. Bone marrow transplants are used to replace blood forming cells and immune cells. This is necessary for patients whose cancer treatment has destroyed their own bone marrow. Because bone marrow cells are among the most sensitive to radiation and chemotherapy, it often must be destroyed along with the cancer. This is true primarily of leukemias, but some other cancers have also been treated this way.
Even if the donor and recipient are well matched, graft-vs.-host disease can still occur. There are many different elements involved in generating immune reactions, and each person is different, unless they are identical twins. Testing can often find donors who match all the major elements, but there are many minor ones that will always be different. How good a match is found also depends upon the urgency of the need and some good luck.

44. Photopheresis For Chronic Graft Vs. Host Disease
Photopheresis for Chronic graft vs. host disease. This educational resource is supported by an unrestricted educational grant from therakos.logo.gif
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Photopheresis for Chronic Graft vs. Host Disease
This educational resource is supported by an unrestricted educational grant from
Published as part of Blood and Marrow Transplantation Reviews - Volume 12, Issue 4
Sunil Abhyankar, MD If you don't see a checkmark, download QuickTime Download: PC Mac 10.5 MB Help Viewing Private Lectures requires QuickTime Check here to determine if QuickTime is properly installed on your computer. Email questions and comments to PLS.Support@cjp.com Return to the BMTR - Vol. 12, Issue 4, contents page. Carden Jennings Publishing Co., Ltd. Featured Resources Optimizing Outcomes with IGIV Therapy Bloodline Reviews Volume 3, Issue 2

45. Extracorporeal Phtopheresis In Acute Graft Vs. Host Disease
Extracorporeal Phtopheresis in Acute graft vs. host disease. This educational resource is supported by an unrestricted educational grant from therakos.logo.gif
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Extracorporeal Phtopheresis in Acute Graft vs. Host Disease
This educational resource is supported by an unrestricted educational grant from
Published as part of Blood and Marrow Transplantation Reviews - Volume 12, Issue 4
Hildegard T. Greinix, MD If you don't see a checkmark, download QuickTime Download: PC Mac 10.5 MB Help Viewing Private Lectures requires QuickTime Check here to determine if QuickTime is properly installed on your computer. Email questions and comments to PLS.Support@cjp.com Return to the BMTR - Vol. 12, Issue 4, contents page. Carden Jennings Publishing Co., Ltd. Featured Resources Optimizing Outcomes with IGIV Therapy Bloodline Reviews Volume 3, Issue 2

46. Acute And Chronic Syngeneic Graft-vs-Host Disease
Acute and Chronic Syngeneic graftvs-host disease. Complexity of Effector Mechanisms in Cyclosporine-Induced Syngeneic graft-vs-host disease.
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Acute and Chronic Syngeneic Graft-vs-Host Disease
Complexity of Effector Mechanisms in Cyclosporine-Induced Syngeneic Graft-vs-Host Disease Allan D. Hess, Christopher J. Thoburn, Weiran Chen and Louis R. Horwitz Administration of the immunosuppressive drug, cyclosporine after syngeneic or autologous bone marrow transplantation elicits a T-lymphocyte-dependent autoimmune syndrome similar to graft-vs-host disease (GVHD). The onset of this autoaggression syndrome, termed syngeneic GVHD, is associated with the development of a highly restricted repertoire of CD8 autoreactive T cells that recognize a peptide from the invariant chain, termed CLIP, presented by MHC class II molecules. Clonal analysis reveals two distinct subsets of autoreactive T cells defined by their activation requirement for either the N terminal or the C terminal flanking regions of CLIP and by their cytokine profile. The present studies reveal that the autoreactive T cell clones requiring the N terminal flanking region of CLIP produce type 1 cytokines (interferon [IFN]- y , interleukin [IL]-2, and tumor necrosis factor- a ). In contrast, the autoreactive T cell clones that require the C terminal flanking region of CLIP produce type 2 cytokines (IL-4, IL-10, Transforming growth factor-

47. Lymphohematopoietic Graft-vs.-Host Reactions Without GVHD
Lymphohematopoietic graftvs.-host reactions can be induced without graft-vs.-host disease in murine mixed chimeras established with a cyclophosphamide-based
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Lymphohematopoietic Graft-vs.-Host Reactions without GVHD
Lymphohematopoietic graft-vs.-host reactions can be induced without graft-vs.-host disease in murine mixed chimeras established with a cyclophosphamide-based nonmyeloablative conditioning regimen Michele R. Pelot, Denise A. Pearson, Kirsten Swenson, Guiling Zhao, Jessica Sachs, Yong-Guang Yang, Megan Sykes 5.3.Pelot Carden Jennings Publishing Co., Ltd. Current Edition
Biology of Blood and Marrow Transplantation Laboratory Hematology Blood and Marrow Transplantation Reviews Heart Surgery Forum

48. Learn About Cord Blood, Stem Cells, And Us
cells? Q What are possible uses of cord blood stem cells in the future? Q What is graft vs. host disease (GVHD)? A graft vs. host
http://www.cordbloodfamilytrust.com/faq/index.cfm?faqID=84

49. ASCO - Browse By Meeting - Alemtuzumab Prevents Acute Graft-vs.-host Disease (aG
globulin and corticosteroids to methotrexate and cyclosporine (or tacrolimus) does not change the rates of acute graftvs-host disease, early infections
http://www.asco.org/ac/1,1003,_12-002636-00_18-0023-00_19-00101296,00.asp?Abstra

50. Genomics|HuGENet EJournal On Graft Vs Host Abstract
none. Health outcome(s) Identification of the major health outcome(s) studied, Acute graft versus host disease (GVHD), grade III or IV;
http://www.cdc.gov/genomics/hugenet/ejournal/GraftVsHostAbst.htm
Your browser does not support script home ejournal Relation of an Interleukin-10 Promoter Polymorphism to Graft-versus-Host Disease and Survival after Hematopoietic-Cell Transplantation back to report
May 6, 2004 Abstraction Template Article Reference
Complete the bibliographic reference for the article according to AJE format. Lin M, et al. Relation of an interleukin-10 promoter polymorphism to graft-versus-host disease and survival after hematopoietic-cell transplantation. NEJM. 2003;349:2201-2210. Category of HuGE information
Specify the types of information (from the list below) available in the article:
  • Prevalence of gene variant
  • Gene-disease association
  • Gene-environment interaction
  • Gene-gene interaction
  • Genetic test evaluation/monitoring
  • Gene-disease associations Study hypotheses or purpose
    The authors study hypotheses or main purpose for conducting the study Hypothesis: Polymorphisms in cytokine promoter regions that enhance or suppress cytokine expression may affect the severity of the inflammatory response involved in GVHD and associated survival. This study is meant to replicate the findings of other studies showing associations between a variety of cytokine promoter-region single-nucleotide polymorphisms and GVHD outcomes Gene(s)
    Identification of the following:
    • Gene name Chromosome location Gene product/function Alleles OMIM #
    Gene name
    Chromosome location
    Gene product/function : Interleukin 10; anti-inflammatory cytokine, influences Th2 differentiation

    51. Blackwell Synergy - Cookie Absent
    This article reviews acute graft vs. host disease (GVHD) as a complication of orthotopic liver transplantation (OLT). The incidence
    http://www.blackwell-synergy.com/links/doi/10.1111/j.1600-6143.2004.00406.x/enha
     Home An Error Occurred Setting Your User Cookie A cookie is a small amount of information that a web site copies onto your hard drive. Synergy uses cookies to improve performance by remembering that you are logged in when you go from page to page. If the cookie cannot be set correctly, then Synergy cannot determine whether you are logged in and a new session will be created for each page you visit. This slows the system down. Therefore, you must accept the Synergy cookie to use the system. What Gets Stored in a Cookie? Synergy only stores a session ID in the cookie, no other information is captured. In general, only the information that you provide, or the choices you make while visiting a web site, can be stored in a cookie. For example, the site cannot determine your email name unless you choose to type it. Allowing a web site to create a cookie does not give that or any other site access to the rest of your computer, and only the site that created the cookie can read it. Please read our for more information about data collected on this site.

    52. Dermatology Times : Photochemotherapy Relieves Case Of Graft Vs. Host Disease.(B
    Dermatology Times Photochemotherapy Relieves Case of graft vs. host disease.(Brief Article) @ HighBeam Research. Read Dermatology
    http://static.highbeam.com/d/dermatologytimes/april012000/photochemotherapyrelie
    Tour Become a Member ... Customer Support Question / Keyword(s): Advanced Search
    • Current Article: Photochemotherapy Relieves Case of Graft vs. Host Disease.(Brief Article)
    Start D Dermatology Times April 01, 2000 ... Photochemotherapy Relieves Case of Graft vs. Host Disease.(Brief Article)
    Photochemotherapy Relieves Case of Graft vs. Host Disease.(Brief Article)
    Dermatology Times; April 01, 2000; Wilson, Fred
    Wilson, Fred
    Dermatology Times
    April 01, 2000
    mycophenolate mofetil, bone marrow, skin thickness, marrow transplant, treatments, dr, sclerotic changes, puva bath, sclerodermic type, patient, patients, immunosuppressive therapy, peter, ulm, joint mobility
    Ulm, Germany A bone marrow transplant recipient with chronic
    sclerodermic graft-versus-host disease (GVHD) showed a strong response
    to UVA1 phototherapy combined with mycophenolate mofetil (CellCept)
    immunosuppressive therapy.
    The case of a 42-year-old man with a 1994 diagnosis of chronic
    myeloid leukemia was described in the Journal of the American Academy of Dermatology by researchers from the the University of Ulm. The patient

    53. Graft-vs-Host Disease : On Medical Dictionary Online
    graftvs-host disease defined on the Free Online Medical Dictionary. Medical terminology definitions graft-vs-host disease. The clinical entity
    http://www.online-medical-dictionary.org/?q=Graft-vs-Host Disease

    54. Clinical Trial: Sargramostim In Reducing Graft-Versus-Host Disease In Patients W
    Determine the efficacy of sargramostim (GMCSF) to mobilize CD34+ hematopoietic stem cells in donors and to reduce graft-vs-host disease in patients after
    http://www.clinicaltrials.gov/ct/gui/show/NCT00053157?order=50

    55. Graft Versus Host Disease GVHD
    Clinical Significance of Skin Biopsies in the Diagnosis and Management of graftvs-host disease in Early Postallogeneic Bone Marrow Transplantation Youwen Zhou
    http://www.edae.gr/graft.html

    56. Graft Vs. Host Disease Study: News: UI Health Care
    Health Care researcher is serving as a coprincipal investigator for a National Institutes of Health (NIH) grant to study graft versus host disease (GVHD) in
    http://www.uihealthcare.com/news/news/2001/01/22graft.html
    News by Medical Specialty 2004 News Archive 2003 News Archive 2002 News Archive ... 1999 News Archive - UI Health Care's digital library Read this month's health-e-newsletter Send comments and questions to
    staff@uihealthcare.com
    University of Iowa UI Health Care News: Week of January, 2001
    UI researcher helps lead study of bone marrow transplantation complication
    A University of Iowa Health Care researcher is serving as a co-principal investigator for a National Institutes of Health (NIH) grant to study Graft versus Host Disease (GVHD) in children. GVHD is a common and life-threatening complication that can follow bone marrow transplantation. Frederick Goldman, M.D., UI associate professor of pediatrics and director of the UI Pediatric Bone Marrow Transplant Unit, received $460,000 of the $2.3 million, five-year NIH grant for his component of the study. The study's lead investigator is Andrew Gilman, M.D., of Children's Mercy Hospital in Kansas City, Mo. The overall study goals are to evaluate the effectiveness of hydroxychloroquine, a new immunosuppressive drug, for treating GVHD and to better under what causes the disease, Goldman said. More than 100 pediatric bone marrow transplant centers nationwide will participate in the trial, which is sponsored by the federal Children's Oncology Group. The study will ultimately involve more than 300 pediatric patients.

    57. Graft-vs-Host Disease After Solid Organ Transplant
    graftvs-host disease (GVHD), a common complication of allogeneic bone marrow transplantation, also may be seen after solid organ transplantation and in
    http://www.medscape.com/viewarticle/452197

    58. Imaging In Bone Marrow Transplantation
    graftvs-host disease. GVHD is a significant posttransplantation complication that occurs in about 50% of allogeneic transplants.
    http://www.medscape.com/viewarticle/417694_2

    59. JW Dermatology -- Sign In
    Late Occurrence of graftvs-host disease. Valks R et al. Late appearance of acute graft-vs-host disease after suspending or tapering immunosuppressive drugs.
    http://dermatology.jwatch.org/cgi/content/full/2001/227/2

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    60. Graft-Versus-Host Disease
    graftvs-host disease After Solid Organ Transplant Sclerodermatous graft-vs-host disease clinical and pathological study of 17 patients.
    http://www.thedoctorsdoctor.com/diseases/graft_vs_host_disease.htm
    Background Graft-versus-host disease (GVH) is a disease of modern medicine. It most commonly occurs in bone marrow transplant patients receiving an allogeneic transplant of immunocompetent lymphocytes. The donor cytotoxic T lymphocytes attack the recipient's organs including skin, gut, liver, and mucous membranes. It may occur in 50% of recipients of allogeneic bone marrow transplants. GVH may also occur after maternofetal blood transfusions, intrauterine exchange transfusions, and administration of non-irradiated blood products to patients with metastatic malignancies or with immunodeficiencies. OUTLINE Epidemiology Disease Associations Pathogenesis Gross Appearance and Clinical Variants ... Internet Links
    EPIDEMIOLOGY CHARACTERIZATION SYNONYMS GVH DISEASE ASSOCIATIONS CHARACTERIZATION SOLID ORGAN TRANSPLANTATION Graft-vs-Host Disease After Solid Organ Transplant
    H. Evin Gulbahce, MD, Charlotte A. Brown, PhD, FACMG, Myra Wick, PhD, Miriam Segall, PhD, and Jose Jessurun, MD Am J Clin Pathol 2003;119:568-573 Abstract quote

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