IKidney - How Not To Cheat On Your Renal Diet you eat. When it comes to adhering to a healthrelated diet, renal disease patients are like anyone else. They tend to resist. As http://www.ikidney.com/iKidney/Lifestyles/NutritionalTips/Hemodialysis/HowNottoC
Extractions: As an End-Stage Renal Disease (ESRD) patient, your diet needs may differ greatly from the concepts of healthy living you hear about in the mainstream. For example, on dialysis, you must limit the potassium intake found in most fruits and vegetables and cut way down on your fluid intake when mainstream diets tell you to drink a lot of water. This can be very confusing! Many of the foods that are described as nutritious can be harmful to renal patients; therefore, it is vitally important to learn as much as you can about the foods you eat. When it comes to adhering to a health-related diet, renal disease patients are like anyone else. They tend to resist. As a former dialysis and current transplant patient, this is a feeling I understand. The diet can be very difficult. Some patients have heard it all, again and again, and its dietary litany drones on like a broken record. The reality is a renal diet is very difficult to adhere to. Frankly, I hate the word "diet" because it conjures up something at which most people do not succeed. I prefer to think of it as "my way" of healthy eating.
Extractions: Proper nutrition can help patients feel better, prevent complications and slow the disease progression. NutriGenie Kidney Disease Nutrition serves the specific needs of ESRD patients and allows them to conveniently monitor the protein, phosphorus, sodium, potassium, calcium and fluid contents of their diet. More products from the author:
Lhasa Apso Kidney Disease - Kai-La-Sha The treatment of renal dysplasia is essentially the same as for any chronic renal failure disease. A recent study suggests that a low protein diet may have http://www.lhasa-apso.org/health/hrd.htm
Extractions: Hereditary kidney disease WHAT IS RENAL DYSPLASIA? Renal dysplasia is a developmental or genetic defect of the kidneys. This makes it quite different from common forms of kidney disease which occur in adult or aged dogs and from other diseases, drugs and/or poisons which may cause inflammation of the kidneys and abnormal results on blood and urine test of kidney function. It is found most commonly in Shih Tzu and Lhasa Apsos. The dog is born with relatively immature kidneys. This changes rapidly in the first six weeks of life. In many animals, immature nephrons (urine-forming units) exist until 6-10 weeks of age. But in this disease, these immature nephrons persist throughout life. Also, some nephron units do not develop and are replaced with fibrous tissue. Sometimes this fibrous tissue represents 20-50% of the kidney, and the kidneys are noticably small and irregular. (Fig.1 ) Other evidence of renal dysplasia includes diffuse interstitial fibrosis in the cortex and medulla (which seems quite peculiar to this disease), reduced numbers of glomeruli (the filtering structure of the kidney where toxins, fluid and electrolytes are removed from the blood), dilated and hypoplastc tubules (through which the fluid or filtrate passes while it is being transformed into final urine), and a variety of sizes of glomeruli. Some glomeruli are 20-30% smaller than normal, some are normal size, and some are embryonic. (Fig. 2)
End-Stage Renal Disease: Choosing A Treatment That's Right For You This etext is for people whose kidneys fail to work. This condition is called end-stage renal disease (ESRD). It also discusses diet and paying for treatment. http://www.diabetic.com/education/pubs/esrd/esrd.htm
Extractions: This e-text is for people whose kidneys fail to work. This condition is called end-stage renal disease (ESRD). Today, there are new and better treatments for ESRD that replace the work of healthy kidneys. By learning about your treatment choices, you can work with your doctor to pick the one that's best for you. No matter which type of treatment you choose, there will be some changes in your life. But with the help of your health care team, family, and friends, you may be able to lead a full, active life. This e-text describes the choices for treatment: hemodialysis, peritoneal dialysis, and kidney transplantation. It gives the pros and cons of each. It also discusses diet and paying for treatment. It gives tips for working with your doctor, nurses, and others who make up your health care team. It provides a list of groups that offer information and services to kidney patients. It also lists magazines, books, and brochures that you can read for more information about treatment. You and your doctor will work together to choose a treatment that's best for you. This e-text can help you make that choice.
Professor Petot Base. Her research included the Modification of diet in renal disease (MDRD), a multicenter clinical intervention study. She is http://www.cwru.edu/med/nutrition/petot.html
19990831-End Stage Renal Disease Network Organizations Activities Factors associated with adherence to the dietary protein intervention in the modification of diet in renal disease study. J Am diet Assoc. 1995;9512951300. http://www.eatright.org/Public/GovernmentAffairs/98_lg083199.cfm
Extractions: Statement of the American Dietetic Association Good Morning. My name is Karen Basinger. I am a registered dietitian with over 16 years of experience working with renal patients and currently am a renal dietitian for TRC Wheaton. I am here today as a representative of the Renal Dietitians Dietetic Practice Group of the American Dietetic Association. ADA is the worlds largest organization of nutrition professionals and has long had an interest in the quality of care provided to End Stage Renal Disease (ESRD) patients. The Renal Dietitians DPG represents close to 2000 renal dietitians dedicated to improving the nutritional care for people with renal disease. I would like to thank you for the opportunity to provide testimony today. The renal dietitian is an integral part of the dialysis team. As we all know, our service population is getting older and coming to dialysis with an increasingly complex set of metabolic and clinical manifestations that requires the expertise of an experienced renal dietitian. The responsibilities of the renal dietitian practicing in ESRD facilities are also becoming more complex and demanding and include:
Renal Disease In Captive Swift Parrots ( 17, No. 4, pp. 206212. renal disease in Captive Swift Parrots (Lathamus discolor) The Effect of diet on Plasma Uric Acid Concentrations. http://www.bioone.org/bioone/?request=get-abstract&issn=1082-6742&volume=017&iss
Extractions: This article has been cited by other articles: Pedrini, M. T., Levey, A. S., Lau, J., Chalmers*, T. C., Wang, P. H. (1996). The Effect of Dietary Protein Restriction on the Progression of Diabetic and Nondiabetic Renal Diseases: A Meta-Analysis. Ann Intern Med [Abstract] [Full Text] Knight, E. L., Stampfer, M. J., Hankinson, S. E., Spiegelman, D., Curhan, G. C. (2003). The Impact of Protein Intake on Renal Function Decline in Women with Normal Renal Function or Mild Renal Insufficiency. Ann Intern Med [Abstract] [Full Text] Rahman, M., Smith, M. C. (1998). Chronic Renal Insufficiency: A Diagnostic and Therapeutic Approach. Arch Intern Med [Abstract] [Full Text] Malvy, D., Maingourd, C., Pengloan, J., Bagros, P., Nivet, H. (1999). Effects of Severe Protein Restriction with Ketoanalogues in Advanced Renal Failure.
NEJM -- The Progression Of Renal Disease Adler, S., Burkart, JM, Greene, T., Hebert, LA, Hunsicker, LG, King, AJ, Klahr, S., Massry, SG, Seifter, JL, Modification of diet in renal disease Study Group http://content.nejm.org/cgi/content/abstract/318/25/1657
Extractions: This article has been cited by other articles: Am. J. Physiol. [Abstract] [Full Text] Adamczak, M., Gross, M.-L., Krtil, J., Koch, A., Tyralla, K., Amann, K., Ritz, E. (2003). Reversal of Glomerulosclerosis after High-Dose Enalapril Treatment in Subtotally Nephrectomized Rats. J Am Soc Nephrol [Abstract] [Full Text] Baricos, W. H., Reed, J. C., Cortez, S. L. (2003). Extracellular Matrix Degradation by Cultured Mesangial Cells: Mediators and Modulators. Exp Biol Med [Abstract] [Full Text] Lin, S.-L., Chen, R.-H., Chen, Y.-M., Chiang, W.-C., Tsai, T.-J., Hsieh, B.-S. (2003). Pentoxifylline Inhibits Platelet-Derived Growth Factor-Stimulated Cyclin D1 Expression in Mesangial Cells by Blocking Akt Membrane Translocation. Mol Pharmacol [Abstract] [Full Text] The Modification of Diet in Renal Disease Study.
09. Pazienti - Patients (June 11, 1999) [85 Links] WRAMC) deke@vs.wramc.amedd.army.mil ESRD The End Stage renal disease Clinic - diet Information (WRAMC) deke@vs.wramc.amedd.army.mil ESRD - The End Stage http://www.sin-italia.org/internef/links09.htm
Extractions: AAKP (American Association of Kidney Patients) [AAKPnat@aol.com] ADA (American Diabetes Association) [customerservice@diabetes.org] AKF (Australian Kidney Foundation) [akf@adelaide.kidney.org.au] ALCER (Asociaciòn para la Lucha contra las Enfermedadesdel Riñòn) [federacio@alcer.org] ... Yahoo! Search Results: Health: Patients (Yahoo! Inc.)
Extractions: Background Calciphylaxis cutis is characterized by media calcification of arteries and, most prominently, of cutaneous and subcutaneous arterioles occurring in renal insufficiency patients. Case Report A 53-year-old woman with chronic cardiac and renal failure complained of painful crural, non-varicosis ulcers. She was hospitalized in an immobilized condition due to both the crural ulcerations and the existing heart-failure state (NYHA III-IV) having pleural and pericardial effusions, atrial fibrillation and weight loss of 30 kg over the past year. Despite normalization of calcium-phosphorus balance and improvement of renal function, the clinical course of crural ulcerations deteriorated during the following 3 months. After failure of surgical debridements, multiple courses of sterile-maggot therapy were introduced at a late stage to stabilize the wounds. The patient died of recurrent wound infections and sepsis paralleled by exacerbations of renal malfunction.
Publications--Renal Disease a common and expensive therapy for end stage renal disease. also handsearch the Journal of renal Nutrition since effect of a low protein diet on predialysis http://www.rand.org/health/epc/pubs/renalsum.html
Extractions: Introduction Chronic dialysis is a common and expensive therapy for end stage renal disease. The number of patients prescribed this therapy is increasing with time. Using a variety of measures, a number of articles studying patients in both the US and other countries have reported the prevalence of malnutrition in this population to range from 25% to 73%. Malnutrition increases morbidity, mortality, and cost, and produces growth retardation in children. It is therefore important to determine methods to adequately assess the nutritional status of dialysis patients, to prevent malnutrition from occurring in these patients, and to adequately treat malnutrition once it is discovered. This evidence report addresses nutritional prognostic, diagnostic, and treatment issues in dialysis patients. Methods We performed a systematic review to assemble and critically appraise relevant studies answering the clinical questions of interest. The technical experts for this Evidence Report are the members of the National Kidney Foundation (NKF) Dialysis Outcomes Quality Initiative (DOQI) Workgroups. With their input we formulated the following key clinical questions: Question 1 . Which measure of nutritional status best predicts patient morbidity/ mortality (and growth rate in children) in chronic dialysis patients?
Metabolic Basis Of Endothelial Dysfunction In Renal Disease Patients l Glomerular filtration rate (GFR) of 15ml/min/1.73m2, as assessed by the prediction equation from the Modification of diet in renal disease (MDRD) Study Group http://crnet.mgh.harvard.edu/clinical_trials/view_trial.asp?ct_id=782&clinic=&No
Blackwell Synergy - Cookie Absent Levey AS, Adler S, Caggiula AW, et al Effects of dietary protein restriction on the progression of advanced renal disease in the Modification of diet in renal http://www.blackwell-synergy.com/links/doi/10.1046/j.1523-1755.2001.00487.x/enha
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Canine Kidney Disease renal failure can also cause hypertension or high blood the initial step in the management of this disease. hypertension is not controlled by dietary management http://www.cah.com/library/caninekidney.html
Extractions: Canine Kidney Disease The kidney (fig. normal kidney ultrasound kidney Let us picture the kidneys filtering mechanism. There are tens of thousands of microscopic funnel shaped tubes called nephrons. These tiny structures are responsible for filtering and reabsorbing the fluids that balance the body. These nephrons are susceptible to damage due to many causes such as poisons, aging, infection, trauma, cancer, auto-immune diseases, and genetic predisposition. If any of these occur the entire nephron stops functioning. Fortunately, due to both the reserve capacity of the kidney and the ability of the nephrons to grow larger, the kidney can still function. If damage to nephrons occurs gradually and the surviving nephrons have enough time to hypertrophy, a kidney can continue to function with as few as 25 percent of its original nephrons. When the number of functioning nephrons drops below 25 percent or when damage occurs too suddenly for the remaining nephrons to compensate, kidney failure occurs. There are two types of kidney failure. Acute kidney failure is a sudden loss of function that is sometimes but not always reversible. Chronic kidney failure is an irreversible loss of function that occurs gradually over months or years. Initial test results can be remarkably similar for both forms of kidney failure. The diagnostic challenge is to determine whether the dog has acute or chronic kidney failure. Making the distinction between chronic and acute failure is crucial because the prognosis and duration of treatment for the two types of kidney disease are different (although some treatment procedures may be similar). At our practices, we recommend yearly base line lab testing starting at age 3. This protocol allows us to help determine if we are dealing with acute or chronic disease.
Eat Right To Feel Right On Hemodialysis Creative Cooking for renal Diabetic Diets Cleveland Clinic Foundation ISBN 0 Lawrence Y. Agodoa, MD EndStage renal disease Program NIDDK, National Institutes http://www.niddk.nih.gov/health/kidney/pubs/kidney-failure/eat-right/eat-right.h
Extractions: Kidney Failure Series Home Kidney Failure Series : Eat Right to Feel Right on Hemodialysis On this page: When you start hemodialysis, you must make many changes in your life. Watching the foods you eat will make you healthier. This publication will help you choose the right foods. Print this publication and use it with a dietitian to help you learn how to eat right to feel right on hemodialysis. Read one section at a time. Then go through the exercise with your dietitian. Once you have completed every exercise, keep a copy of this publication to remind yourself of foods you can eat and foods you need to avoid. Top Food gives you energy and helps your body repair itself. Food is broken down in your stomach and intestines. Your blood picks up nutrients from the digested food and carries them to all your body cells. These cells take nutrients from your blood and put waste products back into the bloodstream. When your kidneys were healthy, they worked around the clock to remove wastes from your blood. The wastes left your body when you urinated. Other wastes are removed in bowel movements. Talk to a dietitian to learn how to eat right on hemodialysis.
Early Renal Disease And ACE-Inhibitors Dietary Modification It is clear that phosphorus restriction is beneficial in the treatment of canine renal disease via decreasing stimulus for PTH (which is http://www.newmanveterinary.com/early.html
Extractions: This page is still under construction; Please see qualifier statement R ecent data in the veterinary and human literature concur regarding the beneficial effects of Angiotensinogen Converting Enzyme (ACE) inhibitors in the management of various stages of renal disease and renal failure. ACE inhibitors, such as enalapril, benazepril, linisopril...and others...are readily employed adjuncts in the management of some cardiomyopathies and congestive heart failure. These indications are independent of that which is to be discussed here for slowing the progression of renal disease and putting off the onset of renal failure. References for portions of this discussion can be found in the Veterinary Forum Compendium for Continuing Education , pp279, March, 1996; Clinics of North America, ( Small Animal), November, 1996; and Hospital Medicine pp11, 1997. A partially altered text version of the latter, which discusses this subject in humans, is available here The ensuing discussion represents the opinion of the author only. Diagnostic and treatment options for veterinary patients remain solely the responsibility of the attending veterinaria n.
THE MERCK MANUAL, Sec. 17, Ch. 222, Renal Failure Dietary modification may be helpful for hypertriglyceridemia, but fibric acid derivatives the rate of progression of the underlying renal disease and reduce http://www.merck.com/mrkshared/mmanual/section17/chapter222/222c.jsp