Huntingtons Disease huntingtons disease Menu. This is a webforum to discuss and comment on huntingtons disease Need advice on diet and vitamins (11/16/98) 930 PM http://neuro-www.mgh.harvard.edu/forum/HuntingtonsDiseaseMenu.html
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Huntington's Disease - Treatment - Neurologychannel such as constipation and incontinence can develop as a result of diet and lack of PhysicalActivity It is important for Huntingtons disease patients to be http://www.neurologychannel.com/huntingtons/treatment.shtml
Extractions: Physicians often prescribe various medications to help control emotional and movement problems. Antipsychotics (hallucinations, delusions, violent outbursts): haloperidol, chlorpromazine, olanzapine (contraindicated if patient has dystonia) Antidepressants (depression, obsessive-compulsive behavior): fluoxetine, sertraline hydrochloride, nortriptyline
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Huntington's Disease People with Huntington s disease can have difficulty in looking the family as knowingthe disease affects someone diet suffers through difficulty in eating and http://www.netdoctor.co.uk/diseases/facts/huntingtons.htm
Extractions: NetDoctor.co.uk Home News and features News Newsletter Features Encyclopaedia Diseases Examinations Medicines Premium services SMS services StayQuit thediet Health centres Allergy and asthma Children's health Depression Eczema ... All health centres Discussion and support Discussion forums Support groups Services Ask the doctor Find a hospital Search Medline Test yourself Information About NetDoctor Commercial opportunities NetDoctor.com Huntington's disease Dr Linda Appai-Kubi , King's College Hospital and Guy's King's ST. Thomas's School of Medicine, London Dr K Ray Chaudhuri , King's College Hospital and Guy's King's ST. Thomas's School of Medicine, London Huntington's disease (HD) was described by George Huntington (1850-1916) who was born in Long Island, New York. He described this disorder in his only known written article called 'on chorea'. Huntington's disease is a genetic disorder (inherited due to a faulty gene) which usually affects people in their 40s and 50s. It primarily affects the brain, with a gradual loss of control of movement, memory and mental ability. HD is also associated with personality changes and depression as well as other mental illnesses. There is currently no cure for HD, although many strategies may be used to improve the quality of life for sufferers and their carers.
Huntington Disease Introduction. What Causes huntingtons disease? How is HD Inherited The individual's physician can offer additional advice about diet and about how to handle swallowing difficulties or http://www.angelfire.com/md/danil/hutington.html
Extractions: DEGENERATION OF CHOLINERGIC AND GABAergic and ENKEPHALIN NEURONS IN THE BASAL GANGLIA AND THE CEREBRAL CORTEX [STRIATUM].Leading to increased activity of the inhibitory [GABAergic ] NEURONS FROM EXTERNAL GLOBUS PALLIDUS TO THE SUBTHALAMUS, WHICH CAUSES DECREASE IN THE GLUTAMATERGIC [EXCITATORY] NEURONAL ACTIVITY ON THE INTERNAL GLOBUS PALLIDUS. Result ia decreased inhibition on the thalamus, and the thalamus increases its excitatory [glutaminergic] effect on the cerebral cortex. Cause of the hyperkinesis. PROGRESSIVE SELECTIVE NEURAL CELL DEATH ASSOCIATED WITH:
Course List Diabetic Retinopathy, 1. diet, Weight Loss and Cardiovascular disease, 3. dietingand Weight Control, 1. Human Genome, 2. huntingtons disease, 2. Hypertension,3. http://www.medceu.com/viewcourses.cfm
Huntington Disease care with attention to nursing, diet, special equipment, and www.kumc.edu/hospital/huntingtons/index.html;Hereditary disease Foundation 11400 West Olympic Blvd http://www.geneclinics.org/profiles/huntington/details.html
Extractions: 25 May 2004 Disease characteristics. Huntington disease (HD) is a progressive disorder of motor, cognitive, and psychiatric disturbances. The mean age of onset is 35 to 44 years and the median survival time is 15 to 18 years after onset. Diagnosis/testing. The diagnosis of HD rests on positive family history , characteristic clinical findings, and the detection of an expansion in the HD gene that is 36 CAG trinucleotide repeats Genetic counseling HD is inherited in an autosomal dominant manner. Offspring of an individual with a mutant allele have a 50% chance of inheriting the disease-causing allele Predictive testing in asymptomatic adults at 50% risk is available, but requires careful thought, including pre-test and post-test genetic counseling , as no treatment exists. Asymptomatic at-risk minor children should not have predictive testing . Although infrequently requested
Huntingtons Aging. The study is the first to suggest that changes in diet can havean effect on the course of Huntington s disease. If decreasing http://www.youngagain2000.com/huntingtons.html
Extractions: That kind of fasting also protects nerve cells from genetically induced damage, says new research from the U.S. National Institute on Aging. The study is the first to suggest that changes in diet can have an effect on the course of Huntington's disease. If decreasing food intake has the same effect on humans, low-calorie diets or diets that feature reduced meal frequency may help delay the onset of Huntington's disease and extend the lives of people with the disease.
Huntington's Disease a person diagnosed with HD maintains a high calorie diet, not only Huntington sDisease Home Page. http//www.macalester.edu/~psych/whathap/UBNRP/Huntington http://www.lib.uchicago.edu/~rd13/hd/huntingtons.html
Extractions: Huntington's Disease (HD) is a dominant genetic disorder. Each child of a person who has HD has a 50% chance of inheriting the disease, and the disease does not skip a generation. HD is caused by a larger than normal CAG repeat in the Huntington gene. This larger than normal CAG repeat produces an abnormal protein that begins to kill brain cells when the person who has the gene reaches middle age. The loss of these cells causes intense symptoms and eventually death. HD was named after Dr. George Huntington. In 1872, he was the first person to document an accurate description of the symptoms and course of the disease. At the time he called it hereditary chorea. The Huntington gene was discovered in 1993 by the Huntington Study Group. The CAG repeats in the Huntington gene, which is located on chromosome 4, code for the protein huntingtin. The larger than normal number of CAGs in a person with HD causes the huntingtin protein to be abnormal, which leads to symptoms. Even though every cell in the body has the gene, only the cells in the brain seem to be affected. A person has two alleles for every gene. One allele is inherited from the mother and one allele is inherited from the father. If either allele of the Huntington gene has the larger than normal CAG repeat, the person will have HD. If neither allele has the larger than normal CAG repeat, the person will not have HD and will not pass it on. When a person has children, only one allele of each gene is passed on. If a person with HD passes on the allele with the larger than normal CAG repeat, the child will have HD. If the allele with the normal CAG is passed on, the child will not have HD.
What Is Huntingtons Disease A high calorie diet can prevent weight loss and improve symptoms such as The Huntington sDisease Association produces a full range of literature that looks at http://www.hda.org.uk/charity/whatishd.html
Extractions: The information contained on this page is available as a Fact Sheet which you can download from our Fact Sheet page. H untington's disease, which is often called HD, is an hereditary disorder of the central nervous system. It used to be known as Huntington's Chorea or HC. Huntington's disease usually develops in adulthood and can cause a very wide range of symptoms. The disease affects both men and women. Huntington's disease is caused by a faulty gene on chromosome 4. The gene, which produces a protein called Huntingtin, was discovered in 1993. In some way - which is not yet understood - the faulty gene leads to a damage of the nerve cells in areas of the brain, including the the basal ganglia and cerebral cortex. This leads to gradual physical, mental and emotional changes.
Extractions: Huntington's Disease (HD) Please note: This is a basic revision aid, not a comprehensive and reliable statement of medical fact. These notes should not be used to guide treatment. For reliable information and advice, consult a qualified practitioner. (Last updated: 19 March 2001) 1:20000 worldwide, all ethnic groups. Some reports say 1:10000. Usually 30-55 years old, but can be earlier or later. Autosomal dominant genetic disorder ( i.e. if one parent has HD, there is a 50% chance of each child developing HD). See our separate guide to genetic conditions for a fuller explanation of genetic transmission. The gene for HD is on chromosome 4 and is involved in producing a protein called Huntingtin, but there is little more useful information on how this leads to damage to the central nervous system. Caudate nucleus atrophy with generalised cerebral atrophy and atrophy of the corpus striatum. The particular mechanisms are unknown. Progressive dementia with increasingly prominent choreiform movements and likely personality and behavioural changes.
Index4 care are important with persons with huntingtons disease and as the disease progressesit becomes more of an issue. They have a high calorie diet and have a http://www.stfx.ca/people/x2000/x2000hyd/index4.htm
Extractions: Personal and Physical Care Needs In Huntingtons Disease there are cognitive changes and impairments. The cognitive areas that affect a persons performance include their speed, reasoning, planning, judgement, decision making, emotional engagement, preservation, impulse control, temper control, perception, awareness, attention, language, learning, memory and timing. Toileting Dressing Dental Care Contractures
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Extractions: path = "http://www.enhancedlifesciences.com/"; Home About Us Products Disorders ... Shopping Cart Muscular Dystrophy Association - The Muscular Dystrophy Association is a voluntary health agency a dedicated partnership between scientists and concerned citizens aimed at conquering neuromuscular diseases that affect more than a million Americans. Neuromuscular Disease Center - Clinical services for neuromuscular disorders as well as the regional MDA Clinic. Strength-L - An internet discussion list dedicated to people who have a form of neuromuscular disease . Information about the group, subscribing instructions and browse the archives. Lincolnshire Post-Polio Library: Neuromuscular Junction Transmission Defects - Article about anticholinesterase-responsive neuromuscular junction transmission defects in post-poliomyelitis fatigue. Neuromuscular Disease Center - Review of various muscle, nerve and neuromuscular disorders; links to relevant sites. Neuromuscular Junction Effects - Description of acetylcholinesterase inhibitors producing destructive changes at the neuromuscular junction. For medical professionals. Disorders of the Neuromuscular Junction - An introduction to disorders of the neuromuscular junction from Baylor College of Medicine.
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