Entrez PubMed sli@sint.azm.nl The cause of ankylosing spondylitis remains unclear. Proof that this disorder is an autoimmune disease attributable to crossreactivity between http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Abstra
The Lupus Lady's Autoimmune Disease Support Pages The Lupus Lady s autoimmune Disease Support Pages, Where friends gather for tea and comfort! ankylosing spondylitis. DESCRIPTION http://thelupuslady.com/autoimmune/sheet/ankylosing.html
Extractions: DESCRIPTION: Ankylosing Spondylitis (ANK-ki-low-sing spon-di-LIE-tis) is a chronic, or long-lasting, disease that primarily affects the spine and may lead to stiffness of the back. The joints and ligaments that normally permit the back to move become inflamed. The joints and bones may grow (fuse) together. The cause is unknown but genetics seems to play a role. A gene called HLA-B27 occurs in 90 percent of those with ankylosing spondylitis. Just because you have the gene, does not mean you will have ankylosing spondylitis. Other factors besides HLA-B27 are involved. Description from: Arthritis Foundation RECOMMENDED READING: Ankylosing Spondylitis: The Facts
Ankylosing Spondylitis -- Product Suggestions And Kits and joint tenderness, supress the production of inflamatory compounds, and inhibit the autoimmune reaction that is at issue in ankylosing spondylitis. http://www.healingwithnutrition.com/products/ankylosingspondylitiskit.html
Extractions: Total Toddy provides vitamin C, vitamin A, beta-carotene, selenium, zinc, l-glutathione, pantothenic acid, copper, niacin, boron, calcium and magnesium, free-form amino acids (to supply protein needed for tissue repair), manganese, folic acid (protects myelin surrounding nerves), and germanium which are all necessary nutrients to fight against ankylosing spondylitis. (One bottle provides a 30 day supply.)
Ankylosing Spondylitis AS (ankylosing spondylitis) is frequently used in medical literature. What causes it? The cause of AS is still unknown. It belongs to the autoimmune family of http://www.irishhealth.com/?level=4&con=388
AS - Ankylosing Spondylitis - Autoimmune Disease ankylosing spondylitis. AS. Appropriate Sustenance. gives. ANOTHER STORY. of. APPRECIABLE SUCCESS. UNDER CONSTRUCTION 6th April 2004. http://wwwstaff.murdoch.edu.au/~tlamont/as/
Extractions: It took a while, but finally i decided there's no future in writing the same stuff each time to new contacts so better put it in a final form. I am glad wrote to so many people about AS, about the starch-free diet as discussed on the forum (LSD/NSD) and about my approach, problems, solutions and successes. Writing to people helped me learn and to clarify my thoughts and ideas so the interaction with new people was as cathartic as the interactions with the more knowledgable co-enquirers from the forums. Here's hoping it was of use to those newly diagnosed people i met. There's so much to know and learn about this stuff that one can only do it gradually. Luckily, much of it is just good natural health principles so it is universally useful, we may have been exposed to it before, and if not then we still acquire useful and real knowledge for life. Lucky for me that i was well resourced at my employment with internet, access to scientific papers and to various scientific disciplines. There is a lot of fellowship and support on the "main" forum and chemical info etc. on others. There is info on the diet - called the London Diet originally - and scientific papers.
LookSmart - Directory - Ankylosing Spondylitis Gale Encyclopedia of Alt. Medicine ankylosing spondylitis Read about this autoimmune disease characterized by inflammation of the joints in the spine. http://search.looksmart.com/p/browse/us1/us317837/us317920/us53948/us54701/us274
Juvenile Ankylosing Spondylitis - Lucile Packard Children's Hospital type of autoimmune disease (a condition in which the body s own defenses begin to attack the body s cells). Treatment for juvenile ankylosing spondylitis The http://www.lpch.org/DiseaseHealthInfo/HealthLibrary/arthritis/juvank.html
Extractions: What is juvenile ankylosing spondylitis? Juvenile ankylosing spondylitis (JAS) is a type of arthritis that affects the spine and the sites where the muscles, tendons, and ligaments are attached to bone. "Ankylosing" means stiff or rigid, "spondyl" means spine, and "itis" refers to inflammation. The disease causes inflammation of the spine and large joints, resulting in stiffness and pain. The disease may result in erosion at the joint between the spine and the hip bone (the sacroiliac joint), and the formation of bony bridges between vertebrae in the spine, fusing those bones. In addition, bones in the chest may fuse. JAS is considered to be a multifactorial condition. Multifactorial inheritance means that "many factors" are involved in causing a health problem. The factors are usually both genetic and environmental, where a combination of genes from both parents, in addition to unknown environmental factors, produce the trait or condition. Often one gender (either males or females) is affected more frequently than the other in multifactorial traits. Multifactorial traits do recur in families because they are partly caused by genes. A group of genes on chromosome 6 code for HLA antigens play a major role in susceptibility and resistance to disease. Specific HLA antigens influence the development of many common disorders. Some of these disorders, such as JAS, are autoimmune related and inherited in a multifactorial manner. When a child has a specific HLA antigen type associated with the disease, he/she is thought to have an increased chance to develop the disorder. The HLA antigen associated with JAS is called B27. Children with the B27 HLA antigen are thought to have an increased chance (or "genetic susceptibility") to develop JAS; however, it is important to understand that a child without this antigen may also develop JAS. This means HLA antigen testing is not diagnostic or accurate for prediction of the condition. Males are affected with JAS three times more often than females.
Extractions: Juvenile Ankylosing Spondylitis Juvenile Ankylosing Spondylitis Juvenile ankylosing spondylitis (JAS) is a type of arthritis that affects the spine and the sites where the muscles, tendons, and ligaments are attached to bone. "Ankylosing" means stiff or rigid, "spondyl" means spine, and "itis" refers to inflammation. The disease causes inflammation of the spine and large joints, resulting in stiffness and pain. The disease may result in erosion at the joint between the spine and the hip bone (the sacroiliac joint), and the formation of bony bridges between vertebrae in the spine, fusing those bones. In addition, bones in the chest may fuse. JAS is considered to be a multifactorial condition. Multifactorial inheritance means that "many factors" are involved in causing a health problem. The factors are usually both genetic and environmental, where a combination of genes from both parents, in addition to unknown environmental factors, produce the trait or condition. Often one gender (either males or females) is affected more frequently than the other in multifactorial traits. Multifactorial traits do recur in families because they are partly caused by genes. A group of genes on chromosome 6 code for HLA antigens play a major role in susceptibility and resistance to disease. Specific HLA antigens influence the development of many common disorders. Some of these disorders, such as JAS, are autoimmune related and inherited in a multifactorial manner. When a child has a specific HLA antigen type associated with the disease, he/she is thought to have an increased chance to develop the disorder. The HLA antigen associated with JAS is called B27. Children with the B27 HLA antigen are thought to have an increased chance (or "genetic susceptibility") to develop JAS; however, it is important to understand that a child without this antigen may also develop JAS. This means HLA antigen testing is not diagnostic or accurate for prediction of the condition. Males are affected with JAS three times more often than females.
Extractions: Dictionaries: General Computing Medical Legal Encyclopedia Word: Word Starts with Ends with Definition Ankylosing Spondylitis (AS) is a chronic, progressive inflammatory arthritis Arthritis (from Greek arthro- , joint + -itis , inflammation) is a group of conditions that affect the health of the bone joints in the body. One in three adult Americans suffer from some form of Arthritis and the disease affects about twice as many women as men. Arthritic diseases include rheumatoid and psoriatic arthritis, which are autoimmune diseases; septic arthritis, caused by joint infection; and the more common osteoarthritis, or degenerative joint disease. Arthritis can be caused from strains and injuries caused by repetitive motion, sports, overexertion, and falls. Unlike the autoimmune diseases, osteoarthritis largely affects older people and results from the degeneration of joint cartilage. Arthritis may also be caused by gout.
Ankylosing Spondylitis Discussion ankylosing spondylitis View cases of ankylosing spondylitis. Clinical The pleuropulmonary manifestations of ankylosing spondylitis. http://www.auntminnie.com/ScottWilliamsMD2/Chest/Autoimmune/Ankspondylitis/Anksp
Extractions: Pulmonary parenchymal findings may mimic those of TB infection with apical fibrocavitary changes and cyst formation. If fungal superinfection has occurred a mycetoma may be identified. High-resolution CT can detect additional abnormalities not identified by plain film radiographs (2). REFERENCES: (1) Semin Arthritis Rheum 1989; Boushea DK, et al. The pleuropulmonary manifestations of ankylosing spondylitis. 18 (4): 277-281 (No abstract available) AJR 1997; Fenlon HM, et al. Plain radiographs and thoracic high-resolution CT in patients with ankylosing spondylitis. 168: 1067-1072 J Thorac Imaging 1992; Tanoue LT . Pulmonary involvement in collagen vascular disease: A review of the pulmonary manifestations of the Marfan syndrome, ankylosing spondylitis, Sjogren's syndrome, and relapsing polychondritis. 7 (2): 62-77
Reference 2 For Ankylosing Spondylitis Discussion Plain radiographs and thoracic highresolution CT in patients with ankylosing spondylitis. Fenlon HM, Casserly I, Sant SM, Breatnach E. http://www.auntminnie.com/ScottWilliamsMD2/Chest/Autoimmune/Ankspondylitis/Ref2.
AS Dietary Primer AS ankylosing spondylitis. Please refer to the glossary at the end of this paper for more information. Modality and Expression of AS and autoimmune Conditions. http://www.kickas.org/as_dietary_primer.shtml
Extractions: 4. Discussion Antibody elevations to Klebsiella pneumonias in AS patients were first reported in 1983 [5] and those original observations have since been confirmed in various centres [6]. Furthermore, antibodies to other gram negative organisms such as Escheric-hia coli, Proteus mirabilis, Pseudomonas aeruginosa, Salmonella spp, Shigella spp, and Yersinia enterolitica have not been found to be elevated in AS patients [5]. Additionally, a strong association between HLA-B27 and AS has been known for over 20 years [1]. These two observations were linked in 1987 [7] in the form of an amino acid homology between B*2705 and K. pneu-moniae nitrogenase. The data presented in this paper suggests a new sequence homology between B*2705 and another protein present in K. pneumoniae, the pulD secretion protein of the starch debranching pullulanase enzyme system (Table 1). Whilst the pulD sequence does not display the 100% homology shown with K. pneumoniae nitrogenase a similarity is nonetheless present in the form of a tetramer, (residues 596-599) (DRDE) and B*2705 (DRED). The pulD sequence (residues 596-599) only differs from the DRED sequence in B*2705 (74-77) and K. pneumoniae
CCHS Clinical Digital Library ankylosing spondylitis Patient/Family Resources. Miscellaneous ankylosing spondylitis Patients/Family Resources Arthritis Foundation Homepage http://cchs-dl.slis.ua.edu/patientinfo/rheumatology/autoimmune/spondyloarthropat
Extractions: Patient/Family Resources by Topic: Rheumatology Ankylosing Spondylitis Patient/Family Resources Pediatrics Spanish Miscellaneous See also: Arthritis Foundation: Homepage American College of Rheumatology: Homepage American Academy of Family Physicians (familydoctor.org): Homepage University of Birmingham (U.K.) Dept of Rheumatology: Homepage American Pain Foundation: Homepage MEDLINE plus Medical Encyclopedia: Table of contents InteliHealth Disease and Condition Guide Table of contents Merck Manual - Second Home Edition
CCHS Clinical Digital Library ankylosing spondylitis Clinical Resources. Miscellaneous ankylosing spondylitis Clinical Resources Arthritis Foundation Homepage; http://cchs-dl.slis.ua.edu/clinical/rheumatology/autoimmune/spondyloarthropathie
Extractions: Clinical Resources by Topic: Rheumatology Ankylosing Spondylitis Clinical Resources Pediatrics Radiology Pathology Clinical Guidelines ... Miscellaneous Resources See also: Chapter 315: Ankylosing Spondylitis, Reactive Arthritis, and Undifferentiated Spondyloarthropathy Table of contents Chapter 16: Back and Neck Pain Table of contents Causes of Back Pain: Access document Congenital Anomalies of the Lumbar Spine:
Arthritis LibraryAnkylosing Spondylitis ankylosing spondylitis is more common among whites than other races and is a link between intestinal bacteria or inflammation and the autoimmune activity (in http://www.hopkinsafter50.com/html/silos/arthritis/libAnkylosing.php
Extractions: Ankylosing spondylitis What Is It? The most serious complication is spinal fracture that can result in quadriplegia; however, most patients suffer no serious disability despite the pain and are able to continue working. The disease may get progressively worse, stabilize, or go into remission at any point. About 25 to 30 percent of AS patients are affected by inflammation and scarring of structures within the eye, and a few patients may develop an abnormality of the aortic valve in the heart or scarring of lung tissue. Ankylosing spondylitis is more common among whites than other races and is three times more prevalent in men than women. What Causes It? o The cause of AS is unknown, though hereditary factors appear to play a role. There is also evidence suggesting a link between intestinal bacteria or inflammation and the autoimmune activity (in which the body's defenses against disease attack healthy tissue) involved in the mechanism of joint deterioration. Prevention There is no known way to prevent AS.
MoonDragon's Health & Wellness: Arthritis RA is an autoimmune disorder a self-attacking-self disease - in which the body s immune system improperly identifies the synovial ankylosing spondylitis (AS http://www.moondragon.org/health/disorders/arthritis.html
Extractions: about options that may be available for your specific situation. BASIC INFORMATION DESCRIPTION Arthritis is the inflammation of one or more joints. It is characterized by pain, swelling, stiffness, deformity, and/or a diminished range of motion. More than 50 million Americans suffer from osteoarthritis, rheumatoid arthritis, and related conditions, such as fibromyalgia, gout, lupus, Lyme disease, psoriatic arthritis. Reiter's syndrome, Sjogren's syndrome, and ankylosing spondylitis. These conditions affect the body's movable, or synovial , joints. Joints of the body are found at the knees, wrists, elbows, fingers, toes, hips, and shoulders. The neck and the back also have joints between the bones of the spine. There are six different types of synovial joints (hinge, ball-and-socket, and so on), but although the types of motion they allow are different, their underlying physiological structure is essentially the same. Two or more adjoining movable bones, whose adjacent surfaces are covered with a layer of cartilage, are surrounded by a fluid-filled capsule made up of ligaments (tough, fibrous tissue). The third is secreted by a thin membrane, the synovial membrane, that lines the inside of the joint capsule. This viscous fluid along with the smooth, rubbery, blue-white cartilage that covers the ends of the bones normally work together to allow the bones within the join to glide smoothly past one another.
A To Z Encyclopedia Topic: Juvenile Ankylosing Spondylitis Juvenile ankylosing spondylitis (JAS) is a type of arthritis ankylosing means stiff or rigid, spondyl means spine such as JAS, are autoimmune related and http://web1.tch.harvard.edu/cfapps/A2ZtopicDisplay.cfm?Topic=Juvenile Ankylosing
Extractions: A PDF version of this document is available. Download PDF now (8 pages / 279 KB). More information on using PDF files. P atients with autoimmune diseases are frequently encountered by family physicians. It is important to understand not only the systemic effects of these diseases but also their ocular manifestations (Table 1) . Most ocular complications involve the cornea but may also include the conjunctiva, uvea, sclera, retina, and surrounding structures (Figure 1) . The majority of these diseases will ultimately need to be referred to an ophthalmologist. See page 937 for definitions of strength-of-evidence levels contained in this article. Rheumatoid Arthritis Approximately 25 percent of patients with rheumatoid arthritis (RA) will have ocular manifestations. These may include keratoconjunctivitis sicca, scleritis, episcleritis, keratitis, peripheral corneal ulceration, and less common entities such as choroiditis, retinal vasculitis, episcleral nodules, retinal detachments, and macular edema. Keratoconjunctivitis sicca, or dry eye syndrome, is the most common ocular manifestation of RA and has a reported prevalence of 15 to 25 percent.
